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, and hypertension. High CgA plasma levels are strictly associated with mortality risk after myocardial infarction or acute coronary syndrome as well as heart failure while increased catestatin concentrations appear to improve post-ischemic recovery by
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previous myocardial infarction ( 25 ). Previous studies have indicated that QTc is associated with the risk of CVDs in different populations ( 26 , 27 , 28 , 29 ). Patients with severe PAD are at a high risk for CVDs, which might affect the QTc. Studies
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MMB . Uric acid is a risk factor for myocardial infarction and stroke: the Rotterdam study . Stroke 2006 37 1503 – 1507 . ( https://doi.org/10.1161/01.STR.0000221716.55088.d4 ) 14 Zhao G Huang L Song M & Song Y . Baseline serum uric acid
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. Comparison of therapeutic triiodothyronine versus metoprolol in the treatment of myocardial infarction in rats . Thyroid 2018 28 799 – 810 . ( https://doi.org/10.1089/thy.2017.0544 ) 56 Weltman NY Pol CJ Zhang Y Wang Y Koder A Raza S
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diabetes mellitus ( 29 ), alterations in lipid levels ( 22 ), atherosclerosis ( 30 ), and myocardial infarction ( 31 ). We also have performed some research on the mechanism of the effect of TSH on hyperlipidemia ( 32 ) and atherosclerosis ( 33 ). SCH can
Department of Performance and Image-enhancing Drugs Research, Android Health Clinic, Utrecht, the Netherlands
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Department of Internal Medicine, Spaarne Gasthuis, Haarlem, the Netherlands
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Department of Performance and Image-enhancing Drugs Research, Android Health Clinic, Utrecht, the Netherlands
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stroke (HR 1.27, 95% CI: 0.91–1.75), but an increased risk was found for myocardial infarction (HR 1.46, 95% CI: 1.06–2.00). After multivariable adjustment for age, sex, smoking, alcohol consumption, body mass index, the Charlson comorbidity index, and
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cardiovascular event was defined as a stroke (CVA or TIA), myocardial infarction, heart failure (NYHA 2-4), or revascularization procedure. After the diagnosis of DTC, 60 of the 619 DTC patients (9.7%) developed a first cardiovascular event within the follow
Endocrine Unit, Royal Victoria Infirmary, Newcastle upon Tyne, UK
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Endocrine Unit, Royal Victoria Infirmary, Newcastle upon Tyne, UK
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(prochymal) after acute myocardial infarction . Journal of the American College of Cardiology 2009 54 2277 – 2286 . ( https://doi.org/10.1016/j.jacc.2009.06.055 ) 19958962 10.1016/j.jacc.2009.06.055 42 Fiorina P Jurewicz M Augello A Vergani A
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Center for Clinical Metabolic Research, Gentofte Hospital, University of Copenhagen, Hellerup, Denmark
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Center for Clinical Metabolic Research, Gentofte Hospital, University of Copenhagen, Hellerup, Denmark
Department of Clinical Medicine, Faculty of Health and Medical Sciences, University of Copenhagen, Copenhagen, Denmark
Novo Nordisk Foundation Center for Basic Metabolic Research, Faculty of Health and Medical Sciences, University of Copenhagen, Copenhagen, Denmark
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Center for Clinical Metabolic Research, Gentofte Hospital, University of Copenhagen, Hellerup, Denmark
Department of Clinical Medicine, Faculty of Health and Medical Sciences, University of Copenhagen, Copenhagen, Denmark
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remaining six completed CVOTs for GLP-1RA are listed in Table 2 . In the majority of the trials, the primary composite CV endpoint was a three-component major adverse CV event (MACE) outcome including CV-related mortality, non-fatal myocardial infarction
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external factors such as sleep disruption, stress or a proinflammatory diet. The most severe consequences of atheromas involve acute events such as acute myocardial infarction or stroke, which usually occur due to rupture of the fibrous external layer of