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Amalie R Lanng, Lærke S Gasbjerg, Natasha C Bergmann, Sigrid Bergmann, Mads M Helsted, Matthew P Gillum, Bolette Hartmann, Jens J Holst, Tina Vilsbøll and Filip K Knop

may cause hepatic steatosis which in some individuals leads to insulin resistance and impaired glucose tolerance ( 4 , 5 ). However, the exact mechanisms underlying these pathophysiological processes have not been identified. Alcohol has been found

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Taísa A R Vicente, Ívina E S Rocha, Roberto Salvatori, Carla R P Oliveira, Rossana M C Pereira, Anita H O Souza, Viviane C Campos, Elenilde G Santos, Rachel D C Araújo Diniz, Eugênia H O Valença, Carlos C Epitácio-Pereira, Mario C P Oliveira, Andrea Mari and Manuel H Aguiar-Oliveira

measured by homeostatic model assessment of insulin resistance (HOMA–IR) – despite increased fat mass percentage (6) . In these subjects, βCF – assessed by the insulin response during an oral glucose tolerance test (OGTT) – also seems to be impaired

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Yao Chen and Shu-ying Fang

, insulin resistance, ovulatory dysfunction and polycystic ovaries. The prevalence of PCOS may be as high as 15–20% of women of reproductive age ( 3 ). Although it was first reported in 1935 by Stein et al ., its etiology is still unclear. PCOS has a

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Gaëtan Prévost, Marie Picot, Marie-Anne Le Solliec, Arnaud Arabo, Hind Berrahmoune, Mouna El Mehdi, Saloua Cherifi, Alexandre Benani, Emmanuelle Nédélec, Françoise Gobet, Valéry Brunel, Jérôme Leprince, Hervé Lefebvre, Youssef Anouar and Nicolas Chartrel

(LX20 Beckman Coulter) and plasma insulin was quantified using an Elecys for automated insulin Assay (COBAS 6000CE, Roche Diagnostics). Usual insulin resistance and insulin secretion indices were calculated at fasting levels and during the oral

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Dorte Glintborg, Magda Lambaa Altinok, Pernille Ravn, Kurt Bjerregaard Stage, Kurt Højlund and Marianne Andersen

Introduction Polycystic ovary syndrome (PCOS) is a common endocrine disorder characterized by insulin resistance and hyperandrogenism ( 1 ). Central obesity is closely associated with insulin resistance and low-grade inflammation ( 2 ) and

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Trevor Lewis, Eva Zeisig and Jamie E Gaida

and associated metabolic conditions, such as insulin resistance ( 5 ), diabetes ( 6 ), hypercholesterolaemia ( 7 ), statins ( 8 ), abdominal fat ( 9 ), the sympathetic nervous system ( 10 , 11 ), and corticosteroid use ( 12 ). Glucocorticoids are a

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Yael Sofer, Nava Nevo, Michal Vechoropoulos, Gabi Shefer, Etty Osher, Nathan Landis, Karen Tordjman, Geoffrey L Hammond and Naftali Stern

of developing diabetes ( 7 ). High SHBG levels are apparently also associated with lower incidence of insulin resistance in men ( 10 ). Furthermore, cross-sectional studies in non-diabetic men and women have shown an inverse relationship between SHBG

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Nikolaj Rittig, Mads Svart, Niels Jessen, Niels Møller, Holger J Møller and Henning Grønbæk

as obesity, sepsis, insulin resistance, diabetes mellitus type 2 (DM2) and non-alcoholic fatty liver disease (NAFLD) ( 1 , 2 , 3 , 4 , 5 , 6 ). These diseases are also associated with changes in the lipid metabolism, accelerated lipolysis and

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Clarissa Souza Barthem, Camila Lüdke Rossetti, Denise P Carvalho and Wagner Seixas da-Silva

against fatty liver and may improve pathway-selective insulin resistance . Diabetes 2013 424 – 434 . ( ) 14 Park SB Lee YJ Chung CK . Bone mineral density changes after ovariectomy in rats as an osteopenic

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Angelo Maria Patti, Kalliopi Pafili, Nikolaos Papanas and Manfredi Rizzo

fibrinogen, together with lower HDL-C levels ( 9 ). Insulin resistance and central adiposity are of crucial importance in the development of MetS, and they appear to correlate with cardiovascular (CV) risk factors, including hypertension, atherogenic