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H A Booij, W D C Gaykema, K A J Kuijpers, M J M Pouwels and H M den Hertog

in the pathogenesis of PSF, it might be a target for treatment of PSF. In this review, we aim to give an overview of the literature on the prevalence and possible predictors of PD in stroke and the association with fatigue. We also searched for

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Ali Abbara, Sophie Clarke, Pei Chia Eng, James Milburn, Devavrata Joshi, Alexander N Comninos, Rozana Ramli, Amrish Mehta, Brynmor Jones, Florian Wernig, Ramesh Nair, Nigel Mendoza, Amir H Sam, Emma Hatfield, Karim Meeran, Waljit S Dhillo and Niamh M Martin

apoplexy presenting to a tertiary endocrine and neurosurgical centre. We reviewed clinical records of patients presenting with pituitary apoplexy to Imperial College Healthcare NHS Trust between 1991 and 2015 and describe presenting clinical features

Open access

Efstratios Kardalas, Stavroula A Paschou, Panagiotis Anagnostis, Giovanna Muscogiuri, Gerasimos Siasos and Andromachi Vryonidou

other clinical entities. The aim of this comprehensive review is to provide current knowledge regarding definition, prevalence and etiology of hypokalemia, as well as, an individualized guide for the optimal diagnostic management and follow-up strategy

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Wafaa M Rashed, Anas Saad, Muneer Al-Husseini, Ahmed Mahmoud Galal, Assem Mohamed Ismael, Ahmed M Al-Tayep, Ayman El Shafie, Mahmoud Ahmed Ali and Ahmad Samir Alfaar

million (2765 patients) with bimodal distribution of 4.0 per million in the age group 0–19 years, 1.3 in the group 20–49 years and 5.0 in the age group older than 50 years. Data of a total of 2,887,468 persons with cancer were reviewed, 117 of whom had

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Jennifer K Y Ko, Thomas F J King, Louise Williams, Sarah M Creighton and Gerard S Conway

gonadectomy and in the absence of guidelines on the most appropriate HRT for this group of women, our clinic philosophy has been to offer choice and follow individual preference. Here, we reviewed our experience on HRT choices made by women with CAIS

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Luminita Nicoleta Cima, Anca Colita and Simona Fica

chronic graft vs host disease implies a risk of consequent gonadotoxicity ( 8 , 9 , 10 ). A review of the literature on gonadal toxicity of HSCT clearly stated that the risk of ovarian damage and POF is very high in HSCT recipients ( 11 ). Gonadal damage

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Emma Jernberg, Anders Bergh and Pernilla Wikström

activation by other factors, as extensively reviewed previously ( 6 , 7 ). Thus, the AR has for a long time been the main target in the treatment of advanced PC, and the combination of castration therapy with 1st-generation antiandrogens (flutamide

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Ann-Kristin Picke, Graeme Campbell, Nicola Napoli, Lorenz C Hofbauer and Martina Rauner

mechanisms, which may differently affect bone metabolism. In both cases, the underlying mechanisms of poor bone strength are not well understood. Considering that T2DM accounts for the majority of diabetes cases (>90%), this review will focus on summarizing

Open access

Huguette S Brink, Aart Jan van der Lely and Joke van der Linden

GD pathogenesis. Combining biomarkers and risk factors into a predictive model may add to early prediction of GD, evoke effective prevention strategies and may ultimately reduce complications associated with GD. The aim of this review is to ( 1

Open access

Carolina Inda, Natalia G Armando, Paula A dos Santos Claro and Susana Silberstein

been implicated in CRHR functions, including ERK1/2 and p38 MAPKs, PKA, Akt, NFkB, GSK-3b/Wnt, RhoA as well as other important signaling molecules such as cAMP, calcium, IP3 and nitric oxide/cGMP ( 10 ). Here, we review the principal characteristics