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Patricia Arroyo Tardio
Patricia Arroyo Tardio
University Hospital Basel, Basel, Switzerland
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Gabriela Baldini
Gabriela Baldini
University Clinic of Medicine, Cantonal Hospital Baselland, Liestal, Switzerland
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Eleonora Seelig
Eleonora Seelig
University Hospital Basel, Basel, Switzerland
University Clinic of Medicine, Cantonal Hospital Baselland, Liestal, Switzerland
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University Clinic of Medicine, Cantonal Hospital Baselland, Liestal, Switzerland
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Introduction The hypothalamopituitary–adrenal (HPA) axis tightly regulates cortisol secretion ( 1 ). Cortisol is secreted in a circadian rhythm with a brisk increase upon awakening and a nadir around midnight ( 1 ). Food is an external factor
Giovanni Fanni
Giovanni Fanni
Department of Medical Sciences, Clinical Diabetes and Metabolism, Uppsala University, Uppsala, Sweden
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Petros Katsogiannos
Petros Katsogiannos
Department of Medical Sciences, Clinical Diabetes and Metabolism, Uppsala University, Uppsala, Sweden
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Bipasha Nandi Jui
Bipasha Nandi Jui
Department of Medical Sciences, Clinical Diabetes and Metabolism, Uppsala University, Uppsala, Sweden
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Magnus Sundbom
Magnus Sundbom
Department of Surgical Sciences, Uppsala University, Uppsala, Sweden
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Susanne Hetty
Susanne Hetty
Department of Medical Sciences, Clinical Diabetes and Metabolism, Uppsala University, Uppsala, Sweden
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Maria J Pereira
Maria J Pereira
Department of Medical Sciences, Clinical Diabetes and Metabolism, Uppsala University, Uppsala, Sweden
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Jan W Eriksson
Jan W Eriksson
Department of Medical Sciences, Clinical Diabetes and Metabolism, Uppsala University, Uppsala, Sweden
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and pancreatic islets but also of others produced by the pituitary and adrenal glands. Therefore, in this study, we assess growth hormone (GH), ACTH, and cortisol levels, which are largely unexplored in this context. Herein, we report exploratory post
Jan W Eriksson
Jan W Eriksson
Department of Medical Sciences, Uppsala University, Uppsala, Sweden
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Reem A Emad
Reem A Emad
Department of Pharmacy, Uppsala University, Uppsala, Sweden
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Martin H Lundqvist
Martin H Lundqvist
Department of Medical Sciences, Uppsala University, Uppsala, Sweden
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Niclas Abrahamsson
Niclas Abrahamsson
Department of Medical Sciences, Uppsala University, Uppsala, Sweden
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Maria C Kjellsson
Maria C Kjellsson
Department of Pharmacy, Uppsala University, Uppsala, Sweden
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quantitative importance of other factors, such as inhibition by insulin and stimulation by amino acids, is disputed ( 8 , 9 ). Hypoglycemia also triggers the release of other counter-regulatory hormones such as catecholamines, cortisol, and growth hormone
Felix Reschke
Felix Reschke
Auf Der Bult Children’s Hospital, Centre for Paediatric Endocrinology, Diabetology, and Clinical Research, Hannover, Germany
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Torben Biester
Torben Biester
Auf Der Bult Children’s Hospital, Centre for Paediatric Endocrinology, Diabetology, and Clinical Research, Hannover, Germany
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Thekla von dem Berge
Thekla von dem Berge
Auf Der Bult Children’s Hospital, Centre for Paediatric Endocrinology, Diabetology, and Clinical Research, Hannover, Germany
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Dagmar Jamiolkowski
Dagmar Jamiolkowski
Auf Der Bult Children’s Hospital, Department of Paediatric Dermatology, Hannover, Germany
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Laura Hasse
Laura Hasse
Auf Der Bult Children’s Hospital, Department of Paediatric Dermatology, Hannover, Germany
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Francesca Dassie
Francesca Dassie
Padua University Hospital, Clinica Medica 3, Department of Medicine (DIMED), Padova, Veneto, Italy
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Pietro Maffei
Pietro Maffei
Padua University Hospital, Clinica Medica 3, Department of Medicine (DIMED), Padova, Veneto, Italy
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Katharina Klee
Katharina Klee
Auf Der Bult Children’s Hospital, Centre for Paediatric Endocrinology, Diabetology, and Clinical Research, Hannover, Germany
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Olga Kordonouri
Olga Kordonouri
Auf Der Bult Children’s Hospital, Centre for Paediatric Endocrinology, Diabetology, and Clinical Research, Hannover, Germany
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Hagen Ott
Hagen Ott
Auf Der Bult Children’s Hospital, Department of Paediatric Dermatology, Hannover, Germany
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Thomas Danne
Thomas Danne
