Lawrence D Hayes
Lawrence D Hayes
Active Ageing Research Group, Department of Medical and Sport Sciences, University of Cumbria, Lancaster, UK
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Peter Herbert
Peter Herbert
School of Sport, Health and Outdoor Education, Trinity Saint David, University of Wales, Carmarthen, UK
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Nicholas F Sculthorpe
Nicholas F Sculthorpe
Institute of Clinical Exercise and Health Science, University of the West of Scotland, Hamilton, UK
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Fergal M Grace
Fergal M Grace
Faculty of Health, Federation University, Victoria, Australia
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exercise session as previously described ( 21 ). Serum concentrations of TT, SHBG and cortisol were measured by electrochemiluminescent immunoassay on the E601 module of the Roche Cobas 6000 (Burgess Hill, West Sussex, UK). Inter-assay coefficients of
L M Mongioì
L M Mongioì
Department of Clinical and Experimental Medicine, University of Catania, Catania, Italy
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R A Condorelli
R A Condorelli
Department of Clinical and Experimental Medicine, University of Catania, Catania, Italy
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S La Vignera
S La Vignera
Department of Clinical and Experimental Medicine, University of Catania, Catania, Italy
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A E Calogero
A E Calogero
Department of Clinical and Experimental Medicine, University of Catania, Catania, Italy
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hydrocortisone dose of 15–25 mg ( 8 ). The conventional treatment involves a double or triple administration of glucocorticoid, thus causing a supraphysiological cortisol exposition. Recently, a new once-daily dual-release hydrocortisone formulation has been
M S Elston
M S Elston
Department of Endocrinology, Waikato Hospital, Hamilton, New Zealand
Waikato Clinical Campus, University of Auckland, Hamilton, New Zealand
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Waikato Clinical Campus, University of Auckland, Hamilton, New Zealand
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V B Crawford
V B Crawford
Department of Endocrinology, Waikato Hospital, Hamilton, New Zealand
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M Swarbrick
M Swarbrick
Department of Radiology, Waikato Hospital, Hamilton, New Zealand
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M S Dray
M S Dray
Department of Pathology, Waikato Hospital, Hamilton, New Zealand
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M Head
M Head
Department of Oncology, Tauranga Hospital, Tauranga, New Zealand
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J V Conaglen
J V Conaglen
Waikato Clinical Campus, University of Auckland, Hamilton, New Zealand
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showed a hypokalaemic metabolic alkalosis ( Table 1 ). Hypercortisolaemia was confirmed with morning cortisol >1655 nmol/L, absence of diurnal cortisol rhythm and markedly increased 24-h urinary free cortisol excretion (24-h UFC). Table 1 Pertinent
Jülide Durmuşoğlu
Jülide Durmuşoğlu
Division of Endocrinology, Department of Medicine, Radboud University Nijmegen Medical Centre, Nijmegen, the Netherlands
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Henri J L M Timmers
Henri J L M Timmers
Division of Endocrinology, Department of Medicine, Radboud University Nijmegen Medical Centre, Nijmegen, the Netherlands
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Pepijn van Houten
Pepijn van Houten
Division of Endocrinology, Department of Medicine, Radboud University Nijmegen Medical Centre, Nijmegen, the Netherlands
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Johan F Langenhuijsen
Johan F Langenhuijsen
Department of Urology, Radboud University Nijmegen Medical Centre, Nijmegen, the Netherlands
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Ad R M M Hermus
Ad R M M Hermus
Division of Endocrinology, Department of Medicine, Radboud University Nijmegen Medical Centre, Nijmegen, the Netherlands
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Annenienke C van de Ven
Annenienke C van de Ven
Division of Endocrinology, Department of Medicine, Radboud University Nijmegen Medical Centre, Nijmegen, the Netherlands
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rates of ACC after resection are high ( 6 , 8 ). ACC may be classified as functional (hormone-secreting) or nonfunctional. Functional adrenal tumors of the cortex can produce cortisol, androgens, estrogens and aldosterone. A functional ACC can cause
