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L E Zijlstra, D M van Velzen, S Simsek, S P Mooijaart, M van Buren, D J Stott, I Ford, J W Jukema, and S Trompet

Introduction Subclinical hyperthyroidism has been linked to atrial fibrillation and coronary artery calcification, whereas subclinical hypothyroidism has been associated with hypercholesterolemia and atherosclerosis ( 1 , 2 ). Despite these

Open access

Xingyao Tang, Zhi-Hui Song, Dawei Wang, Jinkui Yang, Marly Augusto Cardoso, Jian-Bo Zhou, and Rafael Simó

factor for dementia, promotes neurocognitive function and regulation metabolic processes ( 4 ). Thyroid dysfunction, including clinical hyperthyroidism, subclinical hyperthyroidism, subclinical hypothyroidism, and clinical hypothyroidism, has been

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Nadia Sawicka-Gutaj, Ariadna Zybek-Kocik, Michał Kloska, Paulina Ziółkowska, Agata Czarnywojtek, Jerzy Sowiński, Dorota Mańkowska-Wierzbicka, and Marek Ruchała

-apoptotic and proinflammatory properties of this adipocytokine has been recently reported ( 11 , 12 , 13 ). In addition, visfatin serum concentrations in hypothyroidism are associated with free thyroid hormones and antithyroperoxidase antibodies, while in

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Caroline Serrano-Nascimento, Rafael Barrera Salgueiro, Kaio Fernando Vitzel, Thiago Pantaleão, Vânia Maria Corrêa da Costa, and Maria Tereza Nunes

). Therefore, several physiological changes occur in the maternal hypothalamus–pituitary–thyroid axis to successfully achieve both maternal and fetal TH serum levels requirements ( 16 ). ID is commonly associated with maternal hypothyroidism and several

Open access

Brenda Anguiano, Carlos Montes de Oca, Evangelina Delgado-González, and Carmen Aceves

differentiating actions of THs are suppressed, and local hypothyroidism induced by the high DIO3 leads to an increase in cell proliferation ( 2 ). miRNA, miR-379, and miR-154 of the delta-like 1 homolog-deiodinase 3 cluster are increased in serum of prostate

Open access

Karim Gariani and François R Jornayvaz

) - Pituitary gland (TSH) - Thyroid gland (free T4) - Primary hypothyroidism (thyroid disease) - Secondary hypothyroidism (pituitary (TSH) or hypothalamic disease (TRH)) Growth hormone (GH) - Pituitary gland (GH) - Ectopic secretion - Acromegaly

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Dimitra Argyro Vassiliadi, Ioannis Ilias, Maria Pratikaki, Edison Jahaj, Alice G Vassiliou, Maria Detsika, Kleio Ampelakiotou, Marina Koulenti, Konstantinos N Manolopoulos, Stamatis Tsipilis, Evdokia Gavrielatou, Aristidis Diamantopoulos, Alexandros Zacharis, Nicolaos Athanasiou, Stylianos Orfanos, Anastasia Kotanidou, Stylianos Tsagarakis, and Ioanna Dimopoulou

Groups n Low/suppressed TSH NTIS Overt and subclinical thyrotoxicosis Overt and subclinical hypothyroidism Other findings July Sun et al. Retrospective 336 Unselected COVID-19 + 336 NR NR NR NR Lower TSH levels

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Adriano N Cury, Verônica T Meira, Osmar Monte, Marília Marone, Nilza M Scalissi, Cristiane Kochi, Luís E P Calliari, and Carlos A Longui

tremendous controversy regarding the most appropriate treatment schedule for this age group (3) . No strategy targets the underlying autoimmunity, and the control of thyroid hyperfunction is usually achieved by the induction of hypothyroidism. In many

Open access

Stine Linding Andersen and Stig Andersen

likely to explain the higher maternal TSH in the early pregnancy prior to the hCG-peak ( 13 , 15 ). In line with this thought, patients with DIO3 containing hemangiomas present with consumptive hypothyroidism and a high rT3/T3-ratio ( 16 ). These

Open access

Paolo G Arduino, Dora Karimi, Federico Tirone, Veronica Sciannameo, Fulvio Ricceri, Marco Cabras, Alessio Gambino, Davide Conrotto, Stefano Salzano, Mario Carbone, and Roberto Broccoletti

hypothyroidism ( 4 , 5 , 6 ) or Hashimoto’s thyroiditis ( 7 ), while other studies denied it ( 8 ). Considering all the previous deliberations, in such a discordant context, we decided to assess the association of OLP with some comorbidities, focusing our