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Department of Cell Biology and Anatomy, Department of Medicine, Turku PET Centre, Department of Radiology, Medical Imaging Centre of Southwest Finland, Department of Endocrinology, Abdominal Center: Endocrinology, Minerva Foundation Institute for Medical Research, Institute of Biomedicine, University of Turku, FI-20520 Turku, Finland
Department of Cell Biology and Anatomy, Department of Medicine, Turku PET Centre, Department of Radiology, Medical Imaging Centre of Southwest Finland, Department of Endocrinology, Abdominal Center: Endocrinology, Minerva Foundation Institute for Medical Research, Institute of Biomedicine, University of Turku, FI-20520 Turku, Finland
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Department of Cell Biology and Anatomy, Department of Medicine, Turku PET Centre, Department of Radiology, Medical Imaging Centre of Southwest Finland, Department of Endocrinology, Abdominal Center: Endocrinology, Minerva Foundation Institute for Medical Research, Institute of Biomedicine, University of Turku, FI-20520 Turku, Finland
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Department of Cell Biology and Anatomy, Department of Medicine, Turku PET Centre, Department of Radiology, Medical Imaging Centre of Southwest Finland, Department of Endocrinology, Abdominal Center: Endocrinology, Minerva Foundation Institute for Medical Research, Institute of Biomedicine, University of Turku, FI-20520 Turku, Finland
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Introduction Bone is increasingly recognized as an effector in energy metabolism via its interactions with other tissues (1, 2) . The role of bone-derived osteocalcin (OC) as a novel endocrine regulator of glucose and lipid metabolism was
Beijing Key Laboratory of Diabetes Research and Care, Beijing, China
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human energy homeostasis and may be involved in bone metabolism, which may contribute to the complex relationship between fat mass and bone ( 9 ). Although amounts of studies showed either a clear positive or negative effect of whole-body fat mass on
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various energy and glucose metabolism markers in muscle (29) , as well as similar hepatic mitochondrial and peroxisomal fatty acid oxidation capacity when compared with low-fat diet controls (30) . Besides evidence for scenario 1 (a decrease in
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Vazquez MJ Rodriguez-Cuenca S Gonzalez CR Velagapudi VR Morgan DA Schoenmakers E Agassandian K Lage R . Hypothalamic AMPK and fatty acid metabolism mediate thyroid regulation of energy balance . Nature Medicine 2010 16 1001
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increase and any single CSF measurement, indicating that a complex neuroendocrine network, rather than any single component, regulates human glucose and lipid metabolism and energy homeostasis. Alternatively, it may be due to the relatively small sample
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-Kocik, Michał Kloska and Paulina Ziółkowska contributed equally to this work. References 1 Choe SS Huh JY Hwang IJ Kim JI Kim JB Adipose tissue remodeling: its role in energy metabolism and metabolic disorders . Frontiers in Endocrinology
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) 10 Ekmekcioglu C Touitou Y. Chronobiological aspects of food intake and metabolism and their relevance on energy balance and weight regulation . Obesity Reviews 2011 12 14 – 25 . ( doi:10.1111/j.1467-789X.2010.00716.x ) 11 Scheer F
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FA oxidation, VLDL production and secretion, and gluconeogenesis. Thus, during the postprandial period, cellular metabolism rapidly shifts away from energy supply to energy storage (A) and back again. In individuals with an ‘unhealthy’ phenotype (e
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disorders . Journal of Clinical Endocrinology and Metabolism 2011 96 333 – 343 . ( https://doi.org/10.1210/jc.2009-2304 ) 32 Loucks AB . The response of luteinizing hormone pulsatility to 5 days of low energy availability disappears by 14 years of
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Hypothalamic Integration Mechanisms, Netherlands Institute for Neuroscience, Amsterdam, The Netherlands
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energy source. Although TH is known to regulate lipogenesis and lipolysis, there are no data available on the effects of fasting on TH metabolism in WAT. Thus, both liver and WAT contain the molecular machinery for TH metabolism and action. Previous