Azrieli Faculty of Medicine, Bar-Ilan University, Safed, Israel
Search for other papers by Amir Bashkin in
Google Scholar
PubMed
Search for other papers by Eliran Yaakobi in
Google Scholar
PubMed
Azrieli Faculty of Medicine, Bar-Ilan University, Safed, Israel
Search for other papers by Marina Nodelman in
Google Scholar
PubMed
Department of Otolaryngology Head and Neck Surgery, Galilee Medical Center, Nahariya, Israel
Search for other papers by Ohad Ronen in
Google Scholar
PubMed
hospitalization is usually the result of stress due to the acute illness that led to hospitalization or the effect of drug therapy. In rare cases, thyroid dysfunction may result from thyroid disease and without treatment may adversely affect hospitalization
Search for other papers by V Kenan Çelik in
Google Scholar
PubMed
Search for other papers by Sercan Kapancık in
Google Scholar
PubMed
Search for other papers by Turgut Kaçan in
Google Scholar
PubMed
Search for other papers by Selen Baloglu Kaçan in
Google Scholar
PubMed
Search for other papers by Serkan Kapancık in
Google Scholar
PubMed
Search for other papers by Hasan Kılıçgün in
Google Scholar
PubMed
depressive disorder (MDD) and stress ( 9 ). Intracellular and extracellular concentrations of polyamines are important in all these biological orientations. The amounts of polyamines are tightly regulated by the ornithine decarboxylase (ODC), a rate
Search for other papers by Stephen Ball in
Google Scholar
PubMed
Search for other papers by Julian Barth in
Google Scholar
PubMed
Search for other papers by Miles Levy in
Google Scholar
PubMed
Search for other papers by the Society for Endocrinology Clinical Committee in
Google Scholar
PubMed
of development of hyponatraemia The intrinsic ability of the central nervous system to adapt to changing osmolar stress The range and degree of co-morbidities Severe symptoms are unlikely with serum sodium >130 mmol/L and alternative causes of
Search for other papers by Ermina Bach in
Google Scholar
PubMed
Search for other papers by Niels Møller in
Google Scholar
PubMed
Search for other papers by Jens Otto L Jørgensen in
Google Scholar
PubMed
Search for other papers by Mads Buhl in
Google Scholar
PubMed
Search for other papers by Holger Jon Møller in
Google Scholar
PubMed
are indirect, since LPS acts through a number of cytokines and also activates the hypothalamo-pituitary axis (HPA) and stimulates the release of stress hormones (e.g. cortisol) into the blood ( 21 , 22 , 23 ). Since CD163 is strongly expressed in
Department of Clinical Medicine, Aalborg University, Aalborg, Denmark
Search for other papers by Stine Linding Andersen in
Google Scholar
PubMed
Department of Geriatrics, Aalborg University Hospital, Aalborg, Denmark
Search for other papers by Stig Andersen in
Google Scholar
PubMed
Rats s.c. infusion (T4) Postnatal months 1–3 Altered morphology of hippocampal neurons and impaired ability to cope with stress Zhang et al. (22) 2010 Rats s.c. infusion (T4) Postnatal months 1–3 Aberrant organization of
Department of Endocrinology, The Affiliated Jiangning Hospital of Nanjing Medical University, Nanjing, Jiangsu, China
Search for other papers by Chenmin Wei in
Google Scholar
PubMed
Search for other papers by Zichen Zhang in
Google Scholar
PubMed
Search for other papers by Qi Fu in
Google Scholar
PubMed
Search for other papers by Yunqiang He in
Google Scholar
PubMed
Search for other papers by Tao Yang in
Google Scholar
PubMed
Search for other papers by Min Sun in
Google Scholar
PubMed
Introduction Various factors may be responsible for β cell dysfunction, including reduced β cell mass (due to de- or transdifferentiation into other cell type) and β cell exhaustion (related to endoplasmic reticulum (ER) and oxidative stress
