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have been shown to interact with nuclear receptors for sex steroid hormones ( 41 ), which play a critical role in brain organization, as well as peroxisome proliferator-activated receptor gamma (PPARγ) ( 32 , 42 ), an important regulator of
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.102 ) 10.1038/ijo.2015.102 56 Petrick JL Falk RT Hyland PL Caron P Pfeiffer RM Wood SN Dawsey SM Abnet CC Taylor PR Guillemette C , et al . Association between circulating levels of sex steroid hormones and esophageal adenocarcinoma in the FINBAR Study
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supplement, is therefore in reality, a precursor to a potent steroid hormone influencing a wide range of cellular pathways in organs that are highly relevant to the effects of critical illness and may exert its beneficial effects on acute inflammation
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. Environmental Biology of Fishes 2003 321 – 332 . ( https://doi.org/10.1023/A:1027381304091 ) 13 Vazirzadeh A Amiri BM Fostier A . Ovarian development and related changes in steroid hormones in female wild common carp ( Cyprinus carpio carpio
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Department of Nuclear Medicine, CHU de Bordeaux, Pessac, France
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steroid hormone synthesis. It has been suggested that there is an interesting interplay between RA and oestrogen signalling in breast cancer cells particularly with opposite actions on cell proliferation. RARα could be an integral part of the ERα
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. ( doi:10.1210/endo.132.6.8504753 ) 10.1210/endo.132.6.8504753 7 Payne AH Hales DB. Overview of steroidogenic enzymes in the pathway from cholesterol to active steroid hormones . Endocrine Reviews 2004 25 947 – 970 . ( doi:10.1210/er
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and steroid hormones ( Fig. 1 ). Herein, we will discuss the established and putative links between these obesity-generated defects and cancer progression in the knowledge that, in all likelihood, multiple hits from several obesogenic factors are
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Alemany M . Steroid hormones interrelationships in the metabolic syndrome: an introduction to the ponderostat hypothesis . Hormones 2012 11 272 – 289 . ( doi:10.14310/horm.2002.1356 ). 80 Hube F Lietz U Igel M Jensen PB Tornqvist H
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Morbid Obesity Centre, Department of Nutrition, Norwegian National Advisory Unit on Familial Hypercholesterolemia, Department of Gynecology, Institute of Clinical Medicine, Department of Endocrinology, Vestfold Hospital Trust, PO Box 2168, 3103 Tønsberg, Norway
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Morbid Obesity Centre, Department of Nutrition, Norwegian National Advisory Unit on Familial Hypercholesterolemia, Department of Gynecology, Institute of Clinical Medicine, Department of Endocrinology, Vestfold Hospital Trust, PO Box 2168, 3103 Tønsberg, Norway
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Morbid Obesity Centre, Department of Nutrition, Norwegian National Advisory Unit on Familial Hypercholesterolemia, Department of Gynecology, Institute of Clinical Medicine, Department of Endocrinology, Vestfold Hospital Trust, PO Box 2168, 3103 Tønsberg, Norway
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gonadotropins and steroid hormones were analysed in the early follicular phase (cycle day 2–3), while in anovulatory women the analyses were performed randomly. Apo-A1, Apo-B and C-reactive protein (CRP) were analysed by an immunturbidimetric method using
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steroid hormones, which pass cell membranes freely and can reach any targets in the body by diffusion through extravascular fluid. Nor was there objection to classing as hormones many agents that act within tissues, such as prostaglandins within the uterus