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Satoshi Higuchi, Hideki Ota, Takuya Ueda, Yuta Tezuka, Kei Omata, Yoshikiyo Ono, Ryo Morimoto, Masataka Kudo, Fumitoshi Satoh, and Kei Takase

motion assessment for detecting catecholamine-induced cardiac injury. Pathologically, catecholamines have been shown to cause myocardial injury, such as contraction band necrosis, via alpha-receptor-mediated vasoconstriction and oxygen-derived free

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Madalena von Hafe, João Sergio Neves, Catarina Vale, Marta Borges-Canha, and Adelino Leite-Moreira

contractile apparatus of the cardiac myocyte has two subtypes of myosin heavy chains (MHCs): α-MHC and β-MHC – fast and slow myosin, respectively. T3 can upregulate α-MHC and downregulate β-MHC ( 10 ). Therefore, the myocardial hypothyroid state induces a so

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M L M Barreto-Chaves, N Senger, M R Fevereiro, A C Parletta, and A P C Takano

/mice . BioMed Research International 2017 2017 6071031 . ( ) 54 Yao J Eghbali M . Decreased collagen gene expression and absence of fibrosis in thyroid hormone-induced myocardial hypertrophy. Response of cardiac

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Hongyan Wang, Bin Wu, Zichuan Yao, Xianqing Zhu, Yunzhong Jiang, and Song Bai

circulatory volume as a result of lower body weight than that of males. Moreover, these patients had peripheral vasoconstriction induced by catecholamines secreted by a pheochromocytoma, which resulted in further reduction in effective circulatory volume and

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Lang Qin, Xiaoming Zhu, Xiaoxia Liu, Meifang Zeng, Ran Tao, Yan Zhuang, Yiting Zhou, Zhaoyun Zhang, Yehong Yang, Yiming Li, Yongfei Wang, and Hongying Ye

Introduction Hypertension is a common chronic condition in patients with Cushing’s disease (CD) and may be the first sign ( 1 ). The pathogenesis of glucocorticoid-induced hypertension is not fully understood. It is thought to be related in