Translational and Clinical Research Institute, Faculty of Medical Sciences, Newcastle University, Newcastle upon Tyne, UK
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John Walton Muscular Dystrophy Research Centre, Newcastle University and Newcastle Hospitals NHS Foundation Trust, Newcastle upon Tyne, UK
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John Walton Muscular Dystrophy Research Centre, Newcastle University and Newcastle Hospitals NHS Foundation Trust, Newcastle upon Tyne, UK
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John Walton Muscular Dystrophy Research Centre, Newcastle University and Newcastle Hospitals NHS Foundation Trust, Newcastle upon Tyne, UK
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Translational and Clinical Research Institute, Faculty of Medical Sciences, Newcastle University, Newcastle upon Tyne, UK
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John Walton Muscular Dystrophy Research Centre, Newcastle University and Newcastle Hospitals NHS Foundation Trust, Newcastle upon Tyne, UK
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Mercuri E & Aartsma-Rus A . Duchenne muscular dystrophy . Nature Reviews. Disease Primers 2021 7 13 . ( https://doi.org/10.1038/s41572-021-00248-3 ) 2 Matthews E Brassington R Kuntzer T Jichi F & Manzur AY . Corticosteroids for the
Facultad de Medicina, Universidad Complutense de Madrid, Madrid, España
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Servicio de Endocrinología y Nutrición. Instituto de Investigación Sanitaria del Hospital Clínico San Carlos (IdISSC), Hospital Clínico San Carlos, Madrid, España
Centro de Investigación Biomédica en Red de Diabetes y Enfermedades Metabólicas Asociadas (CIBERDEM), Madrid, España
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Servicio de Endocrinología y Nutrición. Instituto de Investigación Sanitaria del Hospital Clínico San Carlos (IdISSC), Hospital Clínico San Carlos, Madrid, España
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and other morbidities with Cushing's syndrome associated with corticosteroids: a review . Integrated Blood Pressure Control 2011 4 7 – 16 . ( https://doi.org/10.2147/IBPC.S9486 ) 40 Sinnema M Maaskant MA Lantman-de Valk HMJV van Nieuwpoort
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empiric treatment among critically ill patients who present with signs or symptoms indicating adrenal insufficiency. A typical stress dose of corticosteroids includes an initial dose of 100 mg HC IV followed by 50 mg HC IV every 6 h. Depending on the
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Department of Medicine, University of Maryland School of Medicine, Baltimore, Maryland, USA
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aldosterone and corticosterone. Collectively, the rodent work summarized in Fig. 2 implicates EO as the humoral intermediary that may link brain Ang II with augmented adrenal secretion of corticosteroids ( Fig. 2 ). Figure 2 Proposed role of EO as a
BARROW Neurological Institute at Phoenix Children’s Hospital, Phoenix, Arizona, USA
Department of Child Health, University of Arizona College of Medicine – Phoenix, Phoenix, Arizona, USA
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Department of Child Health, University of Arizona College of Medicine – Phoenix, Phoenix, Arizona, USA
Department of Biology and Biochemistry, University of Bath, UK
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Department of Child Health, University of Arizona College of Medicine – Phoenix, Phoenix, Arizona, USA
Department of Biology and Biochemistry, University of Bath, UK
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Department of Child Health, University of Arizona College of Medicine – Phoenix, Phoenix, Arizona, USA
School of Biological and Health Systems Engineering, Arizona State University, Tempe, Arizona, USA
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BARROW Neurological Institute at Phoenix Children’s Hospital, Phoenix, Arizona, USA
Department of Child Health, University of Arizona College of Medicine – Phoenix, Phoenix, Arizona, USA
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BARROW Neurological Institute at Phoenix Children’s Hospital, Phoenix, Arizona, USA
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Introduction Adrenal insufficiency (AI) is characterised by inadequate adrenal corticosteroid hormone production and is classified as primary, secondary or tertiary ( 1 ). Primary adrenal insufficiency (PAI) is caused by pathological processes
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