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Xingyao Tang Beijing Tongren Hospital, Capital Medical University

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Zhi-Hui Song Department of Pharmacy, Beijing Tongren Hospital, Capital Medical University, Beijing, China

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Dawei Wang General Practice Department, Beijing Tongren Hospital, Capital Medical University, Beijing, China

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Jinkui Yang Department of Endocrinology, Beijing Tongren Hospital, Capital Medical University, Beijing, China

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Marly Augusto Cardoso Department of Nutrition, School of Public Health, University of Sao Paulo, Sao Paulo, Brazil

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Jian-Bo Zhou Department of Endocrinology, Beijing Tongren Hospital, Capital Medical University, Beijing, China

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Rafael Simó Endocrinology and Nutrition Department, Hospital Universitari Vall d’Hebron, Diabetes and Metabolism Research Unit, Vall d’Hebron Institut de Recerca (VHIR), Universitat Autònoma de Barcelona, Barcelona, Spain
Centro de Investigación Biomédica en Red de Diabetes y Enfermedades Metabólicas Asociadas (CIBERDEM), Instituto de Salud Carlos III (ISCIII), Madrid, Spain

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factor for dementia, promotes neurocognitive function and regulation metabolic processes ( 4 ). Thyroid dysfunction, including clinical hyperthyroidism, subclinical hyperthyroidism, subclinical hypothyroidism, and clinical hypothyroidism, has been

Article Type:
Research Article
DOI:
https://doi.org/10.1530/EC-21-0047
Volume/Issue:
Volume 10: Issue 4
Open access
Nadia Sawicka-Gutaj Department of Endocrinology, Metabolism and Internal Medicine, Poznan University of Medical Sciences, Poznan, Poland

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Ariadna Zybek-Kocik Department of Endocrinology, Metabolism and Internal Medicine, Poznan University of Medical Sciences, Poznan, Poland

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Michał Kloska Lehigh Valley Health Network, Department of Medicine, Lehigh Valley Hospital – Cedar Crest, Allentown, USA

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Paulina Ziółkowska Department of Endocrinology, Metabolism and Internal Medicine, Poznan University of Medical Sciences, Poznan, Poland

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Agata Czarnywojtek Department of Pharmacology, Poznan University of Medical Sciences, Poznan, Poland

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Jerzy Sowiński Department of Endocrinology, Metabolism and Internal Medicine, Poznan University of Medical Sciences, Poznan, Poland

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Dorota Mańkowska-Wierzbicka Department of Gastroenterology, Internal Medicine, Metabolic Diseases and Dietetics, Poznan University of Medical Sciences, Poznan, Poland

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Marek Ruchała Department of Endocrinology, Metabolism and Internal Medicine, Poznan University of Medical Sciences, Poznan, Poland

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, metabolic rate, food intake and energy expenditure ( 6 ). Thus, both hypo- and hyperthyroidism significantly alter these processes, resulting in significant changes in body composition ( 7 , 8 ). Moreover, many thyroid disorders are associated with chronic

Article Type:
Research Article
DOI:
https://doi.org/10.1530/EC-21-0059
Volume/Issue:
Volume 10: Issue 4
Open access
Flavia Letícia Martins Peçanha Instituto de Bioquímica Médica Leopoldo de Meis, Laboratório de Adaptações Metabólicas, Programa de Bioquímica e Biofísica Celular, Universidade Federal do Rio de Janeiro, Cidade Universitária, Rio de Janeiro, Rio de Janeiro, Brazil

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Reinaldo Sousa dos Santos Instituto de Bioquímica Médica Leopoldo de Meis, Laboratório de Adaptações Metabólicas, Programa de Bioquímica e Biofísica Celular, Universidade Federal do Rio de Janeiro, Cidade Universitária, Rio de Janeiro, Rio de Janeiro, Brazil

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Wagner Seixas da-Silva Instituto de Bioquímica Médica Leopoldo de Meis, Laboratório de Adaptações Metabólicas, Programa de Bioquímica e Biofísica Celular, Universidade Federal do Rio de Janeiro, Cidade Universitária, Rio de Janeiro, Rio de Janeiro, Brazil

