Wei Li
Wei Li
Department of Nuclear Medicine, Tianjin Medical University General Hospital, Tianjin, People’s Republic of China
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Qing Huang
Qing Huang
Collage of Tourism and Service Management, Nankai University, Tianjin, People’s Republic of China
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Danyang Sun
Danyang Sun
Department of Nuclear Medicine, Tianjin Medical University General Hospital, Tianjin, People’s Republic of China
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Guizhi Zhang
Guizhi Zhang
Department of Nuclear Medicine, Tianjin Medical University General Hospital, Tianjin, People’s Republic of China
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Jian Tan
Jian Tan
Department of Nuclear Medicine, Tianjin Medical University General Hospital, Tianjin, People’s Republic of China
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conducted in thyroid cancer cell lines. Lentivirus-mediated small interfering RNA (siRNA) targeting of RDM1 was used to downregulate gene expression. Then, experiments were performed to examine the effect of RDM1 silencing on the cell cycle, apoptosis
Jiwen Yang
Jiwen Yang
Department of Nuclear Medicine, Yijishan Hospital of Wannan Medical College, Wuhu City, Anhui Province, China
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Yayin Huang
Yayin Huang
Department of Clinical Laboratory, The Second People’s Hospital of Wuhu, Wuhu City, Anhui Province, China
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Bohan Dong
Bohan Dong
Department of Biochemistry and Molecular Biology, Wannan Medical College, Wuhu City, Anhui Province, China
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Yunhai Dai
Yunhai Dai
Department of Nuclear Medicine, Yijishan Hospital of Wannan Medical College, Wuhu City, Anhui Province, China
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Highlights lncRNA DLEU2 is significantly upregulated in thyroid cancer tissues and cell lines. DLEU2 accelerates the proliferation, migration and aerobic glycolysis while inhibits the apoptosis of thyroid cancer cells. miR-205
Emily Warmington
Emily Warmington
Institute of Metabolism and System Research, University of Birmingham, Birmingham, UK
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Gabrielle Smith
Gabrielle Smith
Institute of Metabolism and System Research, University of Birmingham, Birmingham, UK
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Vasileios Chortis
Vasileios Chortis
Institute of Metabolism and System Research, University of Birmingham, Birmingham, UK
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Raimunde Liang
Raimunde Liang
Division of Endocrinology and Diabetes, University Hospital of Wuerzburg, Wuerzburg, Germany
Department of Neurosurgery, Technical University Munich (TMU), Munich, Germany
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Department of Neurosurgery, Technical University Munich (TMU), Munich, Germany
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Juliane Lippert
Juliane Lippert
Division of Endocrinology and Diabetes, University Hospital of Wuerzburg, Wuerzburg, Germany
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Sonja Steinhauer
Sonja Steinhauer
Division of Endocrinology and Diabetes, University Hospital of Wuerzburg, Wuerzburg, Germany
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Laura-Sophie Landwehr
Laura-Sophie Landwehr
Division of Endocrinology and Diabetes, University Hospital of Wuerzburg, Wuerzburg, Germany
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Constanze Hantel
Constanze Hantel
Department of Endocrinology, Diabetology and Clinical Nutrition, University Hospital Zurich (USZ) and University of Zurich (UZH), Zurich, Switzerland
Medizinische Klinik Und Poliklinik III, University Hospital Carl Gustav Carus, Dresden, Germany
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Medizinische Klinik Und Poliklinik III, University Hospital Carl Gustav Carus, Dresden, Germany
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Katja Kiseljak-Vassiliades
Katja Kiseljak-Vassiliades
Division of Endocrinology Metabolism and Diabetes, University of Colorado Anschutz Medical Campus, Aurora, Colorado, USA
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Margaret E Wierman
Margaret E Wierman
Division of Endocrinology Metabolism and Diabetes, University of Colorado Anschutz Medical Campus, Aurora, Colorado, USA
