Department of Clinical Biochemistry and Laboratory Medicine, Faculty of Medicine, Tabriz University of Medical Sciences, Tabriz, Iran
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Department of Reproductive Biology, Faculty of Advanced Medical Sciences, Tabriz University of Medical Sciences, Tabriz, Iran
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Stem Cell Research Center, Tabriz University of Medical Sciences, Tabriz, Iran
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Department of Clinical Biochemistry and Laboratory Medicine, Faculty of Medicine, Tabriz University of Medical Sciences, Tabriz, Iran
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pathway ( 70 ). Additionally, apelin has antioxidant properties and could increase catalase activity and attenuate reactive oxygen species production ( 71 , 72 ). Since oxidative stress could have detrimental effects on embryo implantation ( 73 , 74
Imperial College London, London, UK
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, to test whether the AR ‘splicing’ event was unique to AR pathway inhibition or was a more general stress response, we subjected LNCaP cells to a variety of cellular stresses including hypoxia (mimicked by CoCl 2 ), DNA damaging stress (by
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Cermak K Shumelinsky F Alexiou J Gebhart MJ . Case reports: subtrochanteric femoral stress fractures after prolonged alendronate therapy . Clinical Orthopaedics and Related Research 2010 468 1991 – 1996 . ( doi:10.1007/s11999
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neuronal degeneration in the hippocampal region after emotional and physical stress at weaning, while wild-type mice show good survival of the hippocampal neurons ( 111 ). Furthermore, the hippocampal neurons derived from Cpe KO mice were rescued by the
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onset of diabetic cardiovascular complications than persistent hyperglycemia. Prior research has demonstrated that acute glycemic fluctuations could lead to CVD through oxidative stress and nuclear factor-κB activation ( 30 , 31 ). Moreover, a positive
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Department of Cell Physiology and Metabolism, Faculty of Medicine, University of Geneva, Geneva, Switzerland
Diabetes Center, Faculty of Medicine, University of Geneva, Geneva, Switzerland
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Diabetes Center, Faculty of Medicine, University of Geneva, Geneva, Switzerland
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Department of Cell Physiology and Metabolism, Faculty of Medicine, University of Geneva, Geneva, Switzerland
Diabetes Center, Faculty of Medicine, University of Geneva, Geneva, Switzerland
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Department of Cell Physiology and Metabolism, Faculty of Medicine, University of Geneva, Geneva, Switzerland
Diabetes Center, Faculty of Medicine, University of Geneva, Geneva, Switzerland
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Diabetes Center, Faculty of Medicine, University of Geneva, Geneva, Switzerland
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-β (TGF-β) family, has been identified as a stress-responsive cytokine and metabolic modulator whose expression is markedly elevated in response to stress conditions, aging, obesity, and diabetes ( 3 , 6 , 7 , 8 , 9 , 10 , 11 , 12 , 13 ). In
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Institute of Metabolism and Systems Research, University of Birmingham, Birmingham, UK
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Institute of Applied Health Research, University of Birmingham, Birmingham, UK
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Institute of Metabolism and Systems Research, University of Birmingham, Birmingham, UK
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Institute of Metabolism and Systems Research, University of Birmingham, Birmingham, UK
Medical Research Council London Institute of Medical Sciences, London, UK
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Institute of Metabolism and Systems Research, University of Birmingham, Birmingham, UK
NIHR Birmingham Biomedical Research Centre, University of Birmingham and University Hospitals Birmingham NHS Foundation Trust, Birmingham, UK
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dosage of which should be increased under stressors, such as infection, trauma or surgery, to prevent life-threatening adrenal crises, with increased stress doses recommended for patients with PAI and COVID-19 (1, 2 , 3 ). Despite advancements in
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Department of Biomedical Engineering, Faculty of Engineering, The Hong Kong Polytechnic University, Hong Kong, China
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Department of Biomedical Engineering, Faculty of Engineering, The Hong Kong Polytechnic University, Hong Kong, China
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dysfunction in type 2 diabetic mouse model via reducing oxidative stress, inflammation and pathological remodeling ( 7 ). However, despite the suggested therapeutic potential of Ang(1–7) for diabetes, whether the expression of endogenous Ang(1–7) and MAS-1 is
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development ( 24 , 25 , 26 ). As the main apolipoprotein in chylomicrons and LDL, APOA4 is related to lipid metabolism, insulin resistance, and oxidative stress ( 27 , 28 , 29 ). FTCD plays an important role in inducing the production of ROS, mitochondrial
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hallmark of critical illness, is the immediate initiation of multiple physiologic processes in an attempt to rebalance the complex dynamic equilibrium, commonly known as homeostasis. This so-called ‘stress response’ comprises many tightly controlled neural