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Michelle J Galvan Metabolic, Nutrition, and Exercise Research (MiNER) Laboratory, Department of Kinesiology, University of Texas at El Paso, El Paso, Texas, USA

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Michael J Sanchez Metabolic, Nutrition, and Exercise Research (MiNER) Laboratory, Department of Kinesiology, University of Texas at El Paso, El Paso, Texas, USA

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Andrew J McAinch Institute for Health and Sport (IHES), Victoria University, Melbourne, Victoria, Australia
Australian Institute for Musculoskeletal Science (AIMSS), Victoria University, Melbourne, Victoria, Australia

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Jeffrey D Covington Department of Pathology, University of Massachusetts Medical School, Worcester, Massachusetts, USA

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Jason B Boyle Metabolic, Nutrition, and Exercise Research (MiNER) Laboratory, Department of Kinesiology, University of Texas at El Paso, El Paso, Texas, USA

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Sudip Bajpeyi Metabolic, Nutrition, and Exercise Research (MiNER) Laboratory, Department of Kinesiology, University of Texas at El Paso, El Paso, Texas, USA

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dramatic increase in obesity, insulin resistance, and type 2 diabetes mellitus (T2DM) ( 2 ). Adherence is further challenging for those with obesity and T2DM suffering from defects in lipid oxidation capacity, musculoskeletal pain, and peripheral neuropathy

Article Type:
Research Article
Collections:
Metabolic Syndrome and Diabetes
DOI:
https://doi.org/10.1530/EC-21-0533
Volume/Issue:
Volume 11: Issue 2
Open access
Valentina Guarnotta Dipartimento di Promozione della Salute, Materno-Infantile, Medicina Interna e Specialistica di Eccellenza ‘G. D’Alessandro’ (PROMISE), Sezione di Malattie Endocrine, del Ricambio e della Nutrizione, Università di Palermo, Palermo, Italy

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Silvia Lucchese Dipartimento di Promozione della Salute, Materno-Infantile, Medicina Interna e Specialistica di Eccellenza ‘G. D’Alessandro’ (PROMISE), Sezione di Malattie Endocrine, del Ricambio e della Nutrizione, Università di Palermo, Palermo, Italy

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Mariagrazia Irene Mineo Dipartimento di Promozione della Salute, Materno-Infantile, Medicina Interna e Specialistica di Eccellenza ‘G. D’Alessandro’ (PROMISE), Sezione di Malattie Endocrine, del Ricambio e della Nutrizione, Università di Palermo, Palermo, Italy

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Donatella Mangione Dipartimento di Promozione della Salute, Materno-Infantile, Medicina Interna e Specialistica di Eccellenza ‘G. D’Alessandro’ (PROMISE), Sezione di Ostetricia e Ginecologia, Università di Palermo, Palermo, Italy

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Renato Venezia Dipartimento di Promozione della Salute, Materno-Infantile, Medicina Interna e Specialistica di Eccellenza ‘G. D’Alessandro’ (PROMISE), Sezione di Ostetricia e Ginecologia, Università di Palermo, Palermo, Italy

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Piero Luigi Almasio Dipartimento di Promozione della Salute, Materno-Infantile, Medicina Interna e Specialistica di Eccellenza ‘G. D’Alessandro’ (PROMISE), Sezione di Gastroenterologia ed Epatologia, Università di Palermo, Palermo, Italy

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Carla Giordano Dipartimento di Promozione della Salute, Materno-Infantile, Medicina Interna e Specialistica di Eccellenza ‘G. D’Alessandro’ (PROMISE), Sezione di Malattie Endocrine, del Ricambio e della Nutrizione, Università di Palermo, Palermo, Italy

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) ( 1 , 2 ). Insulin resistance with total, visceral, and hepatic adiposity is thought to be a major driver of such early maturation ( 3 , 4 ). Non-classical congenital adrenal hyperplasia or heterozygous carriers of 21-hydroxylase deficiency can

Article Type:
Research Article
Collections:
Paediatric Endocrinology Collection
DOI:
https://doi.org/10.1530/EC-21-0118
Volume/Issue:
Volume 10: Issue 7
Open access
Aline Stangherlin Martins Neurofibromatosis Outpatient Reference Center, Department of Nutrition, Federal University of Minas Gerais, Department of Medical Clinic, Federal University of Minas Gerais, Department of Preventive and Social Medicine, Federal University of Minas Gerais, Department of Internal Medicine, Federal University of Minas Gerais, The Neurofibromatosis Institute, Federal University of Minas Gerais, Alameda Álvaro Celso 55, Belo Horizonte, Minas Gerais, Brazil

