Xiuzhen Hou
Xiuzhen Hou
Division 1 of Obstetrics, Cangzhou Central Hospital, Cangzhou, Hebei, China
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Junfeng Zhang
Junfeng Zhang
Division 1 of Obstetrics, Cangzhou Central Hospital, Cangzhou, Hebei, China
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Hehong Ma
Hehong Ma
Division 1 of Obstetrics, Cangzhou Central Hospital, Cangzhou, Hebei, China
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Ming Li
Ming Li
Division 1 of Obstetrics, Cangzhou Central Hospital, Cangzhou, Hebei, China
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Pei Wang
Pei Wang
Division 1 of Obstetrics, Cangzhou Central Hospital, Cangzhou, Hebei, China
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protect the blood-brain barrier against oxidative stress by activating the Nrf2 signaling to regulate inflammation-related genes such as GSH ( 25 ). H/R-induced injury was also implicated in hepatocytes by promoting apoptosis and oxidative stress through
Rosalie Cabry
Rosalie Cabry
Amiens University, Amiens, Haut-de-France, France
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Philippe Merviel
Philippe Merviel
Brest University, Brest, Bretagne, France
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Aicha Madkour
Aicha Madkour
Mohammed V University of Rabat, Reproductive Medicine, Rabat, Morocco
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Elodie Lefranc
Elodie Lefranc
Amiens University, Amiens, Haut-de-France, France
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Florence Scheffler
Florence Scheffler
Amiens University, Amiens, Haut-de-France, France
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Rachel Desailloud
Rachel Desailloud
Amiens University, Amiens, Haut-de-France, France
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Véronique Bach
Véronique Bach
Amiens University, Amiens, Haut-de-France, France
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Moncef Benkhalifa
Moncef Benkhalifa
Amiens University, Amiens, Haut-de-France, France
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disrupters, molecular mechanisms and detection methods . Therapie 2007 62 369 – 386 . ( https://doi.org/10.2515/therapie:2007062 ) 11 Bhardwaj JK Saraf P . Granulosa cell apoptosis by impairing antioxidant defense system and cellular integrity
Chunliang Yang
Chunliang Yang
Department of Integrated Traditional Chinese and Western Medicine, Union Hospital, Tongji Medical College, Huazhong University of Science and Technology, Wuhan, China
The Center for Biomedical Research, Tongji Hospital Research Building, Tongji Hospital, Tongji Medical College, Huazhong University of Science and Technology, Wuhan, China
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The Center for Biomedical Research, Tongji Hospital Research Building, Tongji Hospital, Tongji Medical College, Huazhong University of Science and Technology, Wuhan, China
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Junyi Li
Junyi Li
Department of Integrated Traditional Chinese and Western Medicine, Union Hospital, Tongji Medical College, Huazhong University of Science and Technology, Wuhan, China
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Fei Sun
Fei Sun
The Center for Biomedical Research, Tongji Hospital Research Building, Tongji Hospital, Tongji Medical College, Huazhong University of Science and Technology, Wuhan, China
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Haifeng Zhou
Haifeng Zhou
Department of Integrated Traditional Chinese and Western Medicine, Union Hospital, Tongji Medical College, Huazhong University of Science and Technology, Wuhan, China
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Jia Yang
Jia Yang
Department of Integrated Traditional Chinese and Western Medicine, Union Hospital, Tongji Medical College, Huazhong University of Science and Technology, Wuhan, China
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Chao Yang
Chao Yang
Department of Gerontology, Hubei Provincial Hospital of Integrated Chinese and Western Medicine, Wuhan, China
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signaling including IRS1, AKT and GSK-3β were hypophosphorylated in LKO hepatocytes, which subsequently blunt glucose uptake ( 21 ). SGK1 also exerts dramatic liver protection effect. Existing researches revealed that SGK1 alleviates hepatocyte apoptosis via
Julia Modesto Vicente
Julia Modesto Vicente
Department of Pharmacology, Faculty of Medical Sciences, State University of Campinas, Campinas, Brazil
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Junia Carolina Santos-Silva
Junia Carolina Santos-Silva
Department of Pharmacology, Faculty of Medical Sciences, State University of Campinas, Campinas, Brazil