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Barbara Altieri
Barbara Altieri
Division of Endocrinology and Diabetes, University Hospital of Wuerzburg, Wuerzburg, Germany
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Paul A Foster
Paul A Foster
Institute of Metabolism and System Research, University of Birmingham, Birmingham, UK
Centre for Endocrinology, Diabetes and Metabolism, Birmingham Health Partners, Birmingham, UK
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Centre for Endocrinology, Diabetes and Metabolism, Birmingham Health Partners, Birmingham, UK
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Cristina L Ronchi
Cristina L Ronchi
Institute of Metabolism and System Research, University of Birmingham, Birmingham, UK
Centre for Endocrinology, Diabetes and Metabolism, Birmingham Health Partners, Birmingham, UK
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Centre for Endocrinology, Diabetes and Metabolism, Birmingham Health Partners, Birmingham, UK
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shown to be frequently overexpressed ( 9 , 16 , 17 ) and associated with shorter patient survival ( 16 , 17 ). Moreover, the first-generation inhibitor BI-2536 has been demonstrated to reduce cell viability and induce apoptosis in standard ACC cell
Joanna Klubo-Gwiezdzinska
Joanna Klubo-Gwiezdzinska
National Institute of Health, NIDDK, Office 10 Center Drive, Bethesda, Maryland, USA
Division of Endocrinology, Department of Medicine, Medstar Washington Hospital Center, Washington Hospital Center, Northwest, Washington, District of Columbia, USA
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Division of Endocrinology, Department of Medicine, Medstar Washington Hospital Center, Washington Hospital Center, Northwest, Washington, District of Columbia, USA
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John Costello Jr
John Costello Jr
Department of Pediatrics, Uniformed Services University of the Health Sciences, Bethesda, Maryland, USA
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Kirk Jensen
Kirk Jensen
Department of Pediatrics, Uniformed Services University of the Health Sciences, Bethesda, Maryland, USA
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Aneeta Patel
Aneeta Patel
Department of Pediatrics, Uniformed Services University of the Health Sciences, Bethesda, Maryland, USA
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Rok Tkavc
Rok Tkavc
Department of Pathology, Uniformed Services University of the Health Sciences, Henry M. Jackson Foundation for the Advancement of Military Medicine, Bethesda, Maryland, USA
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Douglas Van Nostrand
Douglas Van Nostrand
Division of Endocrinology, Department of Medicine, Medstar Washington Hospital Center, Washington Hospital Center, Northwest, Washington, District of Columbia, USA
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Kenneth D Burman
Kenneth D Burman
Division of Endocrinology, Department of Medicine, Medstar Washington Hospital Center, Washington Hospital Center, Northwest, Washington, District of Columbia, USA
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Leonard Wartofsky
Leonard Wartofsky
Division of Endocrinology, Department of Medicine, Medstar Washington Hospital Center, Washington Hospital Center, Northwest, Washington, District of Columbia, USA
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Vasyl Vasko
Vasyl Vasko
Department of Pediatrics, Uniformed Services University of the Health Sciences, Bethesda, Maryland, USA
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radioprotective activity in normal cells, amifostine may enhance radiation-inducible apoptosis in cancer cells harboring oncogene mutations. It has to be noted that, despite the clinical use of amifostine in treatment of thyroid cancer patients, there is no
M L Gild
M L Gild
Cancer Genetics Laboratory, Department of Endocrinology, Kolling Institute of Medical Research, Sydney, New South Wales, Australia
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M Bullock
M Bullock
Cancer Genetics Laboratory, Department of Endocrinology, Kolling Institute of Medical Research, Sydney, New South Wales, Australia
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C K Pon
C K Pon
Cancer Genetics Laboratory, Department of Endocrinology, Kolling Institute of Medical Research, Sydney, New South Wales, Australia
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B G Robinson