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Ann Kristine Jansen Neurofibromatosis Outpatient Reference Center, Department of Nutrition, Federal University of Minas Gerais, Department of Medical Clinic, Federal University of Minas Gerais, Department of Preventive and Social Medicine, Federal University of Minas Gerais, Department of Internal Medicine, Federal University of Minas Gerais, The Neurofibromatosis Institute, Federal University of Minas Gerais, Alameda Álvaro Celso 55, Belo Horizonte, Minas Gerais, Brazil

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Luiz Oswaldo Carneiro Rodrigues Neurofibromatosis Outpatient Reference Center, Department of Nutrition, Federal University of Minas Gerais, Department of Medical Clinic, Federal University of Minas Gerais, Department of Preventive and Social Medicine, Federal University of Minas Gerais, Department of Internal Medicine, Federal University of Minas Gerais, The Neurofibromatosis Institute, Federal University of Minas Gerais, Alameda Álvaro Celso 55, Belo Horizonte, Minas Gerais, Brazil

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Camila Maria Matos Neurofibromatosis Outpatient Reference Center, Department of Nutrition, Federal University of Minas Gerais, Department of Medical Clinic, Federal University of Minas Gerais, Department of Preventive and Social Medicine, Federal University of Minas Gerais, Department of Internal Medicine, Federal University of Minas Gerais, The Neurofibromatosis Institute, Federal University of Minas Gerais, Alameda Álvaro Celso 55, Belo Horizonte, Minas Gerais, Brazil

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Marcio Leandro Ribeiro Souza Neurofibromatosis Outpatient Reference Center, Department of Nutrition, Federal University of Minas Gerais, Department of Medical Clinic, Federal University of Minas Gerais, Department of Preventive and Social Medicine, Federal University of Minas Gerais, Department of Internal Medicine, Federal University of Minas Gerais, The Neurofibromatosis Institute, Federal University of Minas Gerais, Alameda Álvaro Celso 55, Belo Horizonte, Minas Gerais, Brazil

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Juliana Ferreira de Souza Neurofibromatosis Outpatient Reference Center, Department of Nutrition, Federal University of Minas Gerais, Department of Medical Clinic, Federal University of Minas Gerais, Department of Preventive and Social Medicine, Federal University of Minas Gerais, Department of Internal Medicine, Federal University of Minas Gerais, The Neurofibromatosis Institute, Federal University of Minas Gerais, Alameda Álvaro Celso 55, Belo Horizonte, Minas Gerais, Brazil

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Maria de Fátima Haueisen Sander Diniz Neurofibromatosis Outpatient Reference Center, Department of Nutrition, Federal University of Minas Gerais, Department of Medical Clinic, Federal University of Minas Gerais, Department of Preventive and Social Medicine, Federal University of Minas Gerais, Department of Internal Medicine, Federal University of Minas Gerais, The Neurofibromatosis Institute, Federal University of Minas Gerais, Alameda Álvaro Celso 55, Belo Horizonte, Minas Gerais, Brazil

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Sandhi Maria Barreto Neurofibromatosis Outpatient Reference Center, Department of Nutrition, Federal University of Minas Gerais, Department of Medical Clinic, Federal University of Minas Gerais, Department of Preventive and Social Medicine, Federal University of Minas Gerais, Department of Internal Medicine, Federal University of Minas Gerais, The Neurofibromatosis Institute, Federal University of Minas Gerais, Alameda Álvaro Celso 55, Belo Horizonte, Minas Gerais, Brazil

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Leonardo Mauricio Diniz Neurofibromatosis Outpatient Reference Center, Department of Nutrition, Federal University of Minas Gerais, Department of Medical Clinic, Federal University of Minas Gerais, Department of Preventive and Social Medicine, Federal University of Minas Gerais, Department of Internal Medicine, Federal University of Minas Gerais, The Neurofibromatosis Institute, Federal University of Minas Gerais, Alameda Álvaro Celso 55, Belo Horizonte, Minas Gerais, Brazil

