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Maebashi Hirosegawa Clinic, Maebashi, Gunma, Japan
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the vasoprotective and vasopressor effects of nesfatin-1 at different doses in mouse models of peripheral arterial remodeling. Additionally, we sought to clarify the underlying mechanisms in cultured human vascular endothelial cells (VECs) and vascular
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People’s Hospital (item number: 2021-AR-059). Adult female C57BL/6J mice were housed withaccess to food and water ad libitum . The PCOS mouse model was established as described previously ( 18 ). In brief, female C57BL/6J mice (4 weeks old) were
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effects of oleuropein in alleviating symptoms of GDM is warranted. In this study, we aimed to study the efficacy of oleuropein in treating GDM in a genetically modified mouse model of GDM. GDM symptoms, including increased blood glucose levels, body
Faculté de Chirurgie Dentaire, Université de Toulouse III, Toulouse, France
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CHU de Toulouse, Laboratoire d’Hématologie, Toulouse, France
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Faculté de Chirurgie Dentaire, Université de Toulouse III, Toulouse, France
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indicate that, after a chronic CE + BZA treatment, mouse models are protected from in vivo induced thrombosis without significant functional alterations to coagulation factor levels. Concerning the question of the thrombotic risk of TSEC, only
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beneficial effects on longevity, with anti-aging and life-extending actions ( 6 ). Several mouse models of GH deficiency or GH resistance, including Ames dwarf, GHRKO and GHRHKO (the strain we used in this work) mice have increased lifespan ( 7 , 8 , 29
Center for Healthy Aging, Technische Universität Dresden, Dresden, Germany
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Center for Healthy Aging, Technische Universität Dresden, Dresden, Germany
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Center for Healthy Aging, Technische Universität Dresden, Dresden, Germany
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DFG Research Center and Cluster of Excellence for Regenerative Therapies, Technical University, Dresden, Germany
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Center for Healthy Aging, Technische Universität Dresden, Dresden, Germany
DFG Research Center and Cluster of Excellence for Regenerative Therapies, Technical University, Dresden, Germany
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Center for Healthy Aging, Technische Universität Dresden, Dresden, Germany
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treatment period, mice were killed to examine the effects on the skeleton. In addition, a mouse model for Cushing’s syndrome due to an N-ethyl-N-nitrosourea (ENU) induced mutation at −120 bp of the promoter region of the corticotropin releasing hormone
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subclinical hypothyroidism on glucose metabolism and the potential mechanisms in vivo , we established a subclinical hypothyroidism mouse model. There were no significant differences in the body weight of SCH mice compared with the control mice, suggesting
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mouse model of adenine-induced renal injury through inhibiting TGF-β1 signaling pathways . Frontiers in Cell and Developmental Biology 2021 9 603802 . ( https://doi.org/10.3389/fcell.2021.603802 ) 7 Zheng L Qin R Rao Z & Xiao W . High
Research Center for Translational Medicine, Cancer Stem Cell Institute, East Hospital, Tongji University School of Medicine, Shanghai, People’s Republic of China
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8 µg/mL polybrene (Sigma). The infected cells were selected with 2 µg/mL puromycin 72 h later; or at this time point, green fluorescence was monitored under inverted fluorescent microscope (BX51, Olympus). PNET mouse models Six to eight
Fujian Maternal-Fetal Clinical Medicine Research Center, Fuzhou, China
Fujian Key Laboratory of Prenatal Diagnosis and Birth Defect, Fuzhou, China
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Fujian Maternal-Fetal Clinical Medicine Research Center, Fuzhou, China
Fujian Key Laboratory of Prenatal Diagnosis and Birth Defect, Fuzhou, China
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development. Therefore, we intend to establish a PCOS mouse model and selectively modulate Mfn2 to regulate the growth and development of oocyte cells. This research aims to elucidate the mechanism of Mfn2 in the MAMs during oocyte development in mice with