B G Robinson
Cancer Genetics Laboratory, Department of Endocrinology, Kolling Institute of Medical Research, Sydney, New South Wales, Australia
Cancer Genetics Laboratory, Department of Endocrinology, Kolling Institute of Medical Research, Sydney, New South Wales, Australia
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Cancer Genetics Laboratory, Department of Endocrinology, Kolling Institute of Medical Research, Sydney, New South Wales, Australia
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R J Clifton-Bligh
R J Clifton-Bligh
Cancer Genetics Laboratory, Department of Endocrinology, Kolling Institute of Medical Research, Sydney, New South Wales, Australia
Cancer Genetics Laboratory, Department of Endocrinology, Kolling Institute of Medical Research, Sydney, New South Wales, Australia
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Cancer Genetics Laboratory, Department of Endocrinology, Kolling Institute of Medical Research, Sydney, New South Wales, Australia
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it is this mechanism that is exploited when targeting its inhibition (5) . HSP90 client proteins include many kinases involved in apoptosis and signaling. These include AKT (and its phosphorylated form), p53, Raf-1, Bcr-abl, and BRAF; many of these
Caio Jordão Teixeira
Caio Jordão Teixeira
Department of Pharmacology, Faculty of Medical Sciences, State University of Campinas, Campinas, Brazil
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Junia Carolina Santos-Silva
Junia Carolina Santos-Silva
Department of Pharmacology, Faculty of Medical Sciences, State University of Campinas, Campinas, Brazil
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Dailson Nogueira de Souza
Dailson Nogueira de Souza
Department of Pharmacology, Faculty of Medical Sciences, State University of Campinas, Campinas, Brazil
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Alex Rafacho
Alex Rafacho
Department of Physiological Sciences, Center of Biological Sciences, Federal University of Santa Catarina, Florianópolis, Brazil
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Gabriel Forato Anhe
Gabriel Forato Anhe
Department of Pharmacology, Faculty of Medical Sciences, State University of Campinas, Campinas, Brazil
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Silvana Bordin
Silvana Bordin
Department of Physiology and Biophysics, Institute of Biomedical Sciences, University of Sao Paulo, Sao Paulo, Brazil
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pregnant rats show evident plasticity, which allows the morphological structures to return to the nonpregnant state just after delivery. Increased apoptosis and reduced proliferation account for the reversal of pancreatic β-cell mass as early as 3–4 days
Robert I Menzies
Robert I Menzies
The University/BHF Centre for Cardiovascular Science, University of Edinburgh, The Queen’s Medical Research Institute, Edinburgh, UK
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Xin Zhao
Xin Zhao
The University/BHF Centre for Cardiovascular Science, University of Edinburgh, The Queen’s Medical Research Institute, Edinburgh, UK
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Linda J Mullins
Linda J Mullins
The University/BHF Centre for Cardiovascular Science, University of Edinburgh, The Queen’s Medical Research Institute, Edinburgh, UK
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John J Mullins
John J Mullins
The University/BHF Centre for Cardiovascular Science, University of Edinburgh, The Queen’s Medical Research Institute, Edinburgh, UK
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Carolynn Cairns
Carolynn Cairns
The University/BHF Centre for Cardiovascular Science, University of Edinburgh, The Queen’s Medical Research Institute, Edinburgh, UK
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Nicola Wrobel
Nicola Wrobel
The University/BHF Centre for Cardiovascular Science, University of Edinburgh, The Queen’s Medical Research Institute, Edinburgh, UK
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Donald R Dunbar
Donald R Dunbar
The University/BHF Centre for Cardiovascular Science, University of Edinburgh, The Queen’s Medical Research Institute, Edinburgh, UK
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Matthew A Bailey
Matthew A Bailey
The University/BHF Centre for Cardiovascular Science, University of Edinburgh, The Queen’s Medical Research Institute, Edinburgh, UK
