Claus H Gravholt
Claus H Gravholt
Department of Endocrinology, Aarhus University Hospital, Aarhus, Denmark
Department of Molecular Medicine, Aarhus University Hospital, Aarhus, Denmark
Department of Clinical Medicine, Aarhus University, Aarhus, Denmark
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Department of Molecular Medicine, Aarhus University Hospital, Aarhus, Denmark
Department of Clinical Medicine, Aarhus University, Aarhus, Denmark
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Alberto Ferlin
Alberto Ferlin
Department of Medicine, Unit of Andrology and Reproductive Medicine, University of Padova, Padova, Italy
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Joerg Gromoll
Joerg Gromoll
Centre of Reproductive Medicine and Andrology, Münster, Germany
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Anders Juul
Anders Juul
Department of Growth and Reproduction Copenhagen University Hospital - Rigshospitalet, Copenhagen, Denmark
Department of Clinical Medicine, Faculty of Health Sciences, University of Copenhagen, Copenhagen, Denmark
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Department of Clinical Medicine, Faculty of Health Sciences, University of Copenhagen, Copenhagen, Denmark
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Armin Raznahan
Armin Raznahan
Section on Developmental Neurogenomics, National Institute of Mental Health Intramural Research Program, National Institutes of Health, Bethesda, Maryland, USA
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Sophie van Rijn
Sophie van Rijn
Clinical Neurodevelopmental Sciences, Leiden University, Leiden, The Netherlands and TRIXY Center of Expertise, Leiden University Treatment and Expertise Centre (LUBEC), Leiden, The Netherlands
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Alan D Rogol
Alan D Rogol
Department of Pediatrics, University of Virginia, Charlottesville, Virginia, USA
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Anne Skakkebæk
Anne Skakkebæk
Department of Molecular Medicine, Aarhus University Hospital, Aarhus, Denmark
Department of Clinical Medicine, Aarhus University, Aarhus, Denmark
Department of Clinical Genetics, Aarhus University Hospital, Aarhus, Denmark
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Department of Clinical Medicine, Aarhus University, Aarhus, Denmark
Department of Clinical Genetics, Aarhus University Hospital, Aarhus, Denmark
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Nicole Tartaglia
Nicole Tartaglia
Department of Pediatrics, Developmental Pediatrics, University of Colorado School of Medicine, Aurora, Colorado, USA
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Hanna Swaab
Hanna Swaab
Clinical Neurodevelopmental Sciences, Leiden University, Leiden, The Netherlands and TRIXY Center of Expertise, Leiden University Treatment and Expertise Centre (LUBEC), Leiden, The Netherlands
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other supernumerary sex chromosomal syndromes ( 6 , 7 , 8 , 9 , 10 ). In addition, it is now clear that the DNA methylation landscape is altered in a genome-wide fashion ( 6 , 9 , 10 , 11 ) and that indeed also the coding RNA transcriptome as well
Athanasios Zervas
Athanasios Zervas
Endocrine Unit, Athens Medical Centre, Athens, Greece
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George Chrousos
George Chrousos
Division of Endocrinology, Metabolism and Diabetes, First Department of Pediatrics, University Research Institute of Maternal and Child Health and Precision Medicine, and UNESCO Chair on Adolescent Health Care, National and Kapodistrian University of Athens, Athens, Greece
National and Kapodistrian University of Athens Medical School, ‘Aghia Sophia’ Children’s Hospital, Athens, Greece
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National and Kapodistrian University of Athens Medical School, ‘Aghia Sophia’ Children’s Hospital, Athens, Greece
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Sarantis Livadas
Sarantis Livadas
Endocrine Unit, Athens Medical Centre, Athens, Greece
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1A is a result of loss of maternal expression of the GNAS gene that encodes the subunit of the Gsα mediating the signal transduction of the PTH receptor, whereas PHP1B results from methylation defects of maternally derived GNAS differentially
Xiao-jun Zhou
Xiao-jun Zhou
Department of Endocrinology, Shandong Provincial Qianfoshan Hospital, Shandong University, Jinan, Shandong, China
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Lin Ding
Lin Ding
Department of Endocrinology, Shandong Provincial Qianfoshan Hospital, Shandong University, Jinan, Shandong, China
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Jia-xin Liu
Jia-xin Liu
Department of Endocrinology, Shandong Provincial Qianfoshan Hospital, Shandong University, Jinan, Shandong, China