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Caio Jordão Teixeira
Caio Jordão Teixeira
Department of Pharmacology, Faculty of Medical Sciences, State University of Campinas, Campinas, Brazil
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Dailson Nogueira de Souza
Dailson Nogueira de Souza
Department of Pharmacology, Faculty of Medical Sciences, State University of Campinas, Campinas, Brazil
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Jean Franciesco Vettorazzi
Jean Franciesco Vettorazzi
Department of Structural and Functional Biology, Institute of Biology, State University of Campinas, Campinas, Brazil
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Fabiola Sales Furtuoso
Fabiola Sales Furtuoso
Department of Pharmacology, Faculty of Medical Sciences, State University of Campinas, Campinas, Brazil
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Isabel Gouveia Adabo
Isabel Gouveia Adabo
Department of Pharmacology, Faculty of Medical Sciences, State University of Campinas, Campinas, Brazil
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Fabio Takeo Sato
Fabio Takeo Sato
Department of Genetics, Evolution, Microbiology and Immunology, Institute of Biology, State University of Campinas, Campinas, Brazil
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Marco Aurélio Ramirez Vinolo
Marco Aurélio Ramirez Vinolo
Department of Genetics, Evolution, Microbiology and Immunology, Institute of Biology, State University of Campinas, Campinas, Brazil
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Everardo Magalhães Carneiro
Everardo Magalhães Carneiro
Department of Structural and Functional Biology, Institute of Biology, State University of Campinas, Campinas, Brazil
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Silvana Bordin
Silvana Bordin
Department of Physiology and Biophysics, Institute of Biomedical Sciences, University of Sao Paulo, Sao Paulo, Brazil
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Gabriel Forato Anhê
Gabriel Forato Anhê
Department of Pharmacology, Faculty of Medical Sciences, State University of Campinas, Campinas, Brazil
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β-cells. The permanent increase in intracellular calcium, however, increases the apoptosis and decreases pancreatic β-cell mass during adulthood, leading these transgenic mice to a switch to a hyperglycemic phenotype later in life ( 28 ). This
Martine Cohen-Solal
Martine Cohen-Solal
Department of Skeletal Diseases, INSERM U1132 & Université de Paris, Hôpital Lariboisière, Paris, France
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Thomas Funck-Brentano
Thomas Funck-Brentano
Department of Skeletal Diseases, INSERM U1132 & Université de Paris, Hôpital Lariboisière, Paris, France
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Pablo Ureña Torres
Pablo Ureña Torres
AURA Nord, Saint Ouen, France
Department of Renal Physiology, Necker Hospital, Université de Paris, Paris, France
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Department of Renal Physiology, Necker Hospital, Université de Paris, Paris, France
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most important physiologic regulators of calcium and phosphate metabolism, together with the kidney. In bone, low extracellular phosphate concentration is crucial for apoptosis of mature chondrocytes in the growth plate and the cascade of events leading
Selina Mäkinen
Selina Mäkinen
Minerva Foundation Institute for Medical Research, Helsinki, Finland
Department of Medicine, University of Helsinki, Helsinki University Central Hospital, Helsinki, Finland
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Department of Medicine, University of Helsinki, Helsinki University Central Hospital, Helsinki, Finland
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Neeta Datta
Neeta Datta
Minerva Foundation Institute for Medical Research, Helsinki, Finland
Department of Medicine, University of Helsinki, Helsinki University Central Hospital, Helsinki, Finland
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Department of Medicine, University of Helsinki, Helsinki University Central Hospital, Helsinki, Finland
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Yen H Nguyen
Yen H Nguyen
Minerva Foundation Institute for Medical Research, Helsinki, Finland
Department of Medicine, University of Helsinki, Helsinki University Central Hospital, Helsinki, Finland
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Department of Medicine, University of Helsinki, Helsinki University Central Hospital, Helsinki, Finland
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Petro Kyrylenko
Petro Kyrylenko