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Christopher J Kenyon
Christopher J Kenyon
The University/BHF Centre for Cardiovascular Science, University of Edinburgh, The Queen’s Medical Research Institute, Edinburgh, UK
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proliferation and apoptosis that increases cell number. Sterol/lipid metabolic pathways are also changed in several ways to allow cholesterol to be channelled towards steroidogenesis. Materials and methods All experiments involving animals were approved
Shikai Gui
Shikai Gui
Department of Neurosurgery, the 2nd affiliated hospital, Jiangxi Medical College, Nanchang University, Nanchang, Jiangxi Province, China
Nanchang University, Nanchang, Jiangxi Province, China
Institute of Neuroscience, Nanchang University, Nanchang, Jiangxi Province, China
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Nanchang University, Nanchang, Jiangxi Province, China
Institute of Neuroscience, Nanchang University, Nanchang, Jiangxi Province, China
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Wanli Yu
Wanli Yu
Department of Neurosurgery, the 2nd affiliated hospital, Jiangxi Medical College, Nanchang University, Nanchang, Jiangxi Province, China
Nanchang University, Nanchang, Jiangxi Province, China
Institute of Neuroscience, Nanchang University, Nanchang, Jiangxi Province, China
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Nanchang University, Nanchang, Jiangxi Province, China
Institute of Neuroscience, Nanchang University, Nanchang, Jiangxi Province, China
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Jiabao Xie
Jiabao Xie
Department of Neurosurgery, the 2nd affiliated hospital, Jiangxi Medical College, Nanchang University, Nanchang, Jiangxi Province, China
Nanchang University, Nanchang, Jiangxi Province, China
Institute of Neuroscience, Nanchang University, Nanchang, Jiangxi Province, China
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Nanchang University, Nanchang, Jiangxi Province, China
Institute of Neuroscience, Nanchang University, Nanchang, Jiangxi Province, China
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Lunshan Peng
Lunshan Peng
Department of Neurosurgery, the 2nd affiliated hospital, Jiangxi Medical College, Nanchang University, Nanchang, Jiangxi Province, China
Nanchang University, Nanchang, Jiangxi Province, China
Institute of Neuroscience, Nanchang University, Nanchang, Jiangxi Province, China
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Nanchang University, Nanchang, Jiangxi Province, China
Institute of Neuroscience, Nanchang University, Nanchang, Jiangxi Province, China
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Yuanyuan Xiong
Yuanyuan Xiong
Department of Neurosurgery, the 2nd affiliated hospital, Jiangxi Medical College, Nanchang University, Nanchang, Jiangxi Province, China
Nanchang University, Nanchang, Jiangxi Province, China
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Nanchang University, Nanchang, Jiangxi Province, China
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Zhen Song
Zhen Song
Nanchang University, Nanchang, Jiangxi Province, China
Department of Urology, the 2nd affiliated hospital, Jiangxi Medical College, Nanchang University, Nanchang, Jiangxi Province, China
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Department of Urology, the 2nd affiliated hospital, Jiangxi Medical College, Nanchang University, Nanchang, Jiangxi Province, China
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Haitao Luo
Haitao Luo
Department of Neurosurgery, the 2nd affiliated hospital, Jiangxi Medical College, Nanchang University, Nanchang, Jiangxi Province, China
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Juexian Xiao
Juexian Xiao
Department of Neurosurgery, the 2nd affiliated hospital, Jiangxi Medical College, Nanchang University, Nanchang, Jiangxi Province, China
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Shengtao Yuan
Shengtao Yuan
Jiangsu Key Laboratory of Drug Screening, China Pharmaceutical University, Nanjing, Jiangsu Province, China
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Zujue Cheng
Zujue Cheng
Department of Neurosurgery, the 2nd affiliated hospital, Jiangxi Medical College, Nanchang University, Nanchang, Jiangxi Province, China
Nanchang University, Nanchang, Jiangxi Province, China
Institute of Neuroscience, Nanchang University, Nanchang, Jiangxi Province, China
