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Karim Gariani Service of Endocrinology, Diabetes, Nutrition and Therapeutic Patient Education, Geneva University Hospitals and Geneva University, Geneva, Switzerland

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François R Jornayvaz Service of Endocrinology, Diabetes, Nutrition and Therapeutic Patient Education, Geneva University Hospitals and Geneva University, Geneva, Switzerland
Diabetes Center, Faculty of Medicine, University of Geneva, Geneva, Switzerland

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hepatic manifestation of the metabolic syndrome ( 3 ). The Metabolic Syndrome is defined by the presence of any three of the five following risk factors: elevated waist circumference, elevated triglycerides (≥1.7 mmol/L), reduced HDL-cholesterol (<1.0 mmol

Article Type:
Review Article
DOI:
https://doi.org/10.1530/EC-20-0490
Volume/Issue:
Volume 10: Issue 2
Open access
Rongpeng Gong Medical College, Qinghai University, Xining, People’s Republic of China

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Gang Luo College of Eco-Environmental Engineering, Qinghai University, Xining, People’s Republic of China

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Mingxiang Wang Medical College, Qinghai University, Xining, People’s Republic of China

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Lingbo Ma Medical College, Qinghai University, Xining, People’s Republic of China

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Shengnan Sun Medical College, Qinghai University, Xining, People’s Republic of China

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Xiaoxing Wei Medical College, Qinghai University, Xining, People’s Republic of China

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suggested that the TG/HDL-C ratio is linked to IR, components of metabolic syndrome (MetS), exercise, physical activity, and smoking, but lack of alcohol intake, in a sample of 1068 healthy Japanese subjects ( 36 ). Similar findings were also reported by

Article Type:
Research Article
DOI:
https://doi.org/10.1530/EC-21-0414
Volume/Issue:
Volume 10: Issue 11
Open access
Bledar Daka Department of Internal Medicine, Department of Clinical Sciences Malmö, Social Medicine and Global Health, University of Gothenburg, PO Box 454, SE-405 30 Gothenburg, Sweden
Department of Internal Medicine, Department of Clinical Sciences Malmö, Social Medicine and Global Health, University of Gothenburg, PO Box 454, SE-405 30 Gothenburg, Sweden

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Thord Rosen Department of Internal Medicine, Department of Clinical Sciences Malmö, Social Medicine and Global Health, University of Gothenburg, PO Box 454, SE-405 30 Gothenburg, Sweden

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Per Anders Jansson Department of Internal Medicine, Department of Clinical Sciences Malmö, Social Medicine and Global Health, University of Gothenburg, PO Box 454, SE-405 30 Gothenburg, Sweden

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Lennart Råstam Department of Internal Medicine, Department of Clinical Sciences Malmö, Social Medicine and Global Health, University of Gothenburg, PO Box 454, SE-405 30 Gothenburg, Sweden

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Charlotte A Larsson Department of Internal Medicine, Department of Clinical Sciences Malmö, Social Medicine and Global Health, University of Gothenburg, PO Box 454, SE-405 30 Gothenburg, Sweden
Department of Internal Medicine, Department of Clinical Sciences Malmö, Social Medicine and Global Health, University of Gothenburg, PO Box 454, SE-405 30 Gothenburg, Sweden

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Ulf Lindblad Department of Internal Medicine, Department of Clinical Sciences Malmö, Social Medicine and Global Health, University of Gothenburg, PO Box 454, SE-405 30 Gothenburg, Sweden

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) . Whether SHBG is a biomarker for obesity and the metabolic syndrome or if it is more directly implicated in the physiopathology of the metabolic syndrome is less clear. Interestingly, a specific membrane receptor for SHBG has been confirmed in different

Article Type:
Research Article
DOI:
https://doi.org/10.1530/EC-12-0057
Volume/Issue:
Volume 2: Issue 1
Open access
Andrea V Haas Division of Endocrinology, Diabetes and Hypertension, Department of Medicine, Brigham and Women’s Hospital, Harvard Medical School, Boston, Massachusetts, USA

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Paul N Hopkins Cardiovascular Genetics, Department of Internal Medicine, University of Utah School of Medicine, Salt Lake City, Utah, USA

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Nancy J Brown Vanderbilt University Medical Center, Nashville, Tennessee, USA

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Luminita H Pojoga Division of Endocrinology, Diabetes and Hypertension, Department of Medicine, Brigham and Women’s Hospital, Harvard Medical School, Boston, Massachusetts, USA

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Jonathan S Williams Division of Endocrinology, Diabetes and Hypertension, Department of Medicine, Brigham and Women’s Hospital, Harvard Medical School, Boston, Massachusetts, USA