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Nilton Alves de Rezende Neurofibromatosis Outpatient Reference Center, Department of Nutrition, Federal University of Minas Gerais, Department of Medical Clinic, Federal University of Minas Gerais, Department of Preventive and Social Medicine, Federal University of Minas Gerais, Department of Internal Medicine, Federal University of Minas Gerais, The Neurofibromatosis Institute, Federal University of Minas Gerais, Alameda Álvaro Celso 55, Belo Horizonte, Minas Gerais, Brazil
Neurofibromatosis Outpatient Reference Center, Department of Nutrition, Federal University of Minas Gerais, Department of Medical Clinic, Federal University of Minas Gerais, Department of Preventive and Social Medicine, Federal University of Minas Gerais, Department of Internal Medicine, Federal University of Minas Gerais, The Neurofibromatosis Institute, Federal University of Minas Gerais, Alameda Álvaro Celso 55, Belo Horizonte, Minas Gerais, Brazil

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Vincent Michael Riccardi Neurofibromatosis Outpatient Reference Center, Department of Nutrition, Federal University of Minas Gerais, Department of Medical Clinic, Federal University of Minas Gerais, Department of Preventive and Social Medicine, Federal University of Minas Gerais, Department of Internal Medicine, Federal University of Minas Gerais, The Neurofibromatosis Institute, Federal University of Minas Gerais, Alameda Álvaro Celso 55, Belo Horizonte, Minas Gerais, Brazil

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previous studies (6, 8) . Both type 1 and type 2 DM contribute to development of VD (12) . Type 1 DM is associated with an inadequate availability of insulin and usually manifests in childhood, whereas type 2 DM is related to insulin resistance and

Article Type:
Research Article
DOI:
https://doi.org/10.1530/EC-15-0102
Volume/Issue:
Volume 5: Issue 1
Open access
E N Dudinskaya
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O N Tkacheva
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M V Shestakova National Research Center for Preventive Medicine, Endocrinology Research Centre, Building 10, Petroverigskiy Lane, Moscow 101990, Russian Federation

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N V Brailova
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I D Strazhesko
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D U Akasheva
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O Y Isaykina
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N V Sharashkina
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D A Kashtanova
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S A Boytsov
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ageing and the development of age-related diseases. In addition, insulin resistance (IR) is considered to be a predictor of atherosclerosis and CVD independently of other risk factors, such as blood lipid levels, and hyperglycaemia results in ageing

Article Type:
Research Article
DOI:
https://doi.org/10.1530/EC-15-0041
Volume/Issue:
Volume 4: Issue 3
Open access
Nafiye Helvaci Department of Internal Medicine, Division of Endocrinology and Metabolism, Hacettepe University School of Medicine, Ankara, Turkey

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Erdem Karabulut Department of Biostatistics, Hacettepe University School of Medicine, Ankara, Turkey

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Ahmet Ugur Demir Department of Chest Diseases, Hacettepe University School of Medicine, Ankara, Turkey

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Bulent Okan Yildiz Department of Internal Medicine, Division of Endocrinology and Metabolism, Hacettepe University School of Medicine, Ankara, Turkey

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fully understood, obesity, insulin resistance and hyperandrogenemia, which are the main features of PCOS, are all proposed to play a role in the development of OSA in patients with PCOS. The objective of this meta-analysis was to examine if there is an

Article Type:
Research Article
DOI:
https://doi.org/10.1530/EC-17-0129
Volume/Issue:
Volume 6: Issue 7
Open access
T Grimmichova Institute of Endocrinology, Narodni, Prague, Czech Republic
2nd Department of Internal Medicine, University Hospital Kralovske Vinohrady and Third Faculty of Medicine, Charles University in Prague, Srobarova, Prague, Czech Republic

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M Haluzik Institute of Endocrinology, Narodni, Prague, Czech Republic
Diabetes Centre, Institute for Clinical and Experimental Medicine (IKEM), Videnska, Prague, Czech Republic

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K Vondra Institute of Endocrinology, Narodni, Prague, Czech Republic

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P Matucha Institute of Endocrinology, Narodni, Prague, Czech Republic

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M Hill Institute of Endocrinology, Narodni, Prague, Czech Republic

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ultrasound-guided fine-needle aspiration ( 6 ). Finally, and the topic of our study, (8) type 2 diabetes (T2DM), obesity and insulin resistance (IR) in the framework of the worldwide epidemic of diabetes and obesity. In general, patients with diabetes have an

