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satiety ( 3 , 4 ). Clinical evidence has shown a significant decrease in GLP-1 levels in patients with impaired glucose metabolism, emphasizing the importance of the gut–pancreas hormone axis in diabetes pathogenesis ( 3 ). However, the molecular
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TT or nearly total thyroidectomy; (iv) none of the patients had clinical evidence of contralateral thyroid carcinoma preoperatively (If any suspicious ultrasound images exist, FNA was performed); (v) sufficient data provided concerning the feature of
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Department of Surgery, Warsaw Medical University, Warsaw, Poland
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appendiceal wall C Adenocarcinoma ex-GCC, poorly differentiated carcinoma type Minimum of focal evidence of goblet cell morphologyA component indistinguishable from poorly differentiated adenocarcinoma (e.g. gland-forming, confluent sheets of signet
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Department of Endocrinology, Sahlgrenska University Hospital, Gothenburg, Sweden
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cytokines, leptin, estradiol and insulin. These substances may inhibit the activity of the hypothalamic–pituitary gonadal axis at multiple levels, thus lowering the concentrations of testosterone ( 7 ). There is also evidence that obesity increases aromatase
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Pathogenic variants identified by targeted panel sequencing in a cohort of 117 patients with congenital hypopituitarism. Patient Gene Allelic variant GnomAD ABraOM Inheritance Evidence of pathogenicity according to ACMG/AMP Final ACMG
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) and cardiovascular profile ( 12 , 13 ) in women with PCOS, as well as in the general population ( 14 , 15 ). However, scientific evidence is controversial, as other studies suggest a negative impact of adrenal hyperandrogenism on metabolic
Developmental Endocrinology Research Group, University of Glasgow, Glasgow, UK
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Institute of Cancer and Genomic Sciences, University of Birmingham, Birmingham, UK
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Centre for Endocrinology, William Harvey Research Institute, Barts and The London Medical School, Queen Mary University of London, London, UK
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Department of Endocrinology, Imperial College Healthcare NHS Trust, Hammersmith Hospital, London, UK
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Department of Paediatric Endocrinology, Makarios Children's Hospital, Nicosia, Cyprus
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Sussex Community NHS Trust, Brighton, UK
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Developmental Endocrinology Research Group, University of Glasgow, Glasgow, UK
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The University of Dublin, Trinity College Dublin, Dublin, Republic of Ireland
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purpose of this article is to provide evidence-based expert consensus guidance on the management of CA with PWS. Aims and objectives The specific areas in PWS which will be covered are: Management in infancy, childhood and adolescence
Institute of Cardiovascular Sciences, University of Birmingham, Birmingham, UK
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Institute of Metabolism and Systems Research, University of Birmingham, Birmingham, UK
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Department of Hepatology and Liver Transplantation, University Hospitals Birmingham (Queen Elizabeth), NHS Hospitals Foundation Trust, Birmingham, UK
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with GI-NET in 2015 and found to have elevated NT proBNP, which was attributed to age and hypertension without evidence of CHD. He remained under follow-up, developing tricuspid regurgitation that became severe and was associated with breathlessness. He
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controls Age range 27–62 yrs Mean age 47.9 + 9.4 60 32M, 28F AT patients showed evidence of dyslipidaemia; higher TGs ( P = 0.039), lower HDL-C ( P = 0.016) and higher TG/HDL-C ratio ( P = 0.036) and elevated apolipoprotein B ( P = 0
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; defined as age at menopause <45 years) or premature ovarian insufficiency (POI; defined as age at menopause <40 years) ( 3 ). The aim of this narrative review was to provide an evidence-based approach to the menopause-associated CVD risk. Moreover, the