Minerva Foundation Institute for Medical Research, Helsinki, Finland
Department of Medicine, University of Helsinki, Helsinki University Central Hospital, Helsinki, Finland
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Department of Medicine, University of Helsinki, Helsinki University Central Hospital, Helsinki, Finland
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Markku Laakso
Markku Laakso
Institute of Clinical Medicine, Internal Medicine, University of Eastern Finland, Kuopio, Finland
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Heikki A Koistinen
Heikki A Koistinen
Minerva Foundation Institute for Medical Research, Helsinki, Finland
Department of Medicine, University of Helsinki, Helsinki University Central Hospital, Helsinki, Finland
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Department of Medicine, University of Helsinki, Helsinki University Central Hospital, Helsinki, Finland
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skeletal muscle protein synthesis and breakdown, as well as apoptosis ( 15 ). Inhibition of Akt phosphorylation was also responsible for simvastatin-induced mitochondrial dysfunction in mouse C2C12 muscle cells ( 16 ). Potential mechanisms for simvastatin
Qinglei Yin
Qinglei Yin
Department of Endocrine and Metabolic Diseases, Shanghai Institute of Endocrine and Metabolic Diseases, Ruijin Hospital, Shanghai Jiao Tong University School of Medicine, Shanghai, China
Shanghai National Clinical Research Center for Metabolic Diseases, Key Laboratory for Endocrine and Metabolic Diseases of the National Health Commission of the PR China, Shanghai National Center for Translational Medicine, Ruijin Hospital, Shanghai Jiao Tong University School of Medicine, Shanghai, China
Guangdong Geriatric Institute, Guangdong Provincial People’s Hospital, Guangdong Academy of Medical Sciences, Guangzhou, China
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Shanghai National Clinical Research Center for Metabolic Diseases, Key Laboratory for Endocrine and Metabolic Diseases of the National Health Commission of the PR China, Shanghai National Center for Translational Medicine, Ruijin Hospital, Shanghai Jiao Tong University School of Medicine, Shanghai, China
Guangdong Geriatric Institute, Guangdong Provincial People’s Hospital, Guangdong Academy of Medical Sciences, Guangzhou, China
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Zhou Jin
Zhou Jin
Department of Endocrine and Metabolic Diseases, Shanghai Institute of Endocrine and Metabolic Diseases, Ruijin Hospital, Shanghai Jiao Tong University School of Medicine, Shanghai, China
Shanghai National Clinical Research Center for Metabolic Diseases, Key Laboratory for Endocrine and Metabolic Diseases of the National Health Commission of the PR China, Shanghai National Center for Translational Medicine, Ruijin Hospital, Shanghai Jiao Tong University School of Medicine, Shanghai, China
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Shanghai National Clinical Research Center for Metabolic Diseases, Key Laboratory for Endocrine and Metabolic Diseases of the National Health Commission of the PR China, Shanghai National Center for Translational Medicine, Ruijin Hospital, Shanghai Jiao Tong University School of Medicine, Shanghai, China
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Yulin Zhou
Yulin Zhou
Department of Endocrine and Metabolic Diseases, Shanghai Institute of Endocrine and Metabolic Diseases, Ruijin Hospital, Shanghai Jiao Tong University School of Medicine, Shanghai, China
Shanghai National Clinical Research Center for Metabolic Diseases, Key Laboratory for Endocrine and Metabolic Diseases of the National Health Commission of the PR China, Shanghai National Center for Translational Medicine, Ruijin Hospital, Shanghai Jiao Tong University School of Medicine, Shanghai, China
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Shanghai National Clinical Research Center for Metabolic Diseases, Key Laboratory for Endocrine and Metabolic Diseases of the National Health Commission of the PR China, Shanghai National Center for Translational Medicine, Ruijin Hospital, Shanghai Jiao Tong University School of Medicine, Shanghai, China
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Dalong Song
Dalong Song
Guangdong Geriatric Institute, Guangdong Provincial People’s Hospital, Guangdong Academy of Medical Sciences, Guangzhou, China
Reproductive Medicine Center, Guangdong Provincial People’s Hospital, Guangdong Academy of Medical Sciences, Guangzhou, China
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Reproductive Medicine Center, Guangdong Provincial People’s Hospital, Guangdong Academy of Medical Sciences, Guangzhou, China
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Chenyang Fu