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Gail K Adler Division of Endocrinology, Diabetes and Hypertension, Department of Medicine, Brigham and Women’s Hospital, Harvard Medical School, Boston, Massachusetts, USA

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Gordon H Williams Division of Endocrinology, Diabetes and Hypertension, Department of Medicine, Brigham and Women’s Hospital, Harvard Medical School, Boston, Massachusetts, USA

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mounting evidence that CV risk is heritable ( 20 ). Members of our group have previously shown that a prevalent caveolin-1 (CAV1) gene variant (rs926198) is related to cardiovascular risk (insulin resistance and the metabolic syndrome) ( 21 , 22 ). CAV1 is

Article Type:
Research Article
DOI:
https://doi.org/10.1530/EC-19-0182
Volume/Issue:
Volume 8: Issue 6
Open access
Monika Karczewska-Kupczewska Department of Internal Medicine and Metabolic Diseases, Medical University of Białystok, Białystok, Poland

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Agnieszka Nikołajuk Department of Prophylaxis of Metabolic Diseases, Institute of Animal Reproduction and Food Research, Polish Academy of Sciences, Olsztyn, Poland

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Magdalena Stefanowicz Department of Metabolic Diseases, Medical University of Białystok, Białystok, Poland

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Natalia Matulewicz Department of Metabolic Diseases, Medical University of Białystok, Białystok, Poland

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Irina Kowalska Department of Internal Medicine and Metabolic Diseases, Medical University of Białystok, Białystok, Poland

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Marek Strączkowski Department of Prophylaxis of Metabolic Diseases, Institute of Animal Reproduction and Food Research, Polish Academy of Sciences, Olsztyn, Poland

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circulating chemerin concentration has been reported in obesity ( 8 , 9 , 10 ) and related comorbidities such as metabolic syndrome ( 11 ), nonalcoholic fatty liver disease (NAFLD) ( 12 ) and type 2 diabetes ( 9 , 13 ). Systemic chemerin concentrations

Article Type:
Research Article
DOI:
https://doi.org/10.1530/EC-20-0084
Volume/Issue:
Volume 9: Issue 5
Open access
Nikolaos Kyriakakis Department of Endocrinology, Leeds Centre for Diabetes & Endocrinology, Leeds Teaching Hospitals NHS Trust, Leeds, UK
Leeds Institute for Cardiovascular and Metabolic Medicine (LICAMM), University of Leeds, Leeds, UK

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Marilena Giannoudi Department of Endocrinology, Leeds Centre for Diabetes & Endocrinology, Leeds Teaching Hospitals NHS Trust, Leeds, UK

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Satish S Kumar Department of Endocrinology, Leeds Centre for Diabetes & Endocrinology, Leeds Teaching Hospitals NHS Trust, Leeds, UK

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Khyatisha Seejore Department of Endocrinology, Leeds Centre for Diabetes & Endocrinology, Leeds Teaching Hospitals NHS Trust, Leeds, UK
Leeds Institute for Cardiovascular and Metabolic Medicine (LICAMM), University of Leeds, Leeds, UK

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Georgios K Dimitriadis Department of Endocrinology, King’s College Hospital NHS Foundation Trust, Denmark Hill, London, UK

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Harpal Randeva Warwickshire Institute for the Study of Diabetes, Endocrinology and Metabolism (WISDEM), University Hospitals Coventry and Warwickshire NHS Trust, Coventry, UK

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Adam Glaser Pediatric Oncology, Leeds General Infirmary, Leeds Teaching Hospitals NHS Trust, Leeds, UK
Leeds Institute of Medical Research, University of Leeds, UK

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Michelle Kwok-Williams Clinical Oncology, Leeds Cancer Centre, Leeds Teaching Hospitals NHS Trust, Leeds, UK

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Georgina Gerrard Clinical Oncology, Leeds Cancer Centre, Leeds Teaching Hospitals NHS Trust, Leeds, UK

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Carmel Loughrey Clinical Oncology, Leeds Cancer Centre, Leeds Teaching Hospitals NHS Trust, Leeds, UK

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Ahmed Al-Qaissi Department of Endocrinology, Leeds Centre for Diabetes & Endocrinology, Leeds Teaching Hospitals NHS Trust, Leeds, UK

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Ramzi Ajjan Leeds Institute for Cardiovascular and Metabolic Medicine (LICAMM), University of Leeds, Leeds, UK

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Julie Lynch Department of Endocrinology, Leeds Centre for Diabetes & Endocrinology, Leeds Teaching Hospitals NHS Trust, Leeds, UK

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Robert D Murray Department of Endocrinology, Leeds Centre for Diabetes & Endocrinology, Leeds Teaching Hospitals NHS Trust, Leeds, UK
Leeds Institute for Cardiovascular and Metabolic Medicine (LICAMM), University of Leeds, Leeds, UK

