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Open access

Rachel D C A Diniz, Renata M Souza, Roberto Salvatori, Alex Franca, Elenilde Gomes-Santos, Thiago O Ferrão, Carla R P Oliveira, João A M Santana, Francisco A Pereira, Rita A A Barbosa, Anita H O Souza, Rossana M C Pereira, Alécia A Oliveira-Santos, Allysson M P Silva, Francisco J Santana-Júnior, Eugênia H O Valença, Viviane C Campos, and Manuel H Aguiar-Oliveira

hepatocellular carcinoma is higher than 10% in 5 years (6) . It is therefore important to define the causes of NAFLD. Adult-onset GH deficiency (AOGHD) constitutes a specific model of metabolic syndrome (7, 8) , with visceral obesity, insulin resistance

Open access

Robert Rapaport, Peter A Lee, Judith L Ross, Paul Saenger, Vlady Ostrow, and Giuseppe Piccoli

reported to have historical auxological data consistent with being born SGA. The effectiveness of GH therapy for increasing short-term growth rates and adult height in patients with various growth disorders is well documented ( 6 , 7 , 8 , 9 , 10

Open access

Aleksandra Gilis-Januszewska, Łukasz Kluczyński, and Alicja Hubalewska-Dydejczyk

). Kelly et al. confirmed that patients with post-traumatic GH deficiency (GHD) have more severe cognitive impairment than those with normal values of GH ( 85 ). Several studies reported more frequent deficits in memory and attention, worse reaction

Open access

A C Paepegaey, M Coupaye, A Jaziri, F Ménesguen, B Dubern, M Polak, J M Oppert, M Tauber, G Pinto, and C Poitou

patients of the transition group had a GH stimulation test in childhood than those without transition. GH deficiency (GHD) was found in almost all of the children when performed in childhood before GH start and was found in half of the adults. Only about

Open access

Guillem Cuatrecasas, Hatice Kumru, M Josep Coves, and Joan Vidal

Introduction Growth hormone deficiency (GHD) has been studied in neurological disorders affecting central nervous systems, such as multiple sclerosis to amyotrophic lateral sclerosis ( 1 , 2 ). Because of the direct impact to the pituitary

Open access

Esben Thyssen Vestergaard, Morten B Krag, Morten M Poulsen, Steen B Pedersen, Niels Moller, Jens Otto Lunde Jorgensen, and Niels Jessen

study was to investigate the potential effect of ghrelin- and GH-induced insulin resistance on circulating RBP4 concentrations. To study GH-independent effects of ghrelin, we conducted one experiment in adults with GH deficiency (GHD) receiving an acute

Open access

Charlotte Höybye, Andreas F H Pfeiffer, Diego Ferone, Jens Sandahl Christiansen, David Gilfoyle, Eva Dam Christoffersen, Eva Mortensen, Jonathan A Leff, and Michael Beckert

Introduction Hypothalamic–pituitary diseases and/or injury can lead to adult growth hormone deficiency (AGHD) ( 1 ). The decrease (or total loss) of growth hormone (GH) production, with a subsequent disruption in the hormone’s relationship

Open access

Werner F Blum, Abdullah Alherbish, Afaf Alsagheir, Ahmed El Awwa, Walid Kaplan, Ekaterina Koledova, and Martin O Savage

, particularly in patients with severe GH deficiency when pre-treatment IGF-I SDS is very low ( 77 ). Epidemiological studies of high IGF-I and IGFBP-3 Epidemiological studies in adults have suggested that high IGF-I concentrations could potentially be