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hexokinase (HK) ( 9 , 10 ). The increase in glucose uptake observed in models of hyperthyroidism has been studied by many research groups, with a particular focus on the effects of THs on GLUT expression ( 11 , 12 , 13 , 14 ). However, little is known

Article Type:
Research Article
DOI:
https://doi.org/10.1530/EC-17-0059
Volume/Issue:
Volume 6: Issue 5
Open access
L E Zijlstra Department of Cardiology, Leiden University Medical Center, Leiden, The Netherlands

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D M van Velzen Department of Internal Medicine, Section of Endocrinology, Northwest Clinics, Alkmaar, The Netherlands

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S Simsek Department of Internal Medicine, Section of Endocrinology, Northwest Clinics, Alkmaar, The Netherlands

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S P Mooijaart Department of Internal Medicine, Section of Gerontology and Geriatrics, Leiden University Medical Center, Leiden, The Netherlands

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M van Buren Department of Nephrology, Leiden University Medical Center, Leiden, The Netherlands
Department of Internal Medicine, HagaHospital, The Hague, The Netherlands

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D J Stott Institute of Cardiovascular and Medical Sciences, College of Medical, Veterinary and Life Sciences, University of Glasgow, Glasgow, UK

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I Ford Robertson Centre for Biostatistics, University of Glasgow, Glasgow, UK

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J W Jukema Department of Cardiology, Leiden University Medical Center, Leiden, The Netherlands

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S Trompet Department of Cardiology, Leiden University Medical Center, Leiden, The Netherlands
Department of Internal Medicine, Section of Gerontology and Geriatrics, Leiden University Medical Center, Leiden, The Netherlands

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Introduction Subclinical hyperthyroidism has been linked to atrial fibrillation and coronary artery calcification, whereas subclinical hypothyroidism has been associated with hypercholesterolemia and atherosclerosis ( 1 , 2 ). Despite these

Article Type:
Research Article
DOI:
https://doi.org/10.1530/EC-19-0502
Volume/Issue:
Volume 9: Issue 1
Open access
Nese Cinar Department of Endocrinology and Metabolism, Hacettepe University School of Medicine, 06100 Sihhiye, Ankara, Turkey

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Alper Gurlek Department of Endocrinology and Metabolism, Hacettepe University School of Medicine, 06100 Sihhiye, Ankara, Turkey

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are associated with insulin resistance (17) , and a relationship between ADP and thyroid hormones may exist via either direct or indirect interactions between them. Controversial results are reported for the experimental studies on hypo/hyperthyroid

Article Type:
Review Article
DOI:
https://doi.org/10.1530/EC-13-0061
Volume/Issue:
Volume 2: Issue 4
Open access
Caiyan Mo Department of Endocrinology, Beijing Tiantan Hospital, Capital Medical University, Beijing, China

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Tao Tong Department of Endocrinology, Beijing Tiantan Hospital, Capital Medical University, Beijing, China

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Ying Guo Department of Endocrinology, Beijing Tiantan Hospital, Capital Medical University, Beijing, China

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Zheng Li Department of Endocrinology, Beijing Tiantan Hospital, Capital Medical University, Beijing, China

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Liyong Zhong Department of Endocrinology, Beijing Tiantan Hospital, Capital Medical University, Beijing, China

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population ( 2 ). Graves' disease (GD) is an autoimmune disease of the thyroid gland and is the most common cause of hyperthyroidism. The incidence is 20–50 cases per 100,000 people, with a peak between the ages of 30 and 50. The binding of thyroid

Article Type:
Research Article
Collections:
Thyroid
DOI:
https://doi.org/10.1530/EC-23-0439
Volume/Issue:
Volume 13: Issue 4
Open access
Anna Olsson-Brown Department of Molecular and Clinical Pharmacology, University of Liverpool, Liverpool, UK
The Clatterbridge Cancer Centre, Wirral, UK

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Rosemary Lord The Clatterbridge Cancer Centre, Wirral, UK

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Joseph Sacco The Clatterbridge Cancer Centre, Wirral, UK
Molecular and Clinical Cancer Medicine, University of Liverpool, Liverpool, UK

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Jonathan Wagg Roche Innovation Center, Basel, Switzerland