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model assessment for insulin resistance (HOMA) equation (fasting plasma glucose (mmol/L) × fasting serum insulin (mU/L) divided by 22.5). The prevalence of metabolic syndrome was determined according to the International Diabetes Federation criteria ( 10

Article Type:
Research Article
Collections:
Hormones and Cancer
DOI:
https://doi.org/10.1530/EC-22-0491
Volume/Issue:
Volume 12: Issue 8
Open access
Hugo R Ramos Department of Internal Medicine, Section of Metabolic Vascular Medicine, Division of Diabetes and Nutritional Sciences, Cardiovascular Endocrinology Laboratory, Faculty of Medicine, Hospital de Urgencias, National University of Córdoba, X5000 Córdoba, Argentina

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Andreas L Birkenfeld Department of Internal Medicine, Section of Metabolic Vascular Medicine, Division of Diabetes and Nutritional Sciences, Cardiovascular Endocrinology Laboratory, Faculty of Medicine, Hospital de Urgencias, National University of Córdoba, X5000 Córdoba, Argentina
Department of Internal Medicine, Section of Metabolic Vascular Medicine, Division of Diabetes and Nutritional Sciences, Cardiovascular Endocrinology Laboratory, Faculty of Medicine, Hospital de Urgencias, National University of Córdoba, X5000 Córdoba, Argentina

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Adolfo J de Bold Department of Internal Medicine, Section of Metabolic Vascular Medicine, Division of Diabetes and Nutritional Sciences, Cardiovascular Endocrinology Laboratory, Faculty of Medicine, Hospital de Urgencias, National University of Córdoba, X5000 Córdoba, Argentina

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ventricular hypertrophy (96) . Conversely, conditions with a cNP deficit are associated with cardiac hypertrophy in hypertensive patients. In this regard, hypertensive patients with the metabolic syndrome present with lower ANP and NT-proBNP levels and

Article Type:
Review Article
DOI:
https://doi.org/10.1530/EC-15-0018
Volume/Issue:
Volume 4: Issue 3
Open access
Barbara J Boucher The Blizard Institute, Queen Mary University of London, London, UK

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-adjusted mortality rates than indigenous Britons, aggravated by their increased rates of obesity, metabolic syndrome, type 2 diabetes and cardiovascular disease ( 31 , 32 ). Obesity increases the rates and severity of vitamin D deficiency through well

Article Type:
Review Article
Collections:
Bone and Mineral Metabolism
DOI:
https://doi.org/10.1530/EC-22-0234
Volume/Issue:
Volume 11: Issue 12
Open access
Xiao-jun Zhou Department of Endocrinology, Shandong Provincial Qianfoshan Hospital, Shandong University, Jinan, Shandong, China

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Lin Ding Department of Endocrinology, Shandong Provincial Qianfoshan Hospital, Shandong University, Jinan, Shandong, China

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Jia-xin Liu Department of Endocrinology, Shandong Provincial Qianfoshan Hospital, Shandong University, Jinan, Shandong, China

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Le-qun Su Department of Pharmacy, Shandong Provincial Qianfoshan Hospital, Shandong University, Jinan, Shandong, China

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Jian-jun Dong Division of Endocrinology, Department of Medicine, Qilu Hospital of Shandong University, Shandong University, Jinan, Shandong, China

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Lin Liao Department of Endocrinology, Shandong Provincial Qianfoshan Hospital, Shandong University, Jinan, Shandong, China

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potentially linking obesity to the metabolic syndrome ( 21 ). Higher visceral adipose tissue DPP4 gene expression in non-diabetic severely obese men with metabolic syndrome was observed than non-metabolic syndrome. An association between the DPP-4 methylation

Article Type:
Research Article
DOI:
https://doi.org/10.1530/EC-18-0523
Volume/Issue:
Volume 8: Issue 4
Open access
Ahmet Uçar
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Nurçin Saka
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Firdevs Baş
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Nihal Hatipoğlu Pediatric Endocrine Unit, Pediatric Endocrine Unit, Istanbul Medical Faculty, Istanbul University, 34093 Fatih, Istanbul, Turkey

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Rüveyde Bundak
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Feyza Darendeliler
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uncertain, and genetic factors have been proposed (2, 3) . PA has been associated with type 2 diabetes, coronary arterial disease (CAD), hypertension, and metabolic syndrome in adulthood (4, 5) . These long-term complications have been linked to low birth

Article Type:
Research Article
DOI:
https://doi.org/10.1530/EC-12-0059
Volume/Issue:
Volume 2: Issue 1
Open access