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Adriano N Cury, Verônica T Meira, Osmar Monte, Marília Marone, Nilza M Scalissi, Cristiane Kochi, Luís E P Calliari and Carlos A Longui

Pediatric Endocrinology and Metabolism 2001 14 229 – 243 . ( doi:10.1515/JPEM.2001.14.3.229 ). 2 Kaguelidou F Carel JC Leger J . Graves' disease in childhood: advances in management with antithyroid drug therapy

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E Vignali, F Cetani, S Chiavistelli, A Meola, F Saponaro, R Centoni, L Cianferotti and C Marcocci

levels (e.g. Paget's disease) should be excluded. Finally, the use of medications which might affect PTH levels or calcium metabolism (estrogens, thiazide diuretics, lithium, bisphosphonates, denosumab and anticonvulsants) should also be ruled out (4

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David P Sonne, Asger Lund, Jens Faber, Jens J Holst, Tina Vilsbøll and Filip K Knop

labelled as detergents necessary for lipid digestion and absorption, but are increasingly recognised as metabolic integrators, capable of regulating glucose homeostasis, lipid metabolism and energy expenditure through nuclear receptors and the G protein

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Meena Asmar, Ali Asmar, Lene Simonsen, Flemming Dela, Jens Juul Holst and Jens Bülow

Introduction The regulation of subcutaneous adipose tissue blood flow (ATBF) is crucial in lipid homeostasis ( 1 , 2 ). It has long been recognized that the ATBF is tightly coupled to adipose tissue metabolism ( 3 , 4 , 5 , 6 ). In

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Carla Scaroni, Nora M Albiger, Serena Palmieri, Davide Iacuaniello, Chiara Graziadio, Luca Damiani, Marialuisa Zilio, Antonio Stigliano, Annamaria Colao, Rosario Pivonello and the Altogether to Beat Cushing’s Syndrome (ABC) study group

assessing cortisol and cortisone levels ( 9 ). The concomitant use of commonly prescribed therapies may alter dexamethasone metabolism, interfering with its use in a suppression test. Some types of medication can interfere with the CYP3A4 enzyme system

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Ursula M M Costa, Carla R P Oliveira, Roberto Salvatori, José A S Barreto-Filho, Viviane C Campos, Francielle T Oliveira, Ivina E S Rocha, Joselina L M Oliveira, Wersley A Silva and Manuel H Aguiar-Oliveira

. References 1 Aguiar-Oliveira MH & Salvatori R. Lifetime growth hormone (GH) deficiency: impact on growth, metabolism, body composition, and survival capacity Chapter ID 160. In Handbook of Growth and Growth Monitoring in Health and Disease . Ed VR Preedy

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J Chycki, A Zajac, M Michalczyk, A Maszczyk and J Langfort

metabolism, the white adipose tissue (WAT) also referred to as subcutaneous or visceral fat (intraabdominal: mesenteric, perirenal and epididymal depots) serves as a primary energy store, and lipolysis in WAT is a crucial process for whole-body energy

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Chao Xu, Xiang-Fei Li, Hong-Yan Tian, Hua-Juan Shi, Ding-Dong Zhang, Kenneth Prudence Abasubong and Wen-Bin Liu

( 2 ). However, to date, the energy metabolism of fish after a GTT has barely been investigated. It is now widely acknowledged that, the energy metabolism of fish is monitored by the intracellular energy sensors ( 1 , 10 ). Indeed, several studies

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Marcus Quinkler, Bertil Ekman, Claudio Marelli, Sharif Uddin, Pierre Zelissen, Robert D Murray and on behalf of the EU-AIR Investigators

absorbed rapidly after oral intake, reaching peak plasma levels within the first hour after ingestion ( 2 ). The mean plasma half-life is 2–4 h ( 3 , 4 ); metabolism occurs predominantly in the liver ( 5 ) but also via the actions of renal 11β

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Yael Sofer, Nava Nevo, Michal Vechoropoulos, Gabi Shefer, Etty Osher, Nathan Landis, Karen Tordjman, Geoffrey L Hammond and Naftali Stern

the SHBG gene per se and also by an array of loci in genes involved in biologic networks such as liver function, lipid metabolism, glucose metabolism, androgen and estrogen receptor function and epigenetic effects ( 29 ). Recent studies have also