Amit Kumar
Amit Kumar
Department of Medicine, Christian Medical College and Hospital, Ludhiana, Punjab, India
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Maria Ghosh
Maria Ghosh
Department of Biochemistry, Christian Medical College and Hospital, Ludhiana, Punjab, India
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Jubbin Jagan Jacob
Jubbin Jagan Jacob
Department of Endocrinology, Christian Medical College and Hospital, Ludhiana, Punjab, India
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.2%, respectively. For precision testing, the CV% for low to normal and high sodium levels was 1.1 and 0.4 mmol/L, respectively. Quantitative determination of serum cortisol was performed using Elecsys Cortisol II kit on Cobas e411/601 fully automated analyzer
Masafumi Tetsuka
Masafumi Tetsuka
Department of Life and Food Science, Obihiro University of Agriculture and Veterinary Medicine, Obihiro, Hokkaido, Japan
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Misato Tanakadate
Misato Tanakadate
Department of Life and Food Science, Obihiro University of Agriculture and Veterinary Medicine, Obihiro, Hokkaido, Japan
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2) ( 13 ). The former is a reductase that predominantly catalyzes inactive cortisone to active cortisol, while the latter is an oxidase that catalyzes the opposite reaction ( 14 ). The expression of these HSD11Bs appeared to be differentially
T P Parikh
T P Parikh
Program in Reproductive and Adult Endocrinology, National Institute of Child Health and Human Development, National Institutes of Health (NIH), Bethesda, Maryland, USA
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B Stolze
B Stolze
Department of Laboratory Medicine, Clinical Center, NIH, Bethesda, Maryland, USA
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Y Ozarda
Y Ozarda
Department of Medical Biochemistry, Faculty of Medicine, Uludag University, Bursa, Turkey
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J Jonklaas
J Jonklaas
Division of Endocrinology and Metabolism, Department of Medicine, Georgetown University, Washington, District of Columbia, USA
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K Welsh
K Welsh
Department of Laboratory Medicine, Clinical Center, NIH, Bethesda, Maryland, USA
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L Masika
L Masika
Department of Laboratory Medicine and Pathology/National Health Laboratory Service Walter Sisulu University, Mthatha, South Africa
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M Hill
M Hill
Program in Reproductive and Adult Endocrinology, National Institute of Child Health and Human Development, National Institutes of Health (NIH), Bethesda, Maryland, USA
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A DeCherney
A DeCherney
Program in Reproductive and Adult Endocrinology, National Institute of Child Health and Human Development, National Institutes of Health (NIH), Bethesda, Maryland, USA
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S J Soldin
S J Soldin
Department of Laboratory Medicine, Clinical Center, NIH, Bethesda, Maryland, USA
Division of Endocrinology and Metabolism, Department of Medicine, Georgetown University, Washington, District of Columbia, USA
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Division of Endocrinology and Metabolism, Department of Medicine, Georgetown University, Washington, District of Columbia, USA
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Introduction Diurnal variations in the serum concentration of steroid hormones and their metabolism, as assessed by urinary excretion, have long been known for cortisol and testosterone ( 1 , 2 , 3 ). Less information exists for the other up
Jana Ernst
Jana Ernst
Department of Anatomy and Cell Biology, Faculty of Medicine, Martin Luther University Halle-Wittenberg, Grosse Steinstrasse, Halle (Saale), Germany
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Katharina Gert
Katharina Gert
Department of Anatomy and Cell Biology, Faculty of Medicine, Martin Luther University Halle-Wittenberg, Grosse Steinstrasse, Halle (Saale), Germany
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Frank Bernhard Kraus
Frank Bernhard Kraus
Central Laboratory, University Hospital Halle (Saale), Ernst-Grube-Strasse, Halle (Saale), Germany
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Ulrike Elisabeth Rolle-Kampczyk
Ulrike Elisabeth Rolle-Kampczyk
Department of Molecular Systems Biology, Helmholtz Centre for Environmental Research Leipzig, Leipzig, Germany
