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Iben Katinka Greiber, Casper P Hagen, Alexander Siegfried Busch, Mikkel Grunnet Mieritz, Lise Aksglæde, Katharina Main, Kristian Almstrup, and Anders Juul

preovulatory human follicles’ fluid . Journal of Clinical Endocrinology and Metabolism 2006 91 4064 – 4069 . ( ) 10.1210/jc.2006-1066 39 Chang HM Klausen C Leung PCK. Antimüllerian hormone inhibits follicle-stimulating

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Sakina Kherra, Wendy Forsyth Paterson, Filiz Mine Cizmecioglu, Jeremy Huw Jones, Mariam Kourime, Heba Hassan Elsedfy, Sameh Tawfik, Andreas Kyriakou, Mohamad Guftar Shaikh, and Malcolm David Cairns Donaldson

). Luteinising hormone (LH) and follicle-stimulating hormone (FSH) were measured using an Abbott AxSYM from 1990 until 2004, by DPC Immulite 2000 (Siemens) until 2014 and Abbott Architect immunoassay thereafter. Testosterone and oestradiol were measured at

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Rossella Cannarella, Andrea Crafa, Sandro La Vignera, Rosita A Condorelli, and Aldo E Calogero

mediates the proliferation and differentiation of Sertoli, germ, and Leydig cells and the mediation of follicle-stimulating hormone (FSH) effects through the common PI3K/AKT pathway ( 7 ). This knowledge translated to humans would possibly highlight the

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Davoud Jafari-Gharabaghlou, Mostafa Vaghari-Tabari, Hajar Oghbaei, Laura Lotz, Reza Zarezadeh, Yeganeh Rastgar Rezaei, Mahnaz Ranjkesh, Mohammad Nouri, Amir Fattahi, Saba Nikanfar, and Ralf Dittrich

, testosterone, LH, and follicle-stimulating hormone (FSH) on resistin expression have been demonstrated in ovarian follicles ( 18 ). Moreover, it has been reported that estradiol could induce adiponectin expression ( 19 ). However, the inhibitory effects of

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Yao Chen and Shu-ying Fang

that were found or discussed in these publications were summarized. The genetic polymorphisms are classified as steroidogenic acute regulatory ( StAR ) gene polymorphisms, gonadotropin-releasing hormone receptor ( GnRHR ) gene polymorphisms, follicle-stimulating

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Sidsel Mathiesen, Kaspar Sørensen, Marianne Ifversen, Casper P Hagen, Jørgen Holm Petersen, Anders Juul, and Klaus Müller

risk of azoospermia). Reproductive hormones and semen samples From a clinical database, we obtained registered daytime circulating levels of follicle-stimulating hormone (FSH), LH, total testosterone, and inhibin B as well as registered semen

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Angela Köninger, Antonella Iannaccone, Ensar Hajder, Mirjam Frank, Boerge Schmidt, Ekkehard Schleussner, Rainer Kimmig, Alexandra Gellhaus, and Hans Dieplinger

(follicle-stimulating hormone) ( n  = 37), laparoscopic ovarian drilling ( n  = 1), in vitro fertilization (IVF) ( n  = 4) and no therapeutic intervention ( n  = 5). Informed written consent was obtained from all patients. The study was approved by the

Open access

Laura van Iersel, Sarah C Clement, Antoinette Y N Schouten-van Meeteren, Annemieke M Boot, Hedi L Claahsen-van der Grinten, Bernd Granzen, K Sen Han, Geert O Janssens, Erna M Michiels, A S Paul van Trotsenburg, W Peter Vandertop, Dannis G van Vuurden, Hubert N Caron, Leontien C M Kremer, and Hanneke M van Santen

-up. Luteinizing hormone and follicle-stimulating hormone deficiency Luteinizing hormone (LH) and follicle-stimulating hormone (FSH) deficiency (LH/FSHD) was defined by repeatedly low LH and/or FSH concentrations in the absence of pubertal development (girls aged

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Arno Téblick, Lies Langouche, and Greet Van den Berghe

hypothalamic hormones, GnRH is discharged in pulses into the hypophyseal portal system. At the level of the pituitary, GnRH stimulates the gonadotropes to synthesize and release stored luteinizing hormone (LH) and follicle-stimulating hormone (FSH). GnRH pulses

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Aaron Lerner, Patricia Jeremias, and Torsten Matthias

-thyroid peroxidase antibodies (TPO). The disease is a common cause of low thyroid hormones with a high thyroid-stimulating hormone (TSH) but some exhibit subclinical hypothyroidism. Interestingly, symptoms of HT and celiac disease (CD) often overlap ( Table 1 ), and