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Mark Coles Kennedy Institute of Rheumatology, University of Oxford, Oxford, UK

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Munir Pirmohamed Department of Molecular and Clinical Pharmacology, University of Liverpool, Liverpool, UK

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clinical trials included both hyperthyroidism and hypothyroidism (Supplementary Table 1, see section on supplementary materials given at the end of this article) but the clinical and biochemical manifestations were not detailed. Radioisotope scanning of

Article Type:
Research Article
DOI:
https://doi.org/10.1530/EC-19-0473
Volume/Issue:
Volume 9: Issue 4
Open access
Waleed K W Al-Badri Orbital center Amsterdam, Department of Ophthalmology, Academic Medical Centre, University of Amsterdam, Amsterdam, the Netherlands

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Hinke Marijke Jellema Orbital center Amsterdam, Department of Ophthalmology, Academic Medical Centre, University of Amsterdam, Amsterdam, the Netherlands

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Arnaud R G G Potvin Orbital center Amsterdam, Department of Ophthalmology, Academic Medical Centre, University of Amsterdam, Amsterdam, the Netherlands

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Ruth M A van Nispen Amsterdam University Medical Center, Vrije Universiteit, Department of Ophthalmology, Amsterdam Public Health research institute, Amsterdam, the Netherlands

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Peter H Bisschop Department of Endocrinology, Academic Medical Centre, University of Amsterdam, Amsterdam, the Netherlands

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Peerooz Saeed Orbital center Amsterdam, Department of Ophthalmology, Academic Medical Centre, University of Amsterdam, Amsterdam, the Netherlands

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hyperthyroidism, most frequently caused by Graves’ disease (GD), 10% of GO patients are hypothyroid, and 5% are euthyroid ( 2 ). Among GD patients, 20–50% develop ophthalmopathy, making GO the most prominent extrathyroidal manifestation of GD ( 3 ). Besides

Article Type:
Review Article
Collections:
Thyroid
DOI:
https://doi.org/10.1530/EC-24-0259
Volume/Issue:
Volume 13: Issue 9
Open access
Jaafar Jaafar Division of Endocrinology and Diabetology, Geneva University Hospitals and Faculty of Medicine, Geneva, Switzerland

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Eugenio Fernandez Department of Oncology, Geneva University Hospitals and Faculty of Medicine, Geneva, Switzerland

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Heba Alwan Division of Endocrinology and Diabetology, Geneva University Hospitals and Faculty of Medicine, Geneva, Switzerland

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Jacques Philippe Division of Endocrinology and Diabetology, Geneva University Hospitals and Faculty of Medicine, Geneva, Switzerland

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Symptomatic; thyroid replacement indicated; limiting instrumental ADL Severe symptoms; limiting self-care ADL; hospitalization indicated Life-threatening consequences; urgent intervention indicated Death Hyperthyroidism Asymptomatic; clinical or

Article Type:
Review Article
DOI:
https://doi.org/10.1530/EC-18-0079
Volume/Issue:
Volume 7: Issue 5
Open access
Brenda Anguiano Departamento de Neurobiología Celular y Molecular, Instituto de Neurobiología, Universidad Nacional Autónoma de México, Campus Juriquilla, Querétaro, México

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Carlos Montes de Oca Departamento de Neurobiología Celular y Molecular, Instituto de Neurobiología, Universidad Nacional Autónoma de México, Campus Juriquilla, Querétaro, México

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Evangelina Delgado-González Departamento de Neurobiología Celular y Molecular, Instituto de Neurobiología, Universidad Nacional Autónoma de México, Campus Juriquilla, Querétaro, México

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Carmen Aceves Departamento de Neurobiología Celular y Molecular, Instituto de Neurobiología, Universidad Nacional Autónoma de México, Campus Juriquilla, Querétaro, México

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prostate weight ( 25 ). On the other hand, the effects of excessive THs on the prostate have been less studied. Figure 1 shows that hyperthyroidism induced during puberty increases testosterone levels but paradoxically reduces prostate weight in adult

Article Type:
Review Article
Collections:
Thyroid
DOI:
https://doi.org/10.1530/EC-21-0581
Volume/Issue:
Volume 11: Issue 2
Open access