Article Type:
Research Article
DOI:
https://doi.org/10.1530/EC-20-0180
Volume/Issue:
Volume 9: Issue 7
Open access
Liza Haqq School of Science and Technology, University of New England, Armidale, New South Wales 2351, Australia

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James McFarlane School of Science and Technology, University of New England, Armidale, New South Wales 2351, Australia

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Gudrun Dieberg School of Science and Technology, University of New England, Armidale, New South Wales 2351, Australia

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Neil Smart School of Science and Technology, University of New England, Armidale, New South Wales 2351, Australia

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hyperandrogenism (clinical manifestations are hirsutism, android alopecia and acne), oligo/amenorrhoea (infrequent or no menstruation), polycystic ovaries and infertility or reduced fertility (3, 4) . Often women with PCOS are obese, which contributes to insulin

Article Type:
Research Article
DOI:
https://doi.org/10.1530/EC-14-0010
Volume/Issue:
Volume 3: Issue 1
Open access
Selina Mäkinen Minerva Foundation Institute for Medical Research, Helsinki, Finland
Department of Medicine, University of Helsinki, Helsinki University Central Hospital, Helsinki, Finland

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Neeta Datta Minerva Foundation Institute for Medical Research, Helsinki, Finland
Department of Medicine, University of Helsinki, Helsinki University Central Hospital, Helsinki, Finland

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Yen H Nguyen Minerva Foundation Institute for Medical Research, Helsinki, Finland
Department of Medicine, University of Helsinki, Helsinki University Central Hospital, Helsinki, Finland

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Petro Kyrylenko Minerva Foundation Institute for Medical Research, Helsinki, Finland
Department of Medicine, University of Helsinki, Helsinki University Central Hospital, Helsinki, Finland

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Markku Laakso Institute of Clinical Medicine, Internal Medicine, University of Eastern Finland, Kuopio, Finland

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Heikki A Koistinen Minerva Foundation Institute for Medical Research, Helsinki, Finland
Department of Medicine, University of Helsinki, Helsinki University Central Hospital, Helsinki, Finland

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simvastatin was most likely involved in muscular adverse effects ( 12 ). The mechanisms whereby statins affect insulin sensitivity in human muscle are unclear. Simvastatin-induced insulin resistance has been proposed to result from decreased lipid synthetic

Article Type:
Research Article
DOI:
https://doi.org/10.1530/EC-20-0444
Volume/Issue:
Volume 9: Issue 11
Open access
Selina Mäkinen Minerva Foundation Institute for Medical Research, Helsinki, Finland
University of Helsinki, Department of Medicine, and Abdominal Center, Endocrinology, Helsinki University Central Hospital, Helsinki, Finland

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Yen H Nguyen Minerva Foundation Institute for Medical Research, Helsinki, Finland
University of Helsinki, Department of Medicine, and Abdominal Center, Endocrinology, Helsinki University Central Hospital, Helsinki, Finland

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Paulina Skrobuk Minerva Foundation Institute for Medical Research, Helsinki, Finland
University of Helsinki, Department of Medicine, and Abdominal Center, Endocrinology, Helsinki University Central Hospital, Helsinki, Finland

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Heikki A Koistinen Minerva Foundation Institute for Medical Research, Helsinki, Finland
University of Helsinki, Department of Medicine, and Abdominal Center, Endocrinology, Helsinki University Central Hospital, Helsinki, Finland

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Introduction Excess dietary fat and sedentary lifestyle predispose to insulin resistance and type 2 diabetes. As skeletal muscle accounts for 80% of glucose disposal under insulin-stimulated conditions ( 1 , 2 ), it is an important target

Article Type:
Research Article
DOI:
https://doi.org/10.1530/EC-17-0039
Volume/Issue:
Volume 6: Issue 5
Open access
Nese Cinar Department of Endocrinology and Metabolism, Hacettepe University School of Medicine, 06100 Sihhiye, Ankara, Turkey

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Alper Gurlek Department of Endocrinology and Metabolism, Hacettepe University School of Medicine, 06100 Sihhiye, Ankara, Turkey

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ADP, vaspin, and visfatin in the context of thyroid dysfunction and the associated changes in adipose tissue and insulin resistance. The reason for choosing them is the increasing evidence regarding their changes in states of thyroid dysfunction

Article Type:
Review Article
DOI:
https://doi.org/10.1530/EC-13-0061
Volume/Issue:
Volume 2: Issue 4
Open access