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Le-qun Su
Le-qun Su
Department of Pharmacy, Shandong Provincial Qianfoshan Hospital, Shandong University, Jinan, Shandong, China
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Jian-jun Dong
Jian-jun Dong
Division of Endocrinology, Department of Medicine, Qilu Hospital of Shandong University, Shandong University, Jinan, Shandong, China
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Lin Liao
Lin Liao
Department of Endocrinology, Shandong Provincial Qianfoshan Hospital, Shandong University, Jinan, Shandong, China
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potentially linking obesity to the metabolic syndrome ( 21 ). Higher visceral adipose tissue DPP4 gene expression in non-diabetic severely obese men with metabolic syndrome was observed than non-metabolic syndrome. An association between the DPP-4 methylation
Filomena Cetani
Filomena Cetani
Department of Clinical and Experimental Medicine, Section of Pathology, Department of Surgical, Surgery Unit, Medicine, General Surgery 3 and Esophageal Surgery, Clinical and Biological Sciences, Department of Oncology
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Chiara Banti
Chiara Banti
Department of Clinical and Experimental Medicine, Section of Pathology, Department of Surgical, Surgery Unit, Medicine, General Surgery 3 and Esophageal Surgery, Clinical and Biological Sciences, Department of Oncology
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Elena Pardi
Elena Pardi
Department of Clinical and Experimental Medicine, Section of Pathology, Department of Surgical, Surgery Unit, Medicine, General Surgery 3 and Esophageal Surgery, Clinical and Biological Sciences, Department of Oncology
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Simona Borsari
Simona Borsari
Department of Clinical and Experimental Medicine, Section of Pathology, Department of Surgical, Surgery Unit, Medicine, General Surgery 3 and Esophageal Surgery, Clinical and Biological Sciences, Department of Oncology
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Paolo Viacava
Paolo Viacava
Department of Clinical and Experimental Medicine, Section of Pathology, Department of Surgical, Surgery Unit, Medicine, General Surgery 3 and Esophageal Surgery, Clinical and Biological Sciences, Department of Oncology
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Paolo Miccoli
Paolo Miccoli
Department of Clinical and Experimental Medicine, Section of Pathology, Department of Surgical, Surgery Unit, Medicine, General Surgery 3 and Esophageal Surgery, Clinical and Biological Sciences, Department of Oncology
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Liborio Torregrossa
Liborio Torregrossa
Department of Clinical and Experimental Medicine, Section of Pathology, Department of Surgical, Surgery Unit, Medicine, General Surgery 3 and Esophageal Surgery, Clinical and Biological Sciences, Department of Oncology
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Fulvio Basolo
Fulvio Basolo
Department of Clinical and Experimental Medicine, Section of Pathology, Department of Surgical, Surgery Unit, Medicine, General Surgery 3 and Esophageal Surgery, Clinical and Biological Sciences, Department of Oncology
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Maria Rosa Pelizzo
Maria Rosa Pelizzo
Department of Clinical and Experimental Medicine, Section of Pathology, Department of Surgical, Surgery Unit, Medicine, General Surgery 3 and Esophageal Surgery, Clinical and Biological Sciences, Department of Oncology
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Massimo Rugge
Massimo Rugge
Department of Clinical and Experimental Medicine, Section of Pathology, Department of Surgical, Surgery Unit, Medicine, General Surgery 3 and Esophageal Surgery, Clinical and Biological Sciences, Department of Oncology
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Gianmaria Pennelli
Gianmaria Pennelli
Department of Clinical and Experimental Medicine, Section of Pathology, Department of Surgical, Surgery Unit, Medicine, General Surgery 3 and Esophageal Surgery, Clinical and Biological Sciences, Department of Oncology
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Guido Gasparri
Guido Gasparri
Department of Clinical and Experimental Medicine, Section of Pathology, Department of Surgical, Surgery Unit, Medicine, General Surgery 3 and Esophageal Surgery, Clinical and Biological Sciences, Department of Oncology
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Mauro Papotti
Mauro Papotti
Department of Clinical and Experimental Medicine, Section of Pathology, Department of Surgical, Surgery Unit, Medicine, General Surgery 3 and Esophageal Surgery, Clinical and Biological Sciences, Department of Oncology
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Marco Volante
Marco Volante
