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Julia Otten Department of Public Health and Clinical Medicine, Umeå University, Umeå, Sweden

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Andreas Stomby Department of Public Health and Clinical Medicine, Umeå University, Umeå, Sweden
Region Jönköping County, Jönköping, Sweden

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Maria Waling Department of Food, Nutrition and Culinary Science, Umeå University, Umeå, Sweden

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Elin Chorell Department of Public Health and Clinical Medicine, Umeå University, Umeå, Sweden

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Mats Ryberg Department of Public Health and Clinical Medicine, Umeå University, Umeå, Sweden

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Michael Svensson Department of Community Medicine and Rehabilitation, Section for Sports Medicine, Umeå University, Umeå Sweden

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Jens Juul Holst NNF Center for Basic Metabolic Research and Department of Biomedical Sciences, University of Copenhagen, Copenhagen, Denmark

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Tommy Olsson Department of Public Health and Clinical Medicine, Umeå University, Umeå, Sweden

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liver, increases plasma amino acids levels, and causes alpha-cell proliferation and hypersecretion of glucagon ( 5 ). It is a matter of debate which amino acids are responsible for signaling to alpha cells to increase glucagon secretion. Alanine and

Article Type:
Research Article
DOI:
https://doi.org/10.1530/EC-21-0171
Volume/Issue:
Volume 10: Issue 9
Open access
Jana Ernst Department of Anatomy and Cell Biology, Faculty of Medicine, Martin Luther University Halle-Wittenberg, Halle (Saale), Germany

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Urszula Grabiec Department of Anatomy and Cell Biology, Faculty of Medicine, Martin Luther University Halle-Wittenberg, Halle (Saale), Germany

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Kathrin Falk Department of Anatomy and Cell Biology, Faculty of Medicine, Martin Luther University Halle-Wittenberg, Halle (Saale), Germany

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Faramarz Dehghani Department of Anatomy and Cell Biology, Faculty of Medicine, Martin Luther University Halle-Wittenberg, Halle (Saale), Germany

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Kristina Schaedlich Department of Anatomy and Cell Biology, Faculty of Medicine, Martin Luther University Halle-Wittenberg, Halle (Saale), Germany

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adipocyte proliferation and differentiation, it furthermore positively affects insulin-stimulated but not basal glucose uptake in 3T3-derived adipocytes ( 38 , 39 , 40 ). Accordingly, an increase of glucose uptake after activation of CB 1 was demonstrated

Article Type:
Research Article
DOI:
https://doi.org/10.1530/EC-19-0548
Volume/Issue:
Volume 9: Issue 2
Open access
Natalio García-Honduvilla Department of Medicine and Medical Specialities, Faculty of Medicine and Health Sciences, University of Alcalá, Alcalá de Henares, Madrid, Spain
Networking Biomedical Research Center on Bioengineering, Biomaterials and Nanomedicine (CIBER-BBN), Madrid, Spain
Ramón y Cajal Institute of Sanitary Research (IRYCIS), Madrid, Spain
University Center of Defense of Madrid (CUD-ACD), Madrid, Spain

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Alberto Cifuentes Department of Medicine and Medical Specialities, Faculty of Medicine and Health Sciences, University of Alcalá, Alcalá de Henares, Madrid, Spain
Networking Biomedical Research Center on Bioengineering, Biomaterials and Nanomedicine (CIBER-BBN), Madrid, Spain
Ramón y Cajal Institute of Sanitary Research (IRYCIS), Madrid, Spain

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Miguel A Ortega Department of Medicine and Medical Specialities, Faculty of Medicine and Health Sciences, University of Alcalá, Alcalá de Henares, Madrid, Spain
Networking Biomedical Research Center on Bioengineering, Biomaterials and Nanomedicine (CIBER-BBN), Madrid, Spain
Ramón y Cajal Institute of Sanitary Research (IRYCIS), Madrid, Spain

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Marta Pastor Biopraxis Research AIE, Álava, Spain

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Garazi Gainza Biopraxis Research AIE, Álava, Spain

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Eusebio Gainza Biopraxis Research AIE, Álava, Spain

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Julia Buján Department of Medicine and Medical Specialities, Faculty of Medicine and Health Sciences, University of Alcalá, Alcalá de Henares, Madrid, Spain
Networking Biomedical Research Center on Bioengineering, Biomaterials and Nanomedicine (CIBER-BBN), Madrid, Spain
Ramón y Cajal Institute of Sanitary Research (IRYCIS), Madrid, Spain
University Center of Defense of Madrid (CUD-ACD), Madrid, Spain

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Melchor Álvarez-Mon Department of Medicine and Medical Specialities, Faculty of Medicine and Health Sciences, University of Alcalá, Alcalá de Henares, Madrid, Spain
Networking Biomedical Research Center on Bioengineering, Biomaterials and Nanomedicine (CIBER-BBN), Madrid, Spain
Ramón y Cajal Institute of Sanitary Research (IRYCIS), Madrid, Spain
University Center of Defense of Madrid (CUD-ACD), Madrid, Spain
Immune System Diseases-Rheumatology and Oncology Service, University Hospital Príncipe de Asturias, Alcalá de Henares, Madrid, Spain

