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Chun-feng Lu, Xiao-qin Ge, Yan Wang, Jian-bin Su, Xue-qin Wang, Dong-mei Zhang, Feng Xu, Wang-shu Liu, and Min Su

of T2D management. In the absence of overt cardiovascular disease, the assessment of heart rate-corrected QT (QTc) interval may have a potential role in screening for cardiovascular complications ( 4 ). Prolonged QTc interval may be a surrogate marker

Open access

Jing Hong, Wen-Yue Liu, Xiang Hu, Fei-Fei Jiang, Ze-Ru Xu, Fang Li, Fei-Xia Shen, and Hong Zhu

end of the T wave, represents the duration of electrical depolarization and repolarization of the ventricular walls ( 5 ). The prolongation of heart rate-corrected QT interval (QTc) is a possible consequence of cardiac autonomic neuropathy, and it is a

Open access

Tao Yuan, Lanping Jiang, Chen Chen, Xiaoyan Peng, Min Nie, Xuemei Li, Xiaoping Xing, Xuewang Li, and Limeng Chen

years and included 19 male and 9 female patients. All patients were normotensive. Elongation of the corrected Q-T interval (QTc > 433 ms) was observed in 13 of 26 patients (ECG data were unavailable in 2 patients). Recurrent muscle weakness, carpopedal

Open access

Anna-Pauliina Iivonen, Johanna Känsäkoski, Atte Karppinen, Leena Kivipelto, Camilla Schalin-Jäntti, Auli Karhu, and Taneli Raivio

–2%) and were p53 negative. No Ki-67 and p53 immunostaining were performed for the third tumor. Available electrocardiograms (ECG) showed that patient 861 had a normal QT interval. Patient ST6 had a prolonged QT interval during transient diabetic

Open access

Maxime Duval, Kalyane Bach-Ngohou, Damien Masson, Camille Guimard, Philippe Le Conte, and David Trewick

arrhythmias. A significant correlation was found between corrected calcium levels and QTc lengthening. No correlation was however reported between calcium levels and arrhythmias. The authors concluded that the evidence for reversible cardiac dysfunction

Open access

Aldo Bonaventura, Fabrizio Montecucco, and Franco Dallegri

QT interval prolongation (49) . The latter is thought to be a possible mechanism leading to fatality during CV events, being precipitated by increased circulating levels of cathecolamines and hypokalemia (50, 51) , even if hypokalemia does not