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Kinnaree Sorapipatcharoen Department of Pediatrics, Faculty of Medicine Ramathibodi Hospital, Mahidol University, Bangkok, Thailand

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Thipwimol Tim-Aroon Department of Pediatrics, Faculty of Medicine Ramathibodi Hospital, Mahidol University, Bangkok, Thailand

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Pat Mahachoklertwattana Department of Pediatrics, Faculty of Medicine Ramathibodi Hospital, Mahidol University, Bangkok, Thailand

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Wasun Chantratita Center for Medical Genomics, Faculty of Medicine Ramathibodi Hospital, Mahidol University, Bangkok, Thailand

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Nareenart Iemwimangsa Center for Medical Genomics, Faculty of Medicine Ramathibodi Hospital, Mahidol University, Bangkok, Thailand

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Insee Sensorn Center for Medical Genomics, Faculty of Medicine Ramathibodi Hospital, Mahidol University, Bangkok, Thailand

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Bhakbhoom Panthan Center for Medical Genomics, Faculty of Medicine Ramathibodi Hospital, Mahidol University, Bangkok, Thailand

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Poramate Jiaranai Center for Medical Genomics, Faculty of Medicine Ramathibodi Hospital, Mahidol University, Bangkok, Thailand

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Saisuda Noojarern Department of Pediatrics, Faculty of Medicine Ramathibodi Hospital, Mahidol University, Bangkok, Thailand

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Patcharin Khlairit Department of Pediatrics, Faculty of Medicine Ramathibodi Hospital, Mahidol University, Bangkok, Thailand

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Sarunyu Pongratanakul Department of Pediatrics, Faculty of Medicine Ramathibodi Hospital, Mahidol University, Bangkok, Thailand

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Chittiwat Suprasongsin Research Center, Faculty of Medicine Ramathibodi Hospital, Mahidol University, Bangkok, Thailand

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Manassawee Korwutthikulrangsri Department of Pediatrics, Faculty of Medicine Ramathibodi Hospital, Mahidol University, Bangkok, Thailand

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Chutintorn Sriphrapradang Department of Medicine, Faculty of Medicine Ramathibodi Hospital, Mahidol University, Bangkok, Thailand

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Preamrudee Poomthavorn Department of Pediatrics, Faculty of Medicine Ramathibodi Hospital, Mahidol University, Bangkok, Thailand

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. Therefore, PAX8 variants might affect sodium iodide symporter expression ( 43 ). This study did not find the variants in the genes related to syndromic defects such as NKX2-1, FOXE1 , JAG1 and TBX1 because the patients with obvious syndromic

Article Type:
Research Article
DOI:
https://doi.org/10.1530/EC-20-0411
Volume/Issue:
Volume 9: Issue 11
Open access
Eugenie S Lim Barts Health NHS Trust, Barts and the London School of Medicine and Dentistry, London, UK

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Shanty G Shah Barts and the London School of Medicine and Dentistry, QMUL, London, UK

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Mona Waterhouse Barts Health NHS Trust, Barts and the London School of Medicine and Dentistry, London, UK

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Scott Akker Barts Health NHS Trust, Barts and the London School of Medicine and Dentistry, London, UK
Barts and the London School of Medicine and Dentistry, QMUL, London, UK

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William Drake Barts Health NHS Trust, Barts and the London School of Medicine and Dentistry, London, UK
Barts and the London School of Medicine and Dentistry, QMUL, London, UK

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Nick Plowman Barts Health NHS Trust, Barts and the London School of Medicine and Dentistry, London, UK
Barts and the London School of Medicine and Dentistry, QMUL, London, UK

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Daniel M Berney Barts Health NHS Trust, Barts and the London School of Medicine and Dentistry, London, UK
Barts and the London School of Medicine and Dentistry, QMUL, London, UK

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Polly Richards Barts Health NHS Trust, Barts and the London School of Medicine and Dentistry, London, UK

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Ashok Adams Barts Health NHS Trust, Barts and the London School of Medicine and Dentistry, London, UK

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Ewa Nowosinska Barts Health NHS Trust, Barts and the London School of Medicine and Dentistry, London, UK

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Carmel Brennan Barts Health NHS Trust, Barts and the London School of Medicine and Dentistry, London, UK

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Maralyn Druce Barts Health NHS Trust, Barts and the London School of Medicine and Dentistry, London, UK
Barts and the London School of Medicine and Dentistry, QMUL, London, UK

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thyroid hormone: the iodination of tyrosine residues on Tg and the intra-molecular coupling reaction of iodinated tyrosine. Thus, a balance between the sodium-iodide symporter (NIS)-mediated iodine influx and TPO-inhibited efflux determines the

Article Type:
Research Article
DOI:
https://doi.org/10.1530/EC-19-0053
Volume/Issue:
Volume 8: Issue 5
Open access
Ashley N Reeb Department of Otolaryngology, Head and Neck Surgery, Saint Louis University School of Medicine, Saint Louis, Missouri, USA