Auf Der Bult Children’s Hospital, Centre for Paediatric Endocrinology, Diabetology, and Clinical Research, Hannover, Germany
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may occur. Eczematous and hyperkeratotic dermatitis and maculopapular eruptions have also been reported. Cushing's syndrome CS is characterised by a cluster of clinical features caused by chronic glucocorticoid excess. Cortisol excess can be
Mohamed Asrih
Mohamed Asrih
Service of Endocrinology, Diabetes, Nutrition and Patient Therapeutic Education, Geneva University Hospitals, Geneva, Switzerland
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Flore Sinturel
Flore Sinturel
Thoracic and Endocrine Surgery Division, Department of Surgery, Geneva University Hospitals (HUG), Geneva, Switzerland
Department of Cell Physiology and Metabolism, Faculty of Medicine, University of Geneva, Geneva, Switzerland
Diabetes Center, Faculty of Medicine, University of Geneva, Geneva, Switzerland
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Department of Cell Physiology and Metabolism, Faculty of Medicine, University of Geneva, Geneva, Switzerland
Diabetes Center, Faculty of Medicine, University of Geneva, Geneva, Switzerland
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Richard Dubos
Richard Dubos
Department and Division of Primary Care Medicine, Geneva University Hospitals (HUG), Geneva, Switzerland
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Idris Guessous
Idris Guessous
Department and Division of Primary Care Medicine, Geneva University Hospitals (HUG), Geneva, Switzerland
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Zoltan Pataky
Zoltan Pataky
Service of Endocrinology, Diabetes, Nutrition and Patient Therapeutic Education, Geneva University Hospitals, Geneva, Switzerland
Diabetes Center, Faculty of Medicine, University of Geneva, Geneva, Switzerland
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Diabetes Center, Faculty of Medicine, University of Geneva, Geneva, Switzerland
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Charna Dibner
Charna Dibner
Thoracic and Endocrine Surgery Division, Department of Surgery, Geneva University Hospitals (HUG), Geneva, Switzerland
Department of Cell Physiology and Metabolism, Faculty of Medicine, University of Geneva, Geneva, Switzerland
Diabetes Center, Faculty of Medicine, University of Geneva, Geneva, Switzerland
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Department of Cell Physiology and Metabolism, Faculty of Medicine, University of Geneva, Geneva, Switzerland
Diabetes Center, Faculty of Medicine, University of Geneva, Geneva, Switzerland
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François R Jornayvaz
François R Jornayvaz
Service of Endocrinology, Diabetes, Nutrition and Patient Therapeutic Education, Geneva University Hospitals, Geneva, Switzerland
Department of Cell Physiology and Metabolism, Faculty of Medicine, University of Geneva, Geneva, Switzerland
Diabetes Center, Faculty of Medicine, University of Geneva, Geneva, Switzerland
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Department of Cell Physiology and Metabolism, Faculty of Medicine, University of Geneva, Geneva, Switzerland
Diabetes Center, Faculty of Medicine, University of Geneva, Geneva, Switzerland
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Karim Gariani
Karim Gariani
Service of Endocrinology, Diabetes, Nutrition and Patient Therapeutic Education, Geneva University Hospitals, Geneva, Switzerland
Diabetes Center, Faculty of Medicine, University of Geneva, Geneva, Switzerland
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Diabetes Center, Faculty of Medicine, University of Geneva, Geneva, Switzerland
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.20) 1.12 (0.35) 1.01 (0.27) Cortisol (nmol/L) 349 (98) 402 (132) 382 (65) 394 (155) 378 (117) 304 (104) 406 (148) 366 (71) Leukocytes (g/L) 5.16 (1.41) 6.96 (1.95) 7.09 (1.44) 6.38 (0.86) 5.30 (1.11) 6.91 (1
Cheryl M Isherwood
Cheryl M Isherwood
Section of Chronobiology, Faculty of Health and Medical Sciences, University of Surrey, Guildford, United Kingdom
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M Denise Robertson
M Denise Robertson
Section of Metabolic Medicine, Food and Macronutrients, Faculty of Health and Medical Sciences, University of Surrey, Guildford, United Kingdom
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Debra J Skene
Debra J Skene
Section of Chronobiology, Faculty of Health and Medical Sciences, University of Surrey, Guildford, United Kingdom
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Jonathan D Johnston
Jonathan D Johnston
Section of Chronobiology, Faculty of Health and Medical Sciences, University of Surrey, Guildford, United Kingdom
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PJ Davies SK Middleton B Raynaud FI & Skene DJ . Effect of acute total sleep deprivation on plasma melatonin, cortisol and metabolite rhythms in females . European Journal of Neuroscience 2020 51 366 – 378 . ( https://doi.org/10.1111/ejn
Svjatoslavs Kistkins
Svjatoslavs Kistkins
Pauls Stradiņš Clinical University Hospital, Riga, Latvia
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Othmar Moser
Othmar Moser
Division of Exercise Physiology and Metabolism, Institute of Sport Science, University of Bayreuth, Bayreuth, Germany
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Vitālijs Ankudovičs
Vitālijs Ankudovičs
Pauls Stradiņš Clinical University Hospital, Riga, Latvia
