mediated by calcium fluxes remains unsolved ( 31 ). The current study can be regarded as a pilot study or descriptive study on the transcriptomic effects of vitamin D supplementation due to lack of functional outcomes. Future work should ideally be
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Elisabet Einarsdottir, Minna Pekkinen, Kaarel Krjutškov, Shintaro Katayama, Juha Kere, Outi Mäkitie, and Heli Viljakainen
Arpna Sharma, Vijay Simha Baddela, Frank Becker, Dirk Dannenberger, Torsten Viergutz, and Jens Vanselow
some other proteins like MLN4 have been known to possess STAR- like activity in promoting cholesterol flux into mitochondria ( 55 ). From our data, it became very clear that the saturated fatty acids tested (PA, SA) elicited very different, partly
Xuechao Jiang, Yonghui Wang, Xiaoying Li, Leqi He, Qian Yang, Wei Wang, Jun Liu, and Bingbing Zha
T cell receptor signaling . PLoS ONE 2012 7 e43200 . ( https://doi.org/10.1371/journal.pone.0043200 ) 32 Ostrakhovitch EA Wang Y Li SS. SAP binds to CD22 and regulates B cell inhibitory signaling and calcium flux . Cellular
Mai Morsi, Torben Schulze, Eike Früh, Dennis Brüning, Uwe Panten, and Ingo Rustenbeck
of the resulting changes in mitochondrial metabolite flux the consumption of oxaloacetate is slower than in fresh islets, where oxaloacetate is likely the first Krebs cycle metabolite to become critically low during prolonged phases of depolarization
David Koeckerling, Jeremy W Tomlinson, and Jeremy F Cobbold
lipid export or degradation is exceeded by lipid import or synthesis. Main sources of hepatic lipid aggregation are a flux of free fatty acids (FFA) from peripheral adipose tissue (59%), followed by hepatic de novo lipogenesis (26%) and dietary intake
Magdalene K Montgomery and Nigel Turner
AMPK-mediated lowering of malonyl-CoA levels and increased CPT1-mediated fatty acid entry into mitochondria. Increased flux of fatty acids into β oxidation generates both NADH and FADH 2 , with the entry of electrons into the ETC via complex II allowing
Madalena von Hafe, João Sergio Neves, Catarina Vale, Marta Borges-Canha, and Adelino Leite-Moreira
-genomic mechanisms. The major effects of THs on the myocardium are mediated by T3 ( Table 1 ), which stimulates nearly all of the transporters and ion channels involved in calcium myocardial fluxes, upregulating sarcoplasmic reticulum calcium-activated ATPase 2
Helle Keinicke, Gao Sun, Caroline M Junker Mentzel, Merete Fredholm, Linu Mary John, Birgitte Andersen, Kirsten Raun, and Marina Kjaergaard
hepatic glycogen content and (4) downregulation of genes converting pyruvate into acetyl-CoA production ( Pdhb , Mpc2 ) ( Fig. 2 and Table 1 ). In addition, the increase in Glut2 expression by FGF21 suggested greater capacity for hepatic glucose flux