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ulcers ( 8 ). Inadequate secretion of ECM proteins ( 9 ), dysregulation of macrophage activity ( 10 , 11 ), proliferation and increased apoptosis of cutaneous fibroblasts, impaired angiogenesis, and re-epithelialization ( 12 , 13 ) have all been

Article Type:
Research Article
DOI:
https://doi.org/10.1530/EC-18-0117
Volume/Issue:
Volume 7: Issue 4
Open access
Fan Zhang Department of Endocrinology and Metabolism, Institute of Endocrinology, Liaoning Provincial Key Laboratory of Endocrine Diseases, The First Affiliated Hospital of China Medical University, Shenyang, Liaoning, China

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Jian Chen Department of Endocrinology and Metabolism, Institute of Endocrinology, Liaoning Provincial Key Laboratory of Endocrine Diseases, The First Affiliated Hospital of China Medical University, Shenyang, Liaoning, China

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Xinyue Lin Department of Endocrinology and Metabolism, Institute of Endocrinology, Liaoning Provincial Key Laboratory of Endocrine Diseases, The First Affiliated Hospital of China Medical University, Shenyang, Liaoning, China

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Shiqiao Peng Department of Endocrinology and Metabolism, Institute of Endocrinology, Liaoning Provincial Key Laboratory of Endocrine Diseases, The First Affiliated Hospital of China Medical University, Shenyang, Liaoning, China

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Xiaohui Yu Department of Endocrinology and Metabolism, Institute of Endocrinology, Liaoning Provincial Key Laboratory of Endocrine Diseases, The First Affiliated Hospital of China Medical University, Shenyang, Liaoning, China

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Zhongyan Shan Department of Endocrinology and Metabolism, Institute of Endocrinology, Liaoning Provincial Key Laboratory of Endocrine Diseases, The First Affiliated Hospital of China Medical University, Shenyang, Liaoning, China

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Weiping Teng Department of Endocrinology and Metabolism, Institute of Endocrinology, Liaoning Provincial Key Laboratory of Endocrine Diseases, The First Affiliated Hospital of China Medical University, Shenyang, Liaoning, China

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series of experiments to explore the mechanism in it ( 6 ). The current study explored the mechanism further from other aspect. THs play a crucial role in fetal brain development and are involved in the proliferation, differentiation, maturation and

Article Type:
Research Article
DOI:
https://doi.org/10.1530/EC-18-0069
Volume/Issue:
Volume 7: Issue 5
Open access
Nassim Ghaffari-Tabrizi-Wizsy Otto Loewi Research Center – Immunology and Pathophysiology, Medical University of Graz, Graz, Austria

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Christina Angelika Passegger Otto Loewi Research Center – Immunology and Pathophysiology, Medical University of Graz, Graz, Austria

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Laura Nebel Otto Loewi Research Center – Immunology and Pathophysiology, Medical University of Graz, Graz, Austria

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Fabian Krismer Otto Loewi Research Center – Immunology and Pathophysiology, Medical University of Graz, Graz, Austria

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Gudrun Herzer-Schneidhofer Otto Loewi Research Center – Immunology and Pathophysiology, Medical University of Graz, Graz, Austria

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Gert Schwach Otto Loewi Research Center – Immunology and Pathophysiology, Medical University of Graz, Graz, Austria

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Roswitha Pfragner Otto Loewi Research Center – Immunology and Pathophysiology, Medical University of Graz, Graz, Austria

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–30%) ( 1 ). In about 25% of cases, MTC is caused by inherited germline-activating mutations of the ‘rearranged during transfection’ (RET) proto-oncogene, which constitutively activates cellular signalling, resulting in persistent cell proliferation and MTC

Article Type:
Research Article
DOI:
https://doi.org/10.1530/EC-18-0431
Volume/Issue:
Volume 8: Issue 5
Open access
V Kenan Çelik Department of Biochemistry, Cumhuriyet University School of Medicine, Sivas, Turkey

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Sercan Kapancık Department of Biochemistry, Cumhuriyet University School of Medicine, Sivas, Turkey

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Turgut Kaçan Department of Medical Oncology, High Specialized Education and Research Hospital, Bursa, Turkey

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Selen Baloglu Kaçan Department of Internal Medicine, Afyonkarahisar State Hospital, Afyon, Turkey

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Serkan Kapancık Department of Biochemistry, Cumhuriyet University School of Medicine, Sivas, Turkey

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Hasan Kılıçgün Department of Nutrition and Dietetics, Health Science Faculty, University of Erzincan, Erzincan, Turkey

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positively charged aliphatic amines. As biological routers, polyamines interact with different macromolecules such as DNA, RNA and proteins and have the ability to regulate their biochemical activities. Polyamines direct cell proliferation by regulating

