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Natalio García-Honduvilla, Alberto Cifuentes, Miguel A Ortega, Marta Pastor, Garazi Gainza, Eusebio Gainza, Julia Buján, and Melchor Álvarez-Mon
Darling M Rojas-Canales, Michaela Waibel, Aurelien Forget, Daniella Penko, Jodie Nitschke, Fran J Harding, Bahman Delalat, Anton Blencowe, Thomas Loudovaris, Shane T Grey, Helen E Thomas, Thomas W H Kay, Chris J Drogemuller, Nicolas H Voelcker, and Patrick T Coates
technologies, such as insulin-secreting tissues from adult and embryonic stem cells and islets from animal sources, which aim to alleviate organ shortage barriers. Currently, cell culture bags made of gas permeable polymers such as poly
Qian Deng, Yue Zhu, Mengmeng Zhang, Aihua Fei, Jiaqi Liang, Jinjin Zheng, Qingping Zhang, Tong Cheng, and Xia Ge
inhibits osteogenic differentiation of bone marrow mesenchymal stem cells via regulating miR-493-5p/ZEB2 signalling . Journal of Biochemistry 2020 167 613 – 621 . ( https://doi.org/10.1093/jb/mvaa011 ) 54 Wang X Ma H Sun J Zheng T Zhao P Li H
Angelica Amorim Amato, Hailey Brit Wheeler, and Bruce Blumberg
multipotent mesenchymal stromal stem cells (a.k.a., mesenchymal stem cells, MSCs) or existing preadipocytes. Activation of specific nuclear hormone receptors by EDCs has been extensively studied as a mechanism underlying obesogen action, since it has the
Davoud Jafari-Gharabaghlou, Mostafa Vaghari-Tabari, Hajar Oghbaei, Laura Lotz, Reza Zarezadeh, Yeganeh Rastgar Rezaei, Mahnaz Ranjkesh, Mohammad Nouri, Amir Fattahi, Saba Nikanfar, and Ralf Dittrich
. Declaration of interest The authors declare that there is no conflict of interest that could be perceived as prejudicing the impartiality of this review. Funding This work was supported by the Stem Cell Research Center of Tabriz University of Medical
Ying Hua, Jinqiong Fang, Xiaocong Yao, and Zhongxin Zhu
from bone marrow mesenchymal stem cells, and, thus, increasing adipogenic differentiation decreases osteogenic differentiation ( 20 ). Another possible explanation is that obesity-induced hypermetabolism caused by enhanced insulin signaling leads to
Kaisa K Ivaska, Maikki K Heliövaara, Pertti Ebeling, Marco Bucci, Ville Huovinen, H Kalervo Väänänen, Pirjo Nuutila, and Heikki A Koistinen
rodent osteoblasts (10, 13, 14, 15) , osteoclast-like cells (16) and in primary human osteoblasts differentiated from bone marrow-derived mesenchymal stem cells (17) . In vitro , insulin signaling promotes osteoblast differentiation and OC expression
Qian Yang, Wencai Ke, Fanfan Pan, Xinmei Huang, Jun Liu, and Bingbing Zha
, hyperthyroidism significantly affected the proliferative potential of human hematopoietic stem/progenitor cells by decreasing the expression of pro- and anti-apoptotic genes, such as B-cell lymphoma-2 (BCL-2), B-cell lymphoma-XL (BCL-xL), and BCL2-associated X
Josephina G Kuiper, Aline C Fenneman, Anne H van der Spek, Elena Rampanelli, Max Nieuwdorp, Myrthe P P van Herk-Sukel, Valery E P P Lemmens, Ernst J Kuipers, Ron M C Herings, and Eric Fliers
and enhance cell differentiation in intestinal cells. Furthermore, increased intracellular TH levels, through Dio3 depletion, significantly reduced the tumorigenic potential of colorectal stem cells ( 9 ). However, these results were obtained from
Giorgio Bedogni, Andrea Mari, Alessandra De Col, Sofia Tamini, Amalia Gastaldelli, and Alessandro Sartorio
Introduction The prevalence of childhood type 2 diabetes mellitus (T2DM) is rapidly increasing worldwide ( 1 ). T2DM is characterized by visceral obesity, insulin resistance and defective β-cell function. In children and adolescents, glucose