Search for other papers by Shih-Rong Lin in
Google Scholar
PubMed
Search for other papers by Shih-Fen Chen in
Google Scholar
PubMed
Search for other papers by Yu-Cih Yang in
Google Scholar
PubMed
Search for other papers by Chung-Y Hsu in
Google Scholar
PubMed
Department of Psychiatry, Hualien Tzu Chi Hospital, Buddhist Tzu Chi Medical Foundation, Hualien, Taiwan
School of Medicine, Tzu Chi University, Hualien, Taiwan
Search for other papers by Yu-Chih Shen in
Google Scholar
PubMed
therapy, a significant risk association has been observed. There are possible explanations that may account for the mechanisms underlying this observation. First, hyperthyroidism has been linked to increased oxidative stress and a deregulated
Department of Endocrinology and Diabetes, Pakistan Kidney and Liver Institute and Research Centre, Knowledge City, Lahore, Pakistan
Search for other papers by Zeeshan Javed in
Google Scholar
PubMed
Division of Bone Diseases, Geneva University Hospitals and Faculty of Medicine, University of Geneva, Geneva, Switzerland
Search for other papers by Maria Papageorgiou in
Google Scholar
PubMed
Search for other papers by Leigh A Madden in
Google Scholar
PubMed
Search for other papers by Alan S Rigby in
Google Scholar
PubMed
Search for other papers by Eric S Kilpatrick in
Google Scholar
PubMed
Search for other papers by Stephen L Atkin in
Google Scholar
PubMed
Search for other papers by Thozhukat Sathyapalan in
Google Scholar
PubMed
, reactive oxygen species (ROS), lipopolysaccharides, thrombin and low shear stress. In turn, by facilitating the interactions of endothelial cells with immune cells, EMPs are involved in endothelial cell modifications, inflammation, coagulation and
Search for other papers by Thiago U Pantaleão in
Google Scholar
PubMed
NUMPEX, Pólo de Xerém, Universidade Federal do Rio de Janeiro, Rio de Janeiro, Brazil
Search for other papers by Andrea C F Ferreira in
Google Scholar
PubMed
Search for other papers by Maria C S Santos in
Google Scholar
PubMed
Search for other papers by Álvaro S P Figueiredo in
Google Scholar
PubMed
Search for other papers by Ruy A N Louzada in
Google Scholar
PubMed
Search for other papers by Doris Rosenthal in
Google Scholar
PubMed
Search for other papers by Denise P Carvalho in
Google Scholar
PubMed
Search for other papers by Vânia M Corrêa da Costa in
Google Scholar
PubMed
activity. Increased oxidative stress was previously associated with the inhibition of D1 ( 26 ), but herein we did not detect any association between increased H 2 O 2 generation and D1 activity. In fact, we have shown that at the highest dose (250 µg/100
Fujian Maternal-Fetal Clinical Medicine Research Center, Fuzhou, China
Fujian Key Laboratory of Prenatal Diagnosis and Birth Defect, Fuzhou, China
Search for other papers by Xiuhua Liao in
Google Scholar
PubMed
Search for other papers by Suqin Zhu in
Google Scholar
PubMed
Search for other papers by Shumin Qiu in
Google Scholar
PubMed
Search for other papers by Hua Cao in
Google Scholar
PubMed
Search for other papers by Wenwen Jiang in
Google Scholar
PubMed
Search for other papers by Huiling Xu in
Google Scholar
PubMed
Search for other papers by Yan Sun in
Google Scholar
PubMed
Fujian Maternal-Fetal Clinical Medicine Research Center, Fuzhou, China
Fujian Key Laboratory of Prenatal Diagnosis and Birth Defect, Fuzhou, China
Search for other papers by Beihong Zheng in
Google Scholar
PubMed
mitochondrial morphology, interorganelle Ca 2+ transport ( 12 , 13 ), lipid transportation, and participates in ER stress ( 14 ) and mitophagy. It is a key mediator of mitochondrial fusion, and its expression is decreased in PCOS patients ( 15 ). Mfn2 has been