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Martin Wabitsch
Martin Wabitsch
Division of Pediatric Endocrinology and Diabetes, Department of Pediatrics and Adolescent Medicine, University Medical Center Ulm, Ulm, Germany
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Faramarz Dehghani
Faramarz Dehghani
Department of Anatomy and Cell Biology, Faculty of Medicine, Martin Luther University Halle-Wittenberg, Grosse Steinstrasse, Halle (Saale), Germany
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Kristina Schaedlich
Kristina Schaedlich
Department of Anatomy and Cell Biology, Faculty of Medicine, Martin Luther University Halle-Wittenberg, Grosse Steinstrasse, Halle (Saale), Germany
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concentration of cortisone. Cholesterol and steroid hormones (cortisol, DHEAS, estradiol, progesterone, testosterone) were analyzed in the Central Laboratory of the University Hospital Halle. Cholesterol was measured using a colorimetric assay (CHOL2
Peter Wolf
Peter Wolf
Division of Endocrinology and Metabolism, Department of Internal Medicine III, Medical University of Vienna, Vienna, Austria
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Yvonne Winhofer
Yvonne Winhofer
Division of Endocrinology and Metabolism, Department of Internal Medicine III, Medical University of Vienna, Vienna, Austria
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Martin Krššák
Martin Krššák
Division of Endocrinology and Metabolism, Department of Internal Medicine III, Medical University of Vienna, Vienna, Austria
High Field MR Centre, Department of Biomedical Imaging and Image-guided Therapy, Medical University of Vienna, Vienna, Austria
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High Field MR Centre, Department of Biomedical Imaging and Image-guided Therapy, Medical University of Vienna, Vienna, Austria
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Michael Krebs
Michael Krebs
Division of Endocrinology and Metabolism, Department of Internal Medicine III, Medical University of Vienna, Vienna, Austria
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morphology of the heart in patients suffering from T2DM and hypothyroidism. Cortisol Death from cardiovascular disease, including heart failure, coronary artery disease and cardiac thromboembolism, is the leading cause of increased mortality observed
M Cherenko
M Cherenko
Department of Medicine, Division of Endocrinology, Leiden University Medical Centre, Leiden, Netherlands
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N M Appelman-Dijkstra
N M Appelman-Dijkstra
Department of Medicine, Division of Endocrinology, Leiden University Medical Centre, Leiden, Netherlands
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A L Priego Zurita
A L Priego Zurita
Department of Medicine, Division of Endocrinology, Leiden University Medical Centre, Leiden, Netherlands
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N R Biermasz
N R Biermasz
Department of Medicine, Division of Endocrinology, Leiden University Medical Centre, Leiden, Netherlands
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O M Dekkers
O M Dekkers
Department of Medicine, Division of Endocrinology, Leiden University Medical Centre, Leiden, Netherlands
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F A Klok
F A Klok
Department of Medicine, Division of Thrombosis and Haemostasis, Leiden University Medical Centre, Leiden, Netherlands
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N Reisch
N Reisch
Department of Medicine IV, LMU University Hospital, LMU Munich, Munich, Germany
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A Aulinas
A Aulinas
Department of Endocrinology, Fundacio de Gestio Sanitaria Hospital de la Santa Creu i Sant Pau, IR-SantPau and CIBERER Unit 747 (ISCIII), Barcelona, Spain
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B Biagetti
B Biagetti
Department of Endocrinology, Hospital Universitari Vall d’Hebron, Barcelona, Spain
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S Cannavo
S Cannavo
Endocrine Unit, University Hospital AOU Policlinico G. Martino, Messina, Italy
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L Canu
L Canu
University Hospital Florence Careggi, Florence, Italy
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M Detomas
M Detomas
Department of Internal Medicine, University Hospital Würzburg, Wuerzburg, Germany
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F Devuyst
F Devuyst
Department of Endocrinology, Hôpital Universitaire de Bruxelles, Hôpital Erasme, Brussels, Belgium