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Andrea Ziegler Department of Otolaryngology, Head and Neck Surgery, Saint Louis University School of Medicine, Saint Louis, Missouri, USA

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Reigh-Yi Lin Department of Otolaryngology, Head and Neck Surgery, Saint Louis University School of Medicine, Saint Louis, Missouri, USA

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differentiation markers thyroglobulin ( TG ), thyroperoxidase ( TPO ), sodium-iodide symporter ( NIS ), or the receptor for thyroid stimulating hormone ( TSHR ) ( Fig. 1B ), confirming the dedifferentiated state of these human FTC cell lines. Figure 1

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DOI:
https://doi.org/10.1530/EC-15-0114
Volume/Issue:
Volume 5: Issue 2
Open access
Erika Urbano Lima Biological Science Department, Thyroid Molecular Sciences Laboratory, Universidade Federal de São Paulo, São Paulo, Brazil
Structural and Functional Biology Program, Universidade Federal de São Paulo, São Paulo, Brazil

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Ileana G S Rubio Biological Science Department, Thyroid Molecular Sciences Laboratory, Universidade Federal de São Paulo, São Paulo, Brazil
Structural and Functional Biology Program, Universidade Federal de São Paulo, São Paulo, Brazil

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Joaquim Custodio Da Silva Department of Bio-regulation, Thyroid Study Laboratory, Health & Science Institute, Federal University of Bahia, Salvador, Brazil
Post-graduate Program in Interactive Processes of Organs and Systems, Health & Science Institute, Federal University of Bahia, Salvador, Brazil

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Ana Luiza Galrão Biological Science Department, Thyroid Molecular Sciences Laboratory, Universidade Federal de São Paulo, São Paulo, Brazil

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Danielle Pêssoa Department of Bio-regulation, Thyroid Study Laboratory, Health & Science Institute, Federal University of Bahia, Salvador, Brazil
Post-graduate Program in Interactive Processes of Organs and Systems, Health & Science Institute, Federal University of Bahia, Salvador, Brazil

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Taise Cerqueira Oliveira Department of Bio-regulation, Thyroid Study Laboratory, Health & Science Institute, Federal University of Bahia, Salvador, Brazil

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Fabiane Carrijo Department of Bio-regulation, Thyroid Study Laboratory, Health & Science Institute, Federal University of Bahia, Salvador, Brazil
Post-graduate Program in Interactive Processes of Organs and Systems, Health & Science Institute, Federal University of Bahia, Salvador, Brazil

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Igor Silva Campos Department of Pathology, Sao Rafael Hospital, Salvador, Brazil

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Luciano Fonseca Espinheira Department of Pathology, Sao Rafael Hospital, Salvador, Brazil
Department of Anatomic Pathology & Legal Medicine, Bahia Federal Medical School, Federal University of Bahia, Salvador, Brazil

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Luiz Jose Sampaio Nuclear Medicine Department, Sao Rafael Hospital, Salvador, Brazil

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Claudio Rogerio Lima Head and Neck Surgery Department, Sao Rafael Hospital, Salvador, Brazil

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Janete Maria Cerutti Structural and Functional Biology Program, Universidade Federal de São Paulo, São Paulo, Brazil
Division of Genetics, Department of Morphology and Genetics, Genetic Basis of Thyroid Tumors Laboratory, Paulista School of Medicine, Universidade Federal de São Paulo, São Paulo, Brazil

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Helton Estrela Ramos Department of Bio-regulation, Thyroid Study Laboratory, Health & Science Institute, Federal University of Bahia, Salvador, Brazil
Post-graduate Program in Interactive Processes of Organs and Systems, Health & Science Institute, Federal University of Bahia, Salvador, Brazil

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.2015.0020 ) 22 Galrao AL Camargo RY Friguglietti CU Moraes L Cerutti JM Serrano-Nascimento C Suzuki MF Medeiros-Neto G Rubio IG. Hypermethylation of a New Distal Sodium/Iodide Symporter (NIS) enhancer (NDE) is associated with reduced NIS expression

Article Type:
Research Article
DOI:
https://doi.org/10.1530/EC-18-0380
Volume/Issue:
Volume 7: Issue 12
Open access
Guoquan Zhu Department of Nuclear Medicine, Zhujiang Hospital of Southern Medical University, Guangzhou, China

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Yuying Deng Department of Nuclear Medicine, Zhujiang Hospital of Southern Medical University, Guangzhou, China

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Liqin Pan Department of Nuclear Medicine, Zhujiang Hospital of Southern Medical University, Guangzhou, China

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Wei Ouyang Department of Nuclear Medicine, Zhujiang Hospital of Southern Medical University, Guangzhou, China

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Huijuan Feng Department of Nuclear Medicine, Zhujiang Hospital of Southern Medical University, Guangzhou, China

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Juqing Wu Department of Nuclear Medicine, Zhujiang Hospital of Southern Medical University, Guangzhou, China

