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Martin Wiegand, David J Halsall, Sarah L Cowan, Kevin Taylor, Robert J B Goudie, Jacobus Preller, and Mark Gurnell

inhibition. The uninhibited AngII may then play a role in the pathogenesis of observed hypertension ( 6 ), inflammation, immunothrombosis and possible fibrosis in COVID-19. Whilst elevated serum cortisol has been identified as a marker of poor prognosis in

Open access

Sweta Budyal, Swati Sachin Jadhav, Rajeev Kasaliwal, Hiren Patt, Shruti Khare, Vyankatesh Shivane, Anurag R Lila, Tushar Bandgar, and Nalini S Shah

Introduction Subclinical Cushing's syndrome (SCS) is defined as autonomous cortisol secretion in patients without typical signs and symptoms of hypercortisolism, as in the classic Cushing's syndrome (CS) (1) . Screening for a rare disorder like SCS

Open access

Ana Podbregar, Tomaž Kocjan, Matej Rakuša, Peter Popović, Manca Garbajs, Katja Goricar, Andrej Janez, and Mojca Jensterle

-up ( 4 , 5 ), increases in NFAIs’ size and development of mild autonomous cortisol excess (MACE) over time have also been reported for this clinical entity ( 6 , 7 , 8 , 9 ). Currently, adequate long-term follow-up strategies of NFAIs after the

Open access

Jan W Eriksson, Reem A Emad, Martin H Lundqvist, Niclas Abrahamsson, and Maria C Kjellsson

quantitative importance of other factors, such as inhibition by insulin and stimulation by amino acids, is disputed ( 8 , 9 ). Hypoglycemia also triggers the release of other counter-regulatory hormones such as catecholamines, cortisol, and growth hormone

Open access

Emmanuelle Motte, Anya Rothenbuhler, Stephan Gaillard, Najiba Lahlou, Cécile Teinturier, Régis Coutant, and Agnès Linglart

Introduction Cushing’s disease (CD) is characterized by adrenocorticotropin (ACTH)-dependent cortisol excess originating from a pituitary adenoma and accounts for approximately 85% of cases of pediatric Cushing’s syndrome ( 1 , 2 , 3 , 4

Open access

Alberto Giacinto Ambrogio, Massimiliano Andrioli, Martina De Martin, Francesco Cavagnini, and Francesca Pecori Giraldi

assess the pattern of ACTH/cortisol responses to desmopressin in the years following transsphenoidal surgery in patients with Cushing’s disease on long-term remission and to establish whether changes in the response to desmopressin in patients during

Open access

Fidéline Bonnet-Serrano, Maxime Barat, Anna Vaczlavik, Anne Jouinot, Lucas Bouys, Christelle Laguillier-Morizot, Corinne Zientek, Catherine Simonneau, Etienne Larger, Laurence Guignat, Lionel Groussin, Guillaume Assié, Jean Guibourdenche, Ioannis Nicolis, Marie-Claude Menet, and Jérôme Bertherat

series) but they can be responsible for autonomous cortisol secretion in about 12% of cases (1–29% among series) and for aldosterone secretion in 2.5% of cases (1.6–3.3% among series) ( 5 ). Primary bilateral macronodular adrenal hyperplasia (PBMAH) is

Open access

Thomas Reinehr, Alexandra Kulle, Juliane Rothermel, Caroline Knop-Schmenn, Nina Lass, Christina Bosse, and Paul-Martin Holterhus

, 21-deoxycorticosterone, deoxycorticosterone, corticosterone, 11-deoxycortisol, cortisol, cortisone, androstenedione, testosterone, dehydroepiandrostenedione sulfate (DHEA-S), estrone (E1), estradiol (E2), luteinizing hormone (LH), follicle

Open access

Lawrence D Hayes, Peter Herbert, Nicholas F Sculthorpe, and Fergal M Grace

exercise session as previously described ( 21 ). Serum concentrations of TT, SHBG and cortisol were measured by electrochemiluminescent immunoassay on the E601 module of the Roche Cobas 6000 (Burgess Hill, West Sussex, UK). Inter-assay coefficients of

Open access

L M Mongioì, R A Condorelli, S La Vignera, and A E Calogero

hydrocortisone dose of 15–25 mg ( 8 ). The conventional treatment involves a double or triple administration of glucocorticoid, thus causing a supraphysiological cortisol exposition. Recently, a new once-daily dual-release hydrocortisone formulation has been