Department of Clinical and Experimental Medicine, Section of Pathology, Department of Surgical, Surgery Unit, Medicine, General Surgery 3 and Esophageal Surgery, Clinical and Biological Sciences, Department of Oncology
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Edda Vignali
Edda Vignali
Department of Clinical and Experimental Medicine, Section of Pathology, Department of Surgical, Surgery Unit, Medicine, General Surgery 3 and Esophageal Surgery, Clinical and Biological Sciences, Department of Oncology
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Federica Saponaro
Federica Saponaro
Department of Clinical and Experimental Medicine, Section of Pathology, Department of Surgical, Surgery Unit, Medicine, General Surgery 3 and Esophageal Surgery, Clinical and Biological Sciences, Department of Oncology
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Claudio Marcocci
Claudio Marcocci
Department of Clinical and Experimental Medicine, Section of Pathology, Department of Surgical, Surgery Unit, Medicine, General Surgery 3 and Esophageal Surgery, Clinical and Biological Sciences, Department of Oncology
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methylation (28, 29) , or other yet unknown predisposing genes might be involved. CDC73 mutations may impair the expression of parafibromin and its focal/global loss at, as determined by immunohistochemistry, was reported in up to 100% of cases (8, 9, 10
Christin Krause
Christin Krause
Epigenetics & Metabolism, Medical Department I, University of Lübeck, Lübeck, Germany
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Martina Grohs
Martina Grohs
Epigenetics & Metabolism, Medical Department I, University of Lübeck, Lübeck, Germany
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Alexander T El Gammal
Alexander T El Gammal
Department of General, Visceral and Thoracic Surgery, University Medical Center Hamburg-Eppendorf, Hamburg, Germany
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Stefan Wolter
Stefan Wolter
Department of General, Visceral and Thoracic Surgery, University Medical Center Hamburg-Eppendorf, Hamburg, Germany
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Hendrik Lehnert
Hendrik Lehnert
Medical Department I, University of Lübeck, Lübeck, Germany
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Oliver Mann
Oliver Mann
Department of General, Visceral and Thoracic Surgery, University Medical Center Hamburg-Eppendorf, Hamburg, Germany
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Jens Mittag
Jens Mittag
Molecular Endocrinology, Medical Department I, University of Lübeck, Lübeck, Germany
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Henriette Kirchner
Henriette Kirchner
Epigenetics & Metabolism, Medical Department I, University of Lübeck, Lübeck, Germany
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driven by changes in promoter methylation ( 38 ). Consequently, our data on decreased hepatic THRB and DIO1 expression are of explicit biological relevance for the understanding of TH economy in the aging liver, as they suggest that the liver might
Liza Das
Liza Das
Department of Endocrinology, Postgraduate Institute of Medical Education and Research, (PGIMER), Chandigarh, India
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Kim Vaiphei
Kim Vaiphei
Department of Histopathology, PGIMER, Chandigarh, India
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Ashutosh Rai
Ashutosh Rai
Department of Translational and Regenerative Medicine, PGIMER, Chandigarh, India
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Chirag Kamal Ahuja
Chirag Kamal Ahuja
Department of Radiology, PGIMER, Chandigarh, India
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Paramjeet Singh
Paramjeet Singh
Department of Radiology, PGIMER, Chandigarh, India
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Ishani Mohapatra
Ishani Mohapatra
Department of Pathology and Laboratory Medicine, Medanta, The Medicity, Gurgaon, India
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Rajesh Chhabra
Rajesh Chhabra
Department of Neurosurgery, PGIMER, Chandigarh, India
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Anil Bhansali
Anil Bhansali
Department of Endocrinology, Postgraduate Institute of Medical Education and Research, (PGIMER), Chandigarh, India
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Bishan Dass Radotra
Bishan Dass Radotra
Department of Histopathology, PGIMER, Chandigarh, India
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Ashley B Grossman
Ashley B Grossman
Centre for Endocrinology, William Harvey Research Institute, Barts and The London School of Medicine and Dentistry, Queen Mary University of London, London, UK
Green Templeton College, University of Oxford, Oxford, UK
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Green Templeton College, University of Oxford, Oxford, UK
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Márta Korbonits
Márta Korbonits
