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after completion of puberty ( 2 ). Hormone replacement therapy (HRT) is required after gonadectomy in order to maintain secondary sexual characteristics, bone and cardiovascular health and to promote general wellbeing and sexual function. In the study by

Introduction The sexual dimorphism in the mechanisms involved in the regulation of somatotroph axis is well documented, and it might account for some of the sex differences in growth rate and body composition ( 1 ). Indeed, sex hormones are

Norway (69°N). Fit Futures 1 was performed at The Research Unit at the University Hospital in North Norway, from September 2010 to April 2011, with 1038 attendees (girls n  = 508) in their first year of upper secondary school (92.9% attendance rate), >95

tissues, particularly on adipose tissue. The sexual dimorphism of androgens in the pathophysiology of T2DM is emphasized. Sex hormone differences between the sexes Males and females produce the same steroid hormones in a similar way. Regarding

-related characteristics and tested using general linear regression models. To compare the predictive value of the three free testosterone algorithms in relation to cardiometabolic conditions, Cox proportional hazards models with age as the underlying time scale were

). Recent reports have shown that oxytocin also contributes to maternal and sexual behaviors, bone formation, and metabolism ( 6 ). Strong evidence now also supports a relationship between oxytocin and diabetes; for example, Kujath et al. found lower

physicians. Methods Patient questionnaire design A 12-item patient questionnaire was developed based on the generally understood clinical characteristics and symptomology of CS, aiming to assess patients’ experiences of symptoms, diagnosis, care

features of the selected studies. Table 1 Characteristics of studies included in the meta-analysis. References Number of treated patients Number of control patients Mean age (years) Baseline sperm concentration of treated patients (million/ml) Baseline

present with a characteristic fat distribution showing deposits such as a cresty neck and at the comb, side of the chest, hip region, and tail head. Large omental fat deposits are also present, but not visible ( Fig. 1 ). Figure 1 Horse suffering from EMS

Nanog that are characteristic for early embryonic stem cells ( 21 , 22 , 23 ). The exact nature of the migration related to proliferation is not clear; some evidence points to PGCs halting mitosis until colonizing the gonadal ridge ( 24 ), whereas