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. Receptors for sex steroid hormones have widespread expression in genital tissues and the brain, and sex hormones are thought to play a role in female sexuality ( 6 ). While no clear-cut relationship between androgen levels and sexual desire has been
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rate of between 16 and 38% (1, 3, 4, 5) . The correct diagnosis of adrenocortical tumors (ACTs) is, therefore, understandably of growing importance. ACTs can be divided into functioning and nonfunctioning tumors based on whether they secrete steroids
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biological origins) compared with men with hypogonadism, there was no difference in optimized inter-injection interval according to diagnosis. This finding is consistent with previous pharmacokinetic findings of injectable TU showing serum and urine steroid
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Jones BF Barry CV Wulczyn KE Thomas BJ , et al . Gonadal steroids and body composition, strength, and sexual function in men . New England Journal of Medicine 2013 369 1011 – 1022 . ( https://doi.org/10.1056/NEJMoa1206168 ) 10 Cohen PG . The
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without hypogonadism augment the activation of the coagulation system ( 23 ). More extreme doses of androgens, as can be seen during anabolic androgenic steroid (AAS) abuse, induce a procoagulant effect related to TF-induced thrombin generation ( 24
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. Journal of Steroid Biochemistry and Molecular Biology 2016 164 127 – 133 . ( https://doi.org/10.1016/j.jsbmb.2015.12.003 ) 16 Ginsberg C Hoofnagle AN Katz R Becker JO Kritchevsky SB Shlipak MG Sarnak MJ Ix JH . The vitamin D metabolite
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outcomes in CCS, including cardiovascular risk, decreased bone density and frail health ( 7 , 8 , 46 , 47 ). Sex steroids have been reported to promote cell growth and proliferation in vitro . Two of the most common cancer types, breast and prostate
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). The high prevalence of Addison’s disease and POI is due the presence of cross-reacting autoantibodies to steroid-producing cells (StCA), targeting antigenic factors including 17alpha hydroxylase/17,20-lyase (17-OH), P-450 side chain cleavage enzyme (P
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-hydroxylase deficiency: the p.Q356X and the novel p.G379V . Clinical Genetics 2010 78 398 – 401 . ( doi:10.1111/j.1399-0004.2010.01403.x ). 15 Wedell A Luthman H . Steroid 21-hydroxylase deficiency: two additional mutations in salt-wasting disease and rapid
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class of steroid hormones present in almost every cell of the body and are essential for survival ( 13 ). Glucocorticoid release is regulated by the hypothalamic-pituitary-adrenal (HPA) axis. The suprachiasmatic nucleus of the hypothalamus releases