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-reperfusion injury through disrupting mitochondrial quality control . Redox Biology 2021 38 101777. ( https://doi.org/10.1016/j.redox.2020.101777 ) 65 Zhou H Ren J Toan S Mui D . Role of mitochondrial quality surveillance in myocardial infarction: from
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dysfunction in rats post myocardial infarction . Journal of Physiology 2014 592 3273 – 3286. ( doi:10.1113/jphysiol.2014.276584 ) 10.1113/jphysiol.2014.276584 126 Lal A Veinot JP Leenen FHH. Critical role of CNS effects of aldosterone in
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member ST2 predict mortality and clinical outcome in acute myocardial infarction . Circulation 2004 109 2186 – 2190 . ( https://doi.org/10.1161/01.CIR.0000127958.21003.5A ) 14 Tune JD Goodwill AG Sassoon DJ Mather KJ Cardiovascular
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Kim CJ Cho MC , et al . Low-density lipoprotein cholesterol level in patients with acute myocardial infarction having percutaneous coronary intervention (the cholesterol paradox) . American Journal of Cardiology 2010 106 1061 – 1068 . ( https
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to myocardial cryoinjury and juvenile zebrafish with no prior cardiac injury both experienced a global suppression of CM proliferation following acute daily exposures to crowding ( 46 ). In regards to non-CMs, exogenous glucocorticoid administration
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0 (0) 2 0 Elderly age (≥ 70 years) 0 (0) 1 0 Heart and/or respiratory failure 3 (33) 1 3 Acute myocardial infarction or ischemic stroke 0 (0) 1 0 Acute infection and/or rheumatological disorder 3 (33
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Department of Respiratory, Shandong Provincial Qianfoshan Hospital, Shandong University, The First Affiliated Hospital of Shandong First Medical University, Shandong Institute of Respiratory Diseases, Jinan, China
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Department of Respiratory, Shandong Provincial Qianfoshan Hospital, Shandong University, The First Affiliated Hospital of Shandong First Medical University, Shandong Institute of Respiratory Diseases, Jinan, China
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– 1174 . ( https://doi.org/10.1172/JCI13505 ) 43 Calvert JW Gundewar S Jha S Greer JJ Bestermann WH Tian R Lefer DJ . Acute metformin therapy confers cardioprotection against myocardial infarction via AMPK-eNOS-mediated signaling
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and nondiabetic patients, including those with multiple cardiovascular and cerebrovascular diseases, such as myocardial infarction and stroke. Animal experiments and clinical studies have demonstrated that vascular calcification and arteriosclerosis
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found to mirror cortisol levels in response to stress, myocardial infarction, and other acute illnesses, and copeptin has been proposed as a prognostic marker in such cases ( 39 ). AVP is known to potentiate corticotropin-releasing hormone (CRH
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occlusion, carotid endarterectomy; b ischemic heart disease, acute myocardial infarction, percutaneous transluminal coronary angioplasty, coronary artery bypass graft; c composite variable of atherosclerotic cerebrovascular and cardiovascular disease; d