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M Langeveld, C Y Tan, M R Soeters, S Virtue, G K Ambler, L P E Watson, P R Murgatroyd, V K Chatterjee, and A Vidal-Puig

ANOVA for the effect of temperature P < 0.001). Figure 4 (A) Plasma glucose, (B) non-esterified free fatty acid (NEFA), (C) cortisol, (D) thyroid-stimulating hormone (TSH), (4) free thyroxine (FT4) (E) and free tri-iodothyronine (FT3) (F) over

Open access

Aleksandra Krygier, Ewelina Szczepanek-Parulska, Dorota Filipowicz, and Marek Ruchała

diagnostic measurement (DRG Instruments GmbH, Germany). The thyroid gland status was characterized by thyroid-stimulating hormone (TSH), free thyroid hormones (free triiodothyronine – fT3 and free thyroxine – fT4) and autoantibodies (anti-thyroid peroxidase

Open access

Chiara Mele, Maria Teresa Samà, Alessandro Angelo Bisoffi, Marina Caputo, Valentina Bullara, Stefania Mai, Gillian Elisabeth Walker, Flavia Prodam, Paolo Marzullo, Gianluca Aimaretti, and Loredana Pagano

-stimulating hormone (TSH), free thyroxine (fT4) and free triiodothyronine (fT3) using an automated chemiluminescence assay system (ADVIA Centaur Systems TSH3/fT4/fT3 Ultra Ready Pack, Siemens Healthcare Diagnostics). Serum levels of thyroglobulin (Tg) were

Open access

Qingrong Pan, Shuxin Gao, Xia Gao, Ning Yang, Zhi Yao, Yanjin Hu, Li Miao, Zhe Chen, and Guang Wang

normal ranges. The normal values for TSH, free triiodothyronine (FT3) and free thyroxine (FT4) were 0.55–4.78 μIU/mL, 2.3–4.2 pg/mL and 8.9–17.6 pg/mL, respectively. Exclusion criteria were as follows: patients under treatment with thyroxine, or anti

Open access

Xia Wu, Zhiling Li, Wenjiang Sun, and Huan Zheng

-C, LDL-C, TG, creatinine (Cr), uric acid (UA), free triiodothyronine (FT3), free thyroxine (FT4), thyroid-stimulating hormone (TSH), TT (total testosterone), and Hcy levels were recorded. Hcy values were determined with an assay kit (MAKER, China) based

Open access

Xiaoya Zheng, Heng Xiao, Jian Long, Qiang Wei, Liping Liu, Liping Zan, and Wei Ren

free triiodothyronine (FT3), free thyroxine (FT4), ultrasensitive thyroid-stimulating hormone (uTSH), and thyroid antithyroglobulin autoantibody (TgAb) were detected with an immunochemical-automated analyzer (Beckman Coulter UniCelDxI 800; Beckman

Open access

Jan Calissendorff and Henrik Falhammar

30 days who developed serious hyperthyroidism ( 8 ). In untreated hyperthyroidism 0.5 mL (375 mg) daily treatment with LS significantly reduces both free thyroxine (FT4) and free triiodothyronine (FT3) within 5–10 days, while TSH remains below the

Open access

Aaron Lerner, Patricia Jeremias, and Torsten Matthias

in the general population is 1–1.5%. It is gluten depended, thus early diagnosis and subsequent adherence to a gluten-free diet is highly recommended ( 5 , 6 ). Pathophysiologically, the enzyme tissue transglutamimnase (tTg) is the autoantigen. By

Open access

Isabel M Abreu, Eva Lau, Bernardo de Sousa Pinto, and Davide Carvalho

Introduction Subclinical hypothyroidism (SCH) is diagnosed biochemically when both serum-free thyroxine (FT4) and free triiodothyronine (FT3) are within the normal range, whereas the serum thyroid-stimulating hormone (TSH) is elevated ( 1

Open access

Xuechao Jiang, Yonghui Wang, Xiaoying Li, Leqi He, Qian Yang, Wei Wang, Jun Liu, and Bingbing Zha

/mL; TRAb <1.75 IU/L. FT3, free triiodothyronine; FT4, free thyroxine; TGAb, thyroglobulin antibody; TPOAb, thyroid peroxidase antibody; TRAb, TSH receptor antibody; TSH, thyroid stimulating hormone. Thyroid function and antithyroid