School of Medicine, Western Sydney University, Sydney, Australia
Search for other papers by Prishila Fookeerah in
Google Scholar
PubMed
Westmead Clinical School, University of Sydney, Sydney, Australia
Search for other papers by Winny Varikatt in
Google Scholar
PubMed
Westmead Clinical School, University of Sydney, Sydney, Australia
Search for other papers by Meena Shingde in
Google Scholar
PubMed
Department of Neurosurgery, Westmead Hospital, Sydney, Australia
Search for other papers by Mark A J Dexter in
Google Scholar
PubMed
School of Medicine, Western Sydney University, Sydney, Australia
Search for other papers by Mark McLean in
Google Scholar
PubMed
disinhibition of prolactin secretion from normal pituitary tissue. One tumour co-expressed TSH, GH and PRL without evidence of hyperthyroidism or hyperprolactinaemia clinically or biochemically. The tumours causing GH excess expressed both SSTR2 and SSTR5. SSTR2
Search for other papers by Min Li in
Google Scholar
PubMed
Search for other papers by Ying Chen in
Google Scholar
PubMed
Search for other papers by Jingjing Jiang in
Google Scholar
PubMed
Search for other papers by Yan Lu in
Google Scholar
PubMed
Search for other papers by Zhiyi Song in
Google Scholar
PubMed
Search for other papers by Shengjie Zhang in
Google Scholar
PubMed
Search for other papers by Chao Sun in
Google Scholar
PubMed
Search for other papers by Hao Ying in
Google Scholar
PubMed
Search for other papers by Xiaofang Fan in
Google Scholar
PubMed
Search for other papers by Yuping Song in
Google Scholar
PubMed
Search for other papers by Jialin Yang in
Google Scholar
PubMed
Search for other papers by Lin Zhao in
Google Scholar
PubMed
Hospital (Shanghai, China). Hyperthyroidism was diagnosed by typical clinical presentation, elevated serum TH, reduced thyroid-stimulating hormone (TSH) and elevated serum TSH receptor antibody (TRAb) levels. One hundred healthy controls were recruited from
Search for other papers by Qian Yang in
Google Scholar
PubMed
Search for other papers by Wencai Ke in
Google Scholar
PubMed
Search for other papers by Fanfan Pan in
Google Scholar
PubMed
Search for other papers by Xinmei Huang in
Google Scholar
PubMed
Search for other papers by Jun Liu in
Google Scholar
PubMed
Search for other papers by Bingbing Zha in
Google Scholar
PubMed
(free thyroxine (FT4), thyrotropin (TSH), TPO antibodies (TPOAb), TG antibodies (TGAb), and thyrotropin receptor antibodies (TRAb)), and RAIU (3 h RAIU and 24 h RAIU). A flowchart of the study is shown in Fig. 1 . Patients were excluded from the study
Search for other papers by Ruth Percik in
Google Scholar
PubMed
Search for other papers by Sherwin Criseno in
Google Scholar
PubMed
Search for other papers by Safwaan Adam in
Google Scholar
PubMed
Search for other papers by Kate Young in
Google Scholar
PubMed
Royal Marsden Hospital, London, UK
Search for other papers by Daniel L Morganstein in
Google Scholar
PubMed
-stimulating hormone (TSH) and gonadotrophin deficiency ( 12 , 18 , 19 ). We, therefore, propose that hypophysitis should be reserved to describe either the symptomatic phase with headache or MRI findings of enlarged pituitary, whilst ICPI-induced hypopituitarism is
Search for other papers by Xiaomin Nie in
Google Scholar
PubMed
Search for other papers by Yiting Xu in
Google Scholar
PubMed
Search for other papers by Xiaojing Ma in
Google Scholar
PubMed
Search for other papers by Yun Shen in
Google Scholar
PubMed
Search for other papers by Yufei Wang in
Google Scholar
PubMed
Search for other papers by Yuqian Bao in
Google Scholar
PubMed
(FT4) and thyroid-stimulating hormone (TSH). The reference range and intra- and interassay coefficients for FT3 were 3.10–6.80 pmol/L, <7.0 and <8.0%, respectively; for FT4, they were 12.00–22.00 pmol/L, <5.0 and <7.0%, respectively; and for TSH, they
Shanghai National Clinical Research Center for Metabolic Diseases, Key Laboratory for Endocrine and Metabolic Diseases of the National Health Commission of the PR China, Shanghai National Center for Translational Medicine, Ruijin Hospital, Shanghai Jiao Tong University School of Medicine, Shanghai, China
Guangdong Geriatric Institute, Guangdong Provincial People’s Hospital, Guangdong Academy of Medical Sciences, Guangzhou, China
Search for other papers by Qinglei Yin in
Google Scholar
PubMed
Shanghai National Clinical Research Center for Metabolic Diseases, Key Laboratory for Endocrine and Metabolic Diseases of the National Health Commission of the PR China, Shanghai National Center for Translational Medicine, Ruijin Hospital, Shanghai Jiao Tong University School of Medicine, Shanghai, China
Search for other papers by Zhou Jin in
Google Scholar
PubMed
Shanghai National Clinical Research Center for Metabolic Diseases, Key Laboratory for Endocrine and Metabolic Diseases of the National Health Commission of the PR China, Shanghai National Center for Translational Medicine, Ruijin Hospital, Shanghai Jiao Tong University School of Medicine, Shanghai, China
Search for other papers by Yulin Zhou in
Google Scholar
PubMed
Reproductive Medicine Center, Guangdong Provincial People’s Hospital, Guangdong Academy of Medical Sciences, Guangzhou, China