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Dmitrijs Blizņuks
Dmitrijs Blizņuks
Institute of Smart Computing Technologies, Riga Technical University, Riga, Latvia
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Timurs Mihailovs
Timurs Mihailovs
Institute of Smart Computing Technologies, Riga Technical University, Riga, Latvia
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Sergejs Lobanovs
Sergejs Lobanovs
Pauls Stradiņš Clinical University Hospital, Riga, Latvia
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Harald Sourij
Harald Sourij
Trials Unit for Interdisciplinary Metabolic Medicine, Division of Endocrinology and Diabetolgoy, Medical University of Graz, Graz, Austria
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Andreas F H Pfeiffer
Andreas F H Pfeiffer
Department of Endocrinology and Metabolic Medicine, Campus Benjamin Franklin, Charité University Medicine, Hindenburgdamm, Berlin, Germany
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Valdis Pīrāgs
Valdis Pīrāgs
Pauls Stradiņš Clinical University Hospital, Riga, Latvia
Faculty of Medicine, University of Latvia, Riga, Latvia
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Faculty of Medicine, University of Latvia, Riga, Latvia
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possibility is that the liver becomes tolerant to the hyperglycaemic effects of glucagon in T2D upon the use of long-acting glucagon agonists. Prolonged fasting also leads to a switch from glucagon to cortisol to maintain hepatic glucose production. In the
Jonathan Hazlehurst
Jonathan Hazlehurst
Department of Diabetes and Endocrinology, University Hospitals Birmingham NHS Foundation Trust, Birmingham, UK
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Bernard Khoo
Bernard Khoo
Endocrinology, Division of Medicine, University College London, London, UK
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Carolina Brito Lobato
Carolina Brito Lobato
Department of Biomedical Sciences, Faculty of Health and Medical Sciences, University of Copenhagen, Copenhagen, Denmark
Department of Medicine, Copenhagen University Hospital – Amager and Hvidovre, Hvidovre, Denmark
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Department of Medicine, Copenhagen University Hospital – Amager and Hvidovre, Hvidovre, Denmark
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Ibiyemi Ilesanmi
Ibiyemi Ilesanmi
Section of Endocrinology and Investigative Medicine, Department of Metabolism, Digestion and Reproduction, Faculty of Medicine, Imperial College London, London, UK
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Sally Abbott
Sally Abbott
Department of Dietetics, University Hospitals Coventry and Warwickshire NHS Trust, Coventry, UK
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Tin Chan
Tin Chan
Faculty of Medicine, Chinese University of Hong Kong, Hong Kong
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Sanesh Pillai
Sanesh Pillai
Centre for Endocrinology, Diabetes and Metabolism, Birmingham Health Partners, Birmingham, UK
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Kate Maslin
Kate Maslin
School of Nursing and Midwifery, University of Plymouth, Plymouth, UK
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Sanjay Purkayastha
Sanjay Purkayastha
Brunel University, London, UK
Imperial College Healthcare NHS Trust, St Mary’s Hospital, London, UK
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Imperial College Healthcare NHS Trust, St Mary’s Hospital, London, UK
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Barbara McGowan
Barbara McGowan
Endocrinology, Guys’ and St Thomas’s NHS Foundation Trust, London, UK
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Rob Andrews
Rob Andrews
University of Exeter Medical School, Exeter, UK
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Eveleigh Nicholson
Eveleigh Nicholson
Portsmouth Hospitals University NHS Trust, Portsmouth, UK
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Katherine McCullough
Katherine McCullough
Royal Surrey County Hospital, Guildford, UK
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Lorraine Albon
Lorraine Albon
University Hospitals Sussex NHS Foundation Trust, Worthing, UK
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Rachel Batterham
Rachel Batterham
Endocrinology, Division of Medicine, University College London, London, UK
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Georgios K Dimitriadis
Georgios K Dimitriadis
King's College Hospital NHS Foundation Trust, London, UK
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Shareen Forbes
Shareen Forbes
BHF Centre for Cardiovascular Science, Queen’s Medical Research Institute, University of Edinburgh, Edinburgh, UK
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Gavin Bewick
Gavin Bewick
School of Life Course Sciences, Faculty of Life Sciences and Medicine, King's College London, London, UK
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Tricia M-M Tan
Tricia M-M Tan
Section of Endocrinology and Investigative Medicine, Department of Metabolism, Digestion and Reproduction, Faculty of Medicine, Imperial College London, London, UK
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histories of PBH), exenatide was not shown to influence counter-regulatory hormone secretion (glucagon, catecholamines, growth hormone, cortisol) during a hypoglycaemic hyperinsulinemic clamp, suggesting that if there is any effect of GLP-1 analogues, it is