Centre for Endocrinology, William Harvey Research Institute, Barts and The London School of Medicine and Dentistry, Queen Mary University of London, London, UK
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Pinaki Dutta
Pinaki Dutta
Department of Endocrinology, Postgraduate Institute of Medical Education and Research, (PGIMER), Chandigarh, India
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oncocytomas (SCO) and granular cell tumours (GCT) are the different subtypes of PPTs ( 3 ) showing close clustering in methylation-based classification studies, suggesting they may have shared histogenesis, yet distinctive morphology ( 11 , 12 , 13 , 14
Kjell Oberg
Kjell Oberg
Uppsala University, Uppsala, Sweden
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Eric Krenning
Eric Krenning
Erasmus Medical Center, Rotterdam, Netherlands
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Anders Sundin
Anders Sundin
Uppsala University, Uppsala, Sweden
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Lisa Bodei
Lisa Bodei
Memorial Sloan Kettering Cancer Center, New York, New York, USA
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Mark Kidd
Mark Kidd
Wren Laboratories, Branford, Connecticut, USA
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Margot Tesselaar
Margot Tesselaar
Netherlands Cancer Institute, Amsterdam, Netherlands
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Valentina Ambrosini
Valentina Ambrosini
University of Bologna, Bologna, Italy
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Richard P Baum
Richard P Baum
Zentralklinik Bad Berka, Bad Berka, Germany
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Matthew Kulke
Matthew Kulke
Dana Farber Cancer Institute, Boston, Massachusetts, USA
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Marianne Pavel
Marianne Pavel
Charite Hospital, Berlin, Germany
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Jaroslaw Cwikla
Jaroslaw Cwikla
University of Warmia and Mazury, Olsztyn, Poland
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Ignat Drozdov
Ignat Drozdov
Wren Laboratories, Branford, Connecticut, USA
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Massimo Falconi
Massimo Falconi
Ospedale San Raffaele, Milan, Italy
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Nicola Fazio
Nicola Fazio
IEO (European Institute of Oncology), Milan, Italy
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Andrea Frilling
Andrea Frilling
Imperial College London, London, UK
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Robert Jensen
Robert Jensen
National Institutes of Health, Bethesda, Maryland, USA
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Klaus Koopmans
Klaus Koopmans
Martini Ziekenhuis, Groningen, Netherlands
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Tiny Korse
Tiny Korse
Netherlands Cancer Institute, Amsterdam, Netherlands
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Dik Kwekkeboom
Dik Kwekkeboom
Erasmus Medical Center, Rotterdam, Netherlands
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Helmut Maecke
Helmut Maecke
University Hospital Freiburg, Freiburg, Germany
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Giovanni Paganelli
Giovanni Paganelli
Instituto Scientifico Romagnolo per lo Studio e la Cura dei Tumori, Meldola, Italy
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Ramon Salazar
Ramon Salazar
Instituto Catala d’Oncologia, Barcelona, Spain
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Stefano Severi
Stefano Severi
Instituto Scientifico Romagnolo per lo Studio e la Cura dei Tumori, Meldola, Italy
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Jonathan Strosberg
Jonathan Strosberg
H. Lee Moffitt Cancer Center, Tampa, Florida, USA
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Vikas Prasad
Vikas Prasad
Charite Hospital, Berlin, Germany
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Aldo Scarpa
Aldo Scarpa
University of Verona, Verona, Italy
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Ashley Grossman
Ashley Grossman
Univeristy of Oxford, Oxford, UK
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Annemeik Walenkamp
Annemeik Walenkamp
University of Groningen, Groningen, Netherlands
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Mauro Cives
Mauro Cives
H. Lee Moffitt Cancer Center, Tampa, Florida, USA
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Irene Virgolini
Irene Virgolini
Medical University Innsbruck, Innsbruck, Austria
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Andreas Kjaer
Andreas Kjaer
Copenhagen University, Copenhagen, Denmark
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Irvin M Modlin
Irvin M Modlin
Yale University, New Haven, Connecticut, USA
