Search Results

You are looking at 51 - 60 of 207 items for

  • Refine by Access: All content x
Clear All Modify Search
Open access

Anastasia P Athanasoulia-Kaspar, Matthias K Auer, Günter K Stalla, and Mira Jakovcevski

, high doses of glucocorticoid substitution therapy and older age at diagnosis are the most consistently identified risk factors associated with increased morbidity and mortality ( 18 , 19 ). In addition, growth hormone deficiency (GHD) has been linked

Open access

Lang Qin, Xiaoming Zhu, Xiaoxia Liu, Meifang Zeng, Ran Tao, Yan Zhuang, Yiting Zhou, Zhaoyun Zhang, Yehong Yang, Yiming Li, Yongfei Wang, and Hongying Ye

Introduction Hypertension is a common chronic condition in patients with Cushing’s disease (CD) and may be the first sign ( 1 ). The pathogenesis of glucocorticoid-induced hypertension is not fully understood. It is thought to be related in

Open access

Filippo Ceccato, Elisa Selmin, Chiara Sabbadin, Miriam Dalla Costa, Giorgia Antonelli, Mario Plebani, Mattia Barbot, Corrado Betterle, Marco Boscaro, and Carla Scaroni

). Furthermore, the clinical picture of SAI may be complicated by the association of other pituitary deficiencies, which could affect both HPA axis and glucocorticoid (GC) treatment ( 5 ). A more severe degree of AI could characterize PAI, due to the impairment

Open access

Marie Freel

document clearly outlines key points in the recognition and management of a new presentation of adrenal insufficiency as well as clarifying how to alter glucocorticoid therapy in the event of intercurrent illness of medical procedures. The lead author of

Open access

Deborah Cosentini, Salvatore Grisanti, Alberto Dalla Volta, Marta Laganà, