Department of Diabetes & Endocrinology, Skåne University Hospital, Malmö, Sweden
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Department of Diabetes & Endocrinology, Skåne University Hospital, Malmö, Sweden
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Department of Ophthalmology, Skåne University Hospital, Malmö, Sweden
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Department of Diabetes & Endocrinology, Skåne University Hospital, Malmö, Sweden
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subsequently activating adenylate cyclase. This activation leads to the generation of cyclic adenosine monophosphate (cAMP). Elevated levels of cAMP induce the proliferation of thyrocytes, thereby fostering thyroid growth and the secretion of thyroid hormones T
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Department of Clinical Biochemistry (KB3014), Department of Technology, Department of Clinical Biochemistry, Department of Biomedical Sciences, Department of Clinical Medicine, Rigshospitalet, University of Copenhagen, 9 Blegdamsvej, DK-2100 Copenhagen, Denmark
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Department of Clinical Biochemistry (KB3014), Department of Technology, Department of Clinical Biochemistry, Department of Biomedical Sciences, Department of Clinical Medicine, Rigshospitalet, University of Copenhagen, 9 Blegdamsvej, DK-2100 Copenhagen, Denmark
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role in overall energy homeostasis. β-adrenergic receptors on adipocytes are activated by circulating catecholamines that stimulate lipolysis in a cAMP-dependent manner. In parallel, natriuretic peptides cause the release of lipids to the circulation by
Department of Medicine, Department of Biomedical Sciences, Department of Endocrinology, Center for Diabetes Research, Gentofte Hospital, University of Copenhagen, Kildegårdsvej 28, DK-2900 Hellerup, Denmark
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Department of Medicine, Department of Biomedical Sciences, Department of Endocrinology, Center for Diabetes Research, Gentofte Hospital, University of Copenhagen, Kildegårdsvej 28, DK-2900 Hellerup, Denmark
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Department of Medicine, Department of Biomedical Sciences, Department of Endocrinology, Center for Diabetes Research, Gentofte Hospital, University of Copenhagen, Kildegårdsvej 28, DK-2900 Hellerup, Denmark
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Department of Medicine, Department of Biomedical Sciences, Department of Endocrinology, Center for Diabetes Research, Gentofte Hospital, University of Copenhagen, Kildegårdsvej 28, DK-2900 Hellerup, Denmark
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mediated through TGR5-induced intracellular cAMP formation and activation of the intracellular type 2 iodothyronine deiodinase (D2), which converts thyroxine (T 4 ) into the active thyroid hormone triiodothyronine (T 3 ). Up to date, human studies have
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10 U/L, and its corresponding sensitivity and specificity were 67.0 and 80.5%, respectively. Circulation TRAb is the hallmark of GD. By binding to the TSH receptor, TRAb enhances the production of cAMP in thyrocytes, thereby promoting the release of
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Department of Physiology, Faculty of Science, Charles University, Prague, Czech Republic
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Department of Physiology, Faculty of Science, Charles University, Prague, Czech Republic
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intestinal peptide, pituitary adenylate cyclase-activating peptide, and noradrenaline induce the transcription factors CCAAT/enhancer binding protein (C/EBP)-β and C/EBP-δ in mouse cortical astrocytes: involvement in cAMP-regulated glycogen metabolism
Research Center for Translational Medicine, Cancer Stem Cell Institute, East Hospital, Tongji University School of Medicine, Shanghai, People’s Republic of China
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-0154 ) 10.1210/me.2003-0154 36 Vandamme T Peeters M Dogan F Pauwels P Van Assche E Beyens M Mortier G Vandeweyer G de Herder W Van Camp G , et al . Whole-exome characterization of pancreatic neuroendocrine tumor cell lines BON-1 and QGP-1 . Journal of
Department of Immunology, Nanjing Medical University, Jiangsu, China
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Tatsuno I Suzuki K Kohn LD . Thyroglobulin (Tg) induces thyroid cell growth in a concentration-specific manner by a mechanism other than thyrotropin/cAMP stimulation . Biochemical and Biophysical Research Communications 2010 391 890 – 894 . ( https
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Department of Pathological Cytology and Anatomy, Foch Hospital, Paris, France
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Department of Clinical Medicine, Gansu University of Chinese Medicine, Lanzhou, Gansu, China
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coworkers found a high expression level of TSHR in adipocytes ( 31 ), with functions similar to those in the thyroid ( 32 ). The expression of TSHR in adipocytes resulted in TSH-stimulating cAMP production ( 33 ). Arpaci and coworkers showed that increased
Department of Laboratory Medicine, Radboud University Medical Centre, Nijmegen, The Netherlands
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Gulfo J Camps N Alcala R Monserrat L Moreno-Navarrete JM Ortega FJ Esteve M Remesar X Fernández-López JA , Modulation of SHBG binding to testosterone and estradiol by sex and morbid obesity . European Journal of Endocrinology 2017 176 393 – 404