Search for other papers by Dalong Song in
Google Scholar
PubMed
Shanghai National Clinical Research Center for Metabolic Diseases, Key Laboratory for Endocrine and Metabolic Diseases of the National Health Commission of the PR China, Shanghai National Center for Translational Medicine, Ruijin Hospital, Shanghai Jiao Tong University School of Medicine, Shanghai, China
Search for other papers by Chenyang Fu in
Google Scholar
PubMed
Division of Endocrinology, Department of Internal Medicine, The First Affiliated Hospital of Zhengzhou University, Zhengzhou, Henan, China
Search for other papers by FengJiao Huang in
Google Scholar
PubMed
Shanghai National Clinical Research Center for Metabolic Diseases, Key Laboratory for Endocrine and Metabolic Diseases of the National Health Commission of the PR China, Shanghai National Center for Translational Medicine, Ruijin Hospital, Shanghai Jiao Tong University School of Medicine, Shanghai, China
Search for other papers by Shu Wang in
Google Scholar
PubMed
–4 months and reached normal free triiodothyronine (FT 3 ) and free thyroxine (FT 4 ) levels. GD patients in remission had been treated with MMI for at least 10 months and maintained FT 3 , FT 4 , TSH and TRAb levels in normal range for at least 3 months
Search for other papers by Fan Zhang in
Google Scholar
PubMed
Search for other papers by Jian Chen in
Google Scholar
PubMed
Search for other papers by Xinyue Lin in
Google Scholar
PubMed
Search for other papers by Shiqiao Peng in
Google Scholar
PubMed
Search for other papers by Xiaohui Yu in
Google Scholar
PubMed
Search for other papers by Zhongyan Shan in
Google Scholar
PubMed
Search for other papers by Weiping Teng in
Google Scholar
PubMed
detect total thyroxine (TT4) and thyroid-stimulating hormone (TSH) levels. Six to eight pups from each group on PND7 were decapitated on ice and the cerebral cortex was removed for Western blotting. The remaining pups were raised to PND39 and subjected to
Department of Oncology, Comprehensive Cancer Centre, Helsinki University Hospital, Helsinki, Finland
Search for other papers by Hanna Karhapää in
Google Scholar
PubMed
Department of Oncology, Comprehensive Cancer Centre, Helsinki University Hospital, Helsinki, Finland
Search for other papers by Siru Mäkelä in
Google Scholar
PubMed
Department of Radiology, HUS Medical Imaging Centre, University of Helsinki and Helsinki University Hospital, Helsinki, Finland
Search for other papers by Hanna Laurén in
Google Scholar
PubMed
Department of Radiology, HUS Medical Imaging Centre, University of Helsinki and Helsinki University Hospital, Helsinki, Finland
Search for other papers by Marjut Jaakkola in
Google Scholar
PubMed
Endocrinology, Abdominal Centre, University of Helsinki and HUS, Helsinki, Finland
Search for other papers by Camilla Schalin-Jäntti in
Google Scholar
PubMed
Department of Oncology, Comprehensive Cancer Centre, Helsinki University Hospital, Helsinki, Finland
Search for other papers by Micaela Hernberg in
Google Scholar
PubMed
thyroid-stimulating hormone (TSH), free thyroxine (fT4), free triiodothyronine (fT3), and thyroid antibodies: TSH receptor antibodies (TSHRAb), thyroid peroxidase antibodies (TPOAb), and antithyroglobulin antibodies. Before treatment initiation, we also
Section of Pacing and Electrophysiology, Division of Cardiology, The First Affiliated Hospital of Nanjing Medical University, Nanjing, Jiangsu, China
Search for other papers by Ling Sun in
Google Scholar
PubMed
Search for other papers by Wenwu Zhu in
Google Scholar
PubMed
Search for other papers by Yuan Ji in
Google Scholar
PubMed
Search for other papers by Ailin Zou in
Google Scholar
PubMed
Dalian Medical University, Dalian, Liaoning, China
Search for other papers by Lipeng Mao in
Google Scholar
PubMed
Dalian Medical University, Dalian, Liaoning, China
Search for other papers by Boyu Chi in
Google Scholar
PubMed
Search for other papers by Jianguang Jiang in
Google Scholar
PubMed
Search for other papers by Xuejun Zhou in
Google Scholar
PubMed
Search for other papers by Qingjie Wang in
Google Scholar
PubMed
Search for other papers by Fengxiang Zhang in
Google Scholar
PubMed
levels of thyroid-stimulating hormone (TSH) and thyroxine (T4), is a common thyroid dysfunction ( 4 , 5 ). Free T3 (FT3) plays various roles in the cardiovascular system ( 6 , 7 ). Many publications have reported that a low FT3 is associated with a poor
Search for other papers by Qingrong Pan in
Google Scholar
PubMed
Search for other papers by Shuxin Gao in
Google Scholar
PubMed
Search for other papers by Xia Gao in
Google Scholar
PubMed
Search for other papers by Ning Yang in
Google Scholar
PubMed
Search for other papers by Zhi Yao in
Google Scholar
PubMed
Search for other papers by Yanjin Hu in
Google Scholar
PubMed
Search for other papers by Li Miao in
Google Scholar
PubMed
Search for other papers by Zhe Chen in
Google Scholar
PubMed
Search for other papers by Guang Wang in
Google Scholar
PubMed
to July 2020. Hypothyroidism was defined as an elevated thyrotropin (TSH) level concomitant with a decreased thyroid hormone. Subclinical hypothyroidism was defined as an increased TSH level when concentrations of thyroid hormones were within their