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Nanchang University, Nanchang, Jiangxi Province, China
Institute of Neuroscience, Nanchang University, Nanchang, Jiangxi Province, China
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were then fixed with 4% paraformaldehyde and stained with 2% crystal violet. Colonies comprising 50 or more cells were counted under a microscope. Apoptosis assay Cell apoptosis was detected using the Apoptosis Detection Kit (KeyGEN BioTECH
Fan Zhang
Fan Zhang
Department of Endocrinology and Metabolism, Institute of Endocrinology, Liaoning Provincial Key Laboratory of Endocrine Diseases, The First Affiliated Hospital of China Medical University, Shenyang, Liaoning, China
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Jian Chen
Jian Chen
Department of Endocrinology and Metabolism, Institute of Endocrinology, Liaoning Provincial Key Laboratory of Endocrine Diseases, The First Affiliated Hospital of China Medical University, Shenyang, Liaoning, China
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Xinyue Lin
Xinyue Lin
Department of Endocrinology and Metabolism, Institute of Endocrinology, Liaoning Provincial Key Laboratory of Endocrine Diseases, The First Affiliated Hospital of China Medical University, Shenyang, Liaoning, China
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Shiqiao Peng
Shiqiao Peng
Department of Endocrinology and Metabolism, Institute of Endocrinology, Liaoning Provincial Key Laboratory of Endocrine Diseases, The First Affiliated Hospital of China Medical University, Shenyang, Liaoning, China
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Xiaohui Yu
Xiaohui Yu
Department of Endocrinology and Metabolism, Institute of Endocrinology, Liaoning Provincial Key Laboratory of Endocrine Diseases, The First Affiliated Hospital of China Medical University, Shenyang, Liaoning, China
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Zhongyan Shan
Zhongyan Shan
Department of Endocrinology and Metabolism, Institute of Endocrinology, Liaoning Provincial Key Laboratory of Endocrine Diseases, The First Affiliated Hospital of China Medical University, Shenyang, Liaoning, China
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Weiping Teng
Weiping Teng
Department of Endocrinology and Metabolism, Institute of Endocrinology, Liaoning Provincial Key Laboratory of Endocrine Diseases, The First Affiliated Hospital of China Medical University, Shenyang, Liaoning, China
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relationship between cognitive impairment due to SCH and the apoptosis in cerebral cortex remains unclear. The p75 neurotrophin receptor (p75 NTR ) is an important member of the death receptor family. Although the role of the p75 NTR -mediated signal
Qian Deng
Qian Deng
Graduate College of Anhui University of Chinese Medicine, Hefei, China
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Yue Zhu
Yue Zhu
Graduate College of Anhui University of Chinese Medicine, Hefei, China
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Mengmeng Zhang
Mengmeng Zhang
Graduate College of Anhui University of Chinese Medicine, Hefei, China
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Aihua Fei
Aihua Fei
Department of Endocrinology, The Second Affiliated Hospital of Anhui University of Chinese Medicine, Hefei, China
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Jiaqi Liang
Jiaqi Liang
Graduate College of Anhui University of Chinese Medicine, Hefei, China
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Jinjin Zheng
Jinjin Zheng
Graduate College of Anhui University of Chinese Medicine, Hefei, China
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Qingping Zhang
Qingping Zhang
College of Acupuncture-moxibustion and Tuina, Anhui University of Chinese Medicine, Hefei, China
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Tong Cheng
Tong Cheng
Department of Geriatrics, Zhongshan Hospital, Fudan University, Shanghai, China
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Xia Ge
Xia Ge
Department of Endocrinology, The Second Affiliated Hospital of Anhui University of Chinese Medicine, Hefei, China
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decreases. Distinct from the features of necrosis, autophagy, and apoptosis, ferroptosis could result in compromised plasma membrane integrity, mild chromatin condensation, cytoplasm and cytoplasmic organelles swelling, increased mitochondrial membrane