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) syndrome (germline MEN-1 mutation), the type of menin mutation was not considered to be of prognostic significance. Likewise, alterations in methylation patterns were not considered clinically useful, whereas O6-methylguanine DNA transferase deficiency was
Christian Høst
Christian Høst
Department of Endocrinology and Internal Medicine and the Medical Research Laboratories, Clinical Institute, Aarhus University Hospital, Aarhus N, Denmark
Department of Pediatrics, Aarhus University Hospital, Aarhus N, Denmark
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Department of Pediatrics, Aarhus University Hospital, Aarhus N, Denmark
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Anders Bojesen
Anders Bojesen
Department of Clinical Genetics, Aarhus University Hospital, Aarhus N, Denmark
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Mogens Erlandsen
Mogens Erlandsen
Section for Biostatistics, Department of Public Health, Aarhus University, Aarhus C, Denmark
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Kristian A Groth
Kristian A Groth
Department of Molecular Medicine, Aarhus University Hospital, Aarhus N, Denmark
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Kurt Kristensen
Kurt Kristensen
Department of Pediatrics, Aarhus University Hospital, Aarhus N, Denmark
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Anne Grethe Jurik
Anne Grethe Jurik
Department of Radiology, Aarhus University Hospital, Aarhus N, Denmark
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Niels H Birkebæk
Niels H Birkebæk
Department of Pediatrics, Aarhus University Hospital, Aarhus N, Denmark
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Claus H Gravholt
Claus H Gravholt
Department of Endocrinology and Internal Medicine and the Medical Research Laboratories, Clinical Institute, Aarhus University Hospital, Aarhus N, Denmark
Department of Molecular Medicine, Aarhus University Hospital, Aarhus N, Denmark
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Department of Molecular Medicine, Aarhus University Hospital, Aarhus N, Denmark
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resistance in KS ( 19 ). Recently, we found profound changes in the methylation and RNA expression pattern throughout the genome among males with KS and enrichment analyses showed that the observed methylation changes was related to development of diabetes
Ishita Gupta
Ishita Gupta
Department of Genetics, College of Medicine and Health Sciences, Sultan Qaboos University, Alkoudh, Sultanate of Oman
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Allal Ouhtit
Allal Ouhtit
Department of Biological and Environmental Sciences, College of Arts and Sciences, Qatar University, Doha, Qatar
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Adil Al-Ajmi
Adil Al-Ajmi
Department of Surgery, College of Medicine and Health Sciences, Sultan Qaboos University, Alkoudh, Sultanate of Oman
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Syed Gauhar A Rizvi
Syed Gauhar A Rizvi
Department of Family Medicine and Public Health, College of Medicine and Health Sciences, Sultan Qaboos University, Alkoudh, Sultanate of Oman
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Hamad Al-Riyami
Hamad Al-Riyami
Department of Genetics, College of Medicine and Health Sciences, Sultan Qaboos University, Alkoudh, Sultanate of Oman
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Marwa Al-Riyami
Marwa Al-Riyami
Department of Pathology, College of Medicine and Health Sciences, Sultan Qaboos University, Alkoudh, Sultanate of Oman
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Yahya Tamimi
Yahya Tamimi
Department of Biochemistry, College of Medicine and Health Sciences, Sultan Qaboos University, Alkoudh, Sultanate of Oman
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11 Alkam Y . Protein expression and methylation of DNA repair genes hMLH1, hMSH2, MGMT and BRCA1 and their correlation with clinicopathological parameters and prognosis in basal-like breast cancer . Histopathology 2013 63 713 – 725 . ( https
Xiaomin Li
Xiaomin Li
Department of Breast Surgery, West China Hospital, Sichuan University, Chengdu, China
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Ling Fang
Ling Fang
Department of Breast Surgery, Cheng Du Shang Jin Nan Fu Hospital, West China Hospital, Sichuan University, Chengdu, China
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Hongjiang Li
Hongjiang Li
Department of Breast Surgery, West China Hospital, Sichuan University, Chengdu, China
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Xiaoqin Yang
Xiaoqin Yang
Department of Breast Surgery, West China Hospital, Sichuan University, Chengdu, China
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comprehensive review, 4-OHE2 and 16α-OHE1 are potential oncogenic molecular markers, 2-OHE1 and 2-OHE2 are primary estrogen metabolites, and estrogen methylation products, such as 2-MeOE1 and 4-MeOE1, are potential tumor suppressor molecular markers ( 15 , 32