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Pan Chen Department of Nuclear Medicine, Zhujiang Hospital of Southern Medical University, Guangzhou, China

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Jing Wang Department of Nuclear Medicine, Zhujiang Hospital of Southern Medical University, Guangzhou, China

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Yanying Chen Department of Nuclear Medicine, Zhujiang Hospital of Southern Medical University, Guangzhou, China

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Jiaxin Luo Department of Nuclear Medicine, Zhujiang Hospital of Southern Medical University, Guangzhou, China

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significantly reduces sodium-iodide symporter (NIS) expression and radioiodine uptake ability and influences RAI therapy to the point of causing RAI-refractory PTC. Jiao et al . ( 16 ) designed their research concerning RAI therapy and PTC with the BRAF V600E

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DOI:
https://doi.org/10.1530/EC-19-0045
Volume/Issue:
Volume 8: Issue 6
Open access
T P McVeigh Discipline of Surgery, Lambe Institute for Translational Research, School of Medicine, NUI Galway, Galway, Ireland

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R J Mulligan Discipline of Surgery, Lambe Institute for Translational Research, School of Medicine, NUI Galway, Galway, Ireland

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U M McVeigh Discipline of Surgery, Lambe Institute for Translational Research, School of Medicine, NUI Galway, Galway, Ireland

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P W Owens Discipline of Surgery, Lambe Institute for Translational Research, School of Medicine, NUI Galway, Galway, Ireland

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N Miller Discipline of Surgery, Lambe Institute for Translational Research, School of Medicine, NUI Galway, Galway, Ireland

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M Bell Department of Endocrinology, School of Medicine, NUI Galway, Galway, Ireland

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F Sebag Department of Endocrine Surgery Centre hospitalo-universitaire de La Conception, Assistance Publique Hôpitaux de Marseille, Marseille, France
Aix-Marseille Université, Faculté de Médecine, Marseille, France

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C Guerin Department of Endocrine Surgery Centre hospitalo-universitaire de La Conception, Assistance Publique Hôpitaux de Marseille, Marseille, France
Aix-Marseille Université, Faculté de Médecine, Marseille, France

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D S Quill Discipline of Surgery, Lambe Institute for Translational Research, School of Medicine, NUI Galway, Galway, Ireland

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J B Weidhaas David Geffen School of Medicine, University of California, Los Angeles, USA

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M J Kerin Discipline of Surgery, Lambe Institute for Translational Research, School of Medicine, NUI Galway, Galway, Ireland

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A J Lowery Discipline of Surgery, Lambe Institute for Translational Research, School of Medicine, NUI Galway, Galway, Ireland

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-1036 ) 10.1210/er.2012-1036 24311738 9 Ryan J Curran CE Hennessy E Newell J Morris JC Kerin MJ Dwyer RM . The Sodium Iodide Symporter (NIS) and potential regulators in normal, benign and malignant human breast tissue. PLoS ONE

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DOI:
https://doi.org/10.1530/EC-17-0196
Volume/Issue:
Volume 6: Issue 8
Open access
Athanasios Zervas Endocrine Unit, Athens Medical Centre, Athens, Greece

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George Chrousos Division of Endocrinology, Metabolism and Diabetes, First Department of Pediatrics, University Research Institute of Maternal and Child Health and Precision Medicine, and UNESCO Chair on Adolescent Health Care, National and Kapodistrian University of Athens, Athens, Greece
National and Kapodistrian University of Athens Medical School, ‘Aghia Sophia’ Children’s Hospital, Athens, Greece

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Sarantis Livadas Endocrine Unit, Athens Medical Centre, Athens, Greece

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genetic causes of dyshormonogenesis include mutations in thyroglobulin (TG), thyroperoxidase (TPO), dual oxidase 2 (DUOX2) and its accessory protein (DUOXA2), the sodium-iodide symporter (SLC5A5), pendrin (SLC26A4), and iodotyrosine deiodinase ( 22

Article Type:
Review Article
DOI:
https://doi.org/10.1530/EC-20-0615
Volume/Issue:
Volume 10: Issue 5
Open access
Bilal B Mughal CNRS/UMR7221, Muséum National d’Histoire Naturelle, Sorbonne Universités, Paris, France

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Jean-Baptiste Fini CNRS/UMR7221, Muséum National d’Histoire Naturelle, Sorbonne Universités, Paris, France

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Barbara A Demeneix CNRS/UMR7221, Muséum National d’Histoire Naturelle, Sorbonne Universités, Paris, France

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risk of neurodevelopmental disease (note: targets not drawn to scale). Perchlorate Perchlorate is a well-characterised inhibitor of the sodium-iodide symporter (NIS) that is expressed in the basal membrane of thyroid follicular cells and

Article Type:
Review Article
DOI:
https://doi.org/10.1530/EC-18-0029
Volume/Issue:
Volume 7: Issue 4
Open access