Article Type:
Research Article
DOI:
https://doi.org/10.1530/EC-17-0137
Volume/Issue:
Volume 6: Issue 8
Open access
Magaly Zappa Univ Paris Diderot, Sorbonne Paris Cité, Paris, France
INSERM, UMR1149 CRI, Paris, France
Department of Radiology, AP-HP, Clichy, France

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Olivia Hentic Department of Gastroenterology and Pancreatology, AP-HP, Clichy, France

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Marie-Pierre Vullierme Department of Radiology, AP-HP, Clichy, France

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Matthieu Lagadec Department of Radiology, AP-HP, Clichy, France

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Maxime Ronot Univ Paris Diderot, Sorbonne Paris Cité, Paris, France
INSERM, UMR1149 CRI, Paris, France
Department of Radiology, AP-HP, Clichy, France

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Philippe Ruszniewski Univ Paris Diderot, Sorbonne Paris Cité, Paris, France
INSERM, UMR1149 CRI, Paris, France
Department of Gastroenterology and Pancreatology, AP-HP, Clichy, France

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Valérie Vilgrain Univ Paris Diderot, Sorbonne Paris Cité, Paris, France
INSERM, UMR1149 CRI, Paris, France
Department of Radiology, AP-HP, Clichy, France

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the proliferation marker Ki67. The system that was recently proposed for GEP-NETs by the European Neuroendocrine Tumor Society (ENETS) and recommended by WHO uses either mitotic rate or the Ki67 labelling index ( 2 ). Except for neuroendocrine

Article Type:
Research Article
DOI:
https://doi.org/10.1530/EC-16-0092
Volume/Issue:
Volume 6: Issue 1
Open access
Giovanni Tulipano Unit of Pharmacology, Department of Molecular and Translational Medicine, University of Brescia, Brescia, Italy

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some GH-secreting tumor cells responsive to an AMPK activator. These factors may include proliferation rate, activation of mTOR pathway downstream of cell surface growth factor receptors, basal AMPK activity and differences in cellular bioenergetics

Article Type:
Review Article
DOI:
https://doi.org/10.1530/EC-19-0482
Volume/Issue:
Volume 9: Issue 2
Open access
Stavroula A Paschou Division of Endocrinology, Metabolism and Diabetes, First Department of Pediatrics, ‘Aghia Sophia’ Children’s Hospital, Medical School, National and Kapodistrian University of Athens, Athens, Greece

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Nektaria Papadopoulou-Marketou Division of Endocrinology, Metabolism and Diabetes, First Department of Pediatrics, ‘Aghia Sophia’ Children’s Hospital, Medical School, National and Kapodistrian University of Athens, Athens, Greece

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George P Chrousos Division of Endocrinology, Metabolism and Diabetes, First Department of Pediatrics, ‘Aghia Sophia’ Children’s Hospital, Medical School, National and Kapodistrian University of Athens, Athens, Greece

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Christina Kanaka-Gantenbein Division of Endocrinology, Metabolism and Diabetes, First Department of Pediatrics, ‘Aghia Sophia’ Children’s Hospital, Medical School, National and Kapodistrian University of Athens, Athens, Greece

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, this complex recognition is not capable itself of stimulating the naive T lymphocyte for proliferation and differentiation into an active T lymphocyte, but costimulatory molecules are necessary. Such molecules are the B7.1 (CD80) and B7.2 (CD86

Article Type:
Review Article
DOI:
https://doi.org/10.1530/EC-17-0347
Volume/Issue:
Volume 7: Issue 1
Open access
Maria Angela D'amico Section of Human Morphology, Department of Medicine and Aging Sciences, G. d'Annunzio University of Chieti–Pescara, Via Dei Vestini 31, 66013 Chieti, Italy

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Barbara Ghinassi Section of Human Morphology, Department of Medicine and Aging Sciences, G. d'Annunzio University of Chieti–Pescara, Via Dei Vestini 31, 66013 Chieti, Italy

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Pascal Izzicupo Section of Human Morphology, Department of Medicine and Aging Sciences, G. d'Annunzio University of Chieti–Pescara, Via Dei Vestini 31, 66013 Chieti, Italy

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Lamberto Manzoli Section of Human Morphology, Department of Medicine and Aging Sciences, G. d'Annunzio University of Chieti–Pescara, Via Dei Vestini 31, 66013 Chieti, Italy

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A Di Baldassarre Section of Human Morphology, Department of Medicine and Aging Sciences, G. d'Annunzio University of Chieti–Pescara, Via Dei Vestini 31, 66013 Chieti, Italy

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(23, 24) . Vasostatins have been linked to vasculogenesis and remodeling (12) . In contrast to catestatin, vasostatin inhibits VEGF-induced endothelial cell proliferation and migration and the formation of capillary-like structures (25) . However

Article Type:
Review Article
DOI:
https://doi.org/10.1530/EC-14-0027
Volume/Issue:
Volume 3: Issue 2
Open access