Chenyang Fu
Department of Endocrine and Metabolic Diseases, Shanghai Institute of Endocrine and Metabolic Diseases, Ruijin Hospital, Shanghai Jiao Tong University School of Medicine, Shanghai, China
Shanghai National Clinical Research Center for Metabolic Diseases, Key Laboratory for Endocrine and Metabolic Diseases of the National Health Commission of the PR China, Shanghai National Center for Translational Medicine, Ruijin Hospital, Shanghai Jiao Tong University School of Medicine, Shanghai, China
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Shanghai National Clinical Research Center for Metabolic Diseases, Key Laboratory for Endocrine and Metabolic Diseases of the National Health Commission of the PR China, Shanghai National Center for Translational Medicine, Ruijin Hospital, Shanghai Jiao Tong University School of Medicine, Shanghai, China
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FengJiao Huang
FengJiao Huang
Reproductive Medicine Center, Guangdong Provincial People’s Hospital, Guangdong Academy of Medical Sciences, Guangzhou, China
Division of Endocrinology, Department of Internal Medicine, The First Affiliated Hospital of Zhengzhou University, Zhengzhou, Henan, China
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Division of Endocrinology, Department of Internal Medicine, The First Affiliated Hospital of Zhengzhou University, Zhengzhou, Henan, China
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Shu Wang
Shu Wang
Department of Endocrine and Metabolic Diseases, Shanghai Institute of Endocrine and Metabolic Diseases, Ruijin Hospital, Shanghai Jiao Tong University School of Medicine, Shanghai, China
Shanghai National Clinical Research Center for Metabolic Diseases, Key Laboratory for Endocrine and Metabolic Diseases of the National Health Commission of the PR China, Shanghai National Center for Translational Medicine, Ruijin Hospital, Shanghai Jiao Tong University School of Medicine, Shanghai, China
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Shanghai National Clinical Research Center for Metabolic Diseases, Key Laboratory for Endocrine and Metabolic Diseases of the National Health Commission of the PR China, Shanghai National Center for Translational Medicine, Ruijin Hospital, Shanghai Jiao Tong University School of Medicine, Shanghai, China
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mRNA with target prediction of differentially expressed lncRNAs, and NRCAM and JUNB were identified. Previous reports showed that NRCAM can activate ERK and AKT pathway to inhibit cell apoptosis and promote cell proliferation ( 38 , 39 , 40
Manon Engels
Manon Engels
Department of Paediatrics, Radboud Amalia Children’s Hospital, Radboud university medical center, Nijmegen, The Netherlands
Laboratory Medicine, Radboud Institute for Molecular Life Sciences (RIMLS), Radboud university medical center, Nijmegen, The Netherlands
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Laboratory Medicine, Radboud Institute for Molecular Life Sciences (RIMLS), Radboud university medical center, Nijmegen, The Netherlands
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Paul N Span
Paul N Span
Radiation Oncology, Radiotherapy and OncoImmunology Laboratory, RIMLS, Radboud university medical center, Nijmegen, The Netherlands
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Rod T Mitchell
Rod T Mitchell
MRC Centre for Reproductive Health, University of Edinburgh, The Queen’s Medical Research Institute, Edinburgh, UK
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Joop J T M Heuvel
Joop J T M Heuvel
Laboratory Medicine, Radboud Institute for Molecular Life Sciences (RIMLS), Radboud university medical center, Nijmegen, The Netherlands
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Monica A Marijnissen-van Zanten
Monica A Marijnissen-van Zanten
Department of Pathology, Radboud university medical center, Nijmegen, The Netherlands
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Antonius E van Herwaarden
Antonius E van Herwaarden
Laboratory Medicine, Radboud Institute for Molecular Life Sciences (RIMLS), Radboud university medical center, Nijmegen, The Netherlands
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Christina A Hulsbergen-van de Kaa
Christina A Hulsbergen-van de Kaa
Department of Pathology, Radboud university medical center, Nijmegen, The Netherlands
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Egbert Oosterwijk
Egbert Oosterwijk
Department of Urology, Radboud university medical center, Nijmegen, The Netherlands
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Nike M Stikkelbroeck
Nike M Stikkelbroeck
Department of Internal Medicine, Radboud university medical center, Nijmegen, The Netherlands
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Lee B Smith
Lee B Smith
MRC Centre for Reproductive Health, University of Edinburgh, The Queen’s Medical Research Institute, Edinburgh, UK
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Fred C G J Sweep
Fred C G J Sweep
Laboratory Medicine, Radboud Institute for Molecular Life Sciences (RIMLS), Radboud university medical center, Nijmegen, The Netherlands
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Hedi L Claahsen-van der Grinten
Hedi L Claahsen-van der Grinten
Department of Paediatrics, Radboud Amalia Children’s Hospital, Radboud university medical center, Nijmegen, The Netherlands
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factor GATA-6, cell proliferation, apoptosis, and apoptosis-related proteins Bcl-2 and Bax in human fetal testis . Journal of Clinical Endocrinology and Metabolism 2003 88 1858 – 1865 . ( doi:10.1210/jc.2002-021647 ) 10.1210/jc.2002-021647 38
Anello Marcello Poma
Anello Marcello Poma
Department of Surgical, Medical, Molecular Pathology and Critical Area, University of Pisa, Pisa, Italy
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Riccardo Giannini
Riccardo Giannini
Department of Surgical, Medical, Molecular Pathology and Critical Area, University of Pisa, Pisa, Italy
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Paolo Piaggi
Paolo Piaggi
National Institute of Diabetes and Digestive and Kidney Diseases, National Institutes of Health, Phoenix, Arizona, USA
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Clara Ugolini
Clara Ugolini
Department of Laboratory Medicine, Section of Pathology, Azienda Ospedaliero-Universitaria Pisana, Pisa, Italy
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Gabriele Materazzi
Gabriele Materazzi
Department of Surgical, Medical, Molecular Pathology and Critical Area, University of Pisa, Pisa, Italy
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Paolo Miccoli
Paolo Miccoli
Department of Surgical, Medical, Molecular Pathology and Critical Area, University of Pisa, Pisa, Italy
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Paolo Vitti
Paolo Vitti
Department of Clinical and Experimental Medicine, University of Pisa, Pisa, Italy
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Fulvio Basolo
Fulvio Basolo
Department of Surgical, Medical, Molecular Pathology and Critical Area, University of Pisa, Pisa, Italy
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). Furthermore, functional studies in breast cancer highlighted an important role of SDPR in apoptosis promotion ( 36 ) and in the suppression of the epithelial–mesenchymal transition ( 54 ). Lastly, SLC26A4 encodes for pendrin, a chloride–iodide transporter
Fei Wu
Fei Wu
Department of Nuclear Medicine, Yancheng City No. 1 People’s Hospital, Yancheng, China
Department of Nuclear Medicine, The Affiliated Hospital of Jiangsu University, Zhenjiang, China
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Department of Nuclear Medicine, The Affiliated Hospital of Jiangsu University, Zhenjiang, China
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Chaoming Mao
Chaoming Mao
Department of Nuclear Medicine, The Affiliated Hospital of Jiangsu University, Zhenjiang, China
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Xiao Mou
Xiao Mou
Department of Nuclear Medicine, The Affiliated Hospital of Jiangsu University, Zhenjiang, China
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Chengcheng Xu
Chengcheng Xu
Department of Nuclear Medicine, The Affiliated Hospital of Jiangsu University, Zhenjiang, China
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Tingting Zheng
Tingting Zheng
Department of Nuclear Medicine, The Affiliated Hospital of Jiangsu University, Zhenjiang, China
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Ling Bu
Ling Bu
Department of Clinical Medicine, Jiangsu Health Vocational College, Nanjing, China
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Xuan Luo
Xuan Luo
Department of Nuclear Medicine, The Affiliated Hospital of Jiangsu University, Zhenjiang, China
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Qingyan Lu
Qingyan Lu
Department of Laboratory Medicine, Xuzhou Central Hospital, Xuzhou, China
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Xuefeng Wang
Xuefeng Wang
Department of Nuclear Medicine, The Affiliated Hospital of Jiangsu University, Zhenjiang, China
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://doi.org/10.1210/jc.2005-1475 ) 19 Liu H Zheng T Mao Y Xu C Wu F Bu L Mou X Zhou Y Yuan G Wang S gammadelta Tau cells enhance B cells for antibody production in Hashimoto’s thyroiditis, and retinoic acid induces apoptosis of the