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H Falhammar
H Falhammar
Department of Endocrinology, Karolinska University Hospital and Department of Molecular Medicine and Surgery, Karolinska Institutet, Stockholm, Sweden
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R A Feelders
R A Feelders
Department of Internal Medicine, Division of Endocrinology, Erasmus MC, Rotterdam, Netherlands
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F Ferrau
F Ferrau
Endocrine Unit, University Hospital AOU Policlinico G. Martino, Messina, Italy
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F Gatto
F Gatto
IRCCS Ospedale Policlinico San Martino, Genova, Genoa, Italy
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C Grasselli
C Grasselli
Cardiovascular Medicine Unit, AUSL-IRCCS, Reggio Emilia, Italy
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P van Houten
P van Houten
Department of Internal Medicine, Division of Endocrinology, Radboud University Medical Center, Nijmegen, Netherlands
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C Hoybye
C Hoybye
Department of Endocrinology, Karolinska University Hospital and Department of Molecular Medicine and Surgery, Karolinska Institutet, Stockholm, Sweden
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A M Isidori
A M Isidori
Department of Experimental Medicine, Sapienza University of Rome, Rome, Italy
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A Kyrilli
A Kyrilli
Department of Endocrinology, Hôpital Universitaire de Bruxelles, Hôpital Erasme, Brussels, Belgium
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P Loli
P Loli
Division of Endocrinology, San Raffaele Vita-Salute University, IRCCS San Raffaele Hospital Milan, Italy
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D Maiter
D Maiter
Department of Endocrinology, Cliniques universitaires Saint-Luc – UCLouvain, Brussels, Belgium
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E Nowak
E Nowak
Department of Medicine IV, LMU University Hospital, LMU Munich, Munich, Germany
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R Pivonello
R Pivonello
Dipartimento di Medicina Clinica e Chirurgia, Sezione di Endocrinologia, Diabetologia, Andrologia e Nutrizione, Università “Federico II” di Napoli, Naples, Italy
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O Ragnarsson
O Ragnarsson
Sahlgrenska Academy, Wallenberg Centre for Molecular and Translational Medicine, Institute of Medicine (O.R.), University of Gothenburg, Sweden
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R V Steenaard
R V Steenaard
Department of Internal Medicine, Máxima MC, Veldhoven, Netherlands
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N Unger
N Unger
University Hospital Essen, Department of Endocrinology, Diabetes and Metabolism, Essen, Germany
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A van de Ven
A van de Ven
Department of Internal Medicine, Division of Endocrinology, Radboud University Medical Center, Nijmegen, Netherlands
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S M Webb
S M Webb
Department of Endocrinology, Fundacio de Gestio Sanitaria Hospital de la Santa Creu i Sant Pau, IR-SantPau and CIBERER Unit 747 (ISCIII), Barcelona, Spain
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D Yeste
D Yeste
Pediatric Endocrinology Service, Hospital Universitari Vall d’Hebron, Universitat Autònoma de Barcelona, Barcelona, Spain. CIBER Enfermedades Raras, Instituto Carlos III, Madrid, Spain
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S F Ahmed
S F Ahmed
Department of Medicine, Division of Endocrinology, Leiden University Medical Centre, Leiden, Netherlands
University of Glasgow, Office for Rare Conditions, Glasgow, UK
University of Glasgow, Developmental Endocrinology Research Group, Royal Hospital for Children, Glasgow, UK
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University of Glasgow, Office for Rare Conditions, Glasgow, UK
University of Glasgow, Developmental Endocrinology Research Group, Royal Hospital for Children, Glasgow, UK
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A M Pereira
A M Pereira
Department of Endocrinology & Metabolism, Amsterdam University Medical Centre, Amsterdam, Noord-Holland, Netherlands
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has been demonstrated that the primary risk factors associated with VTE include older age (>69 years), reduced mobility, acute severe infections, previous cardiovascular events, higher midnight plasma cortisol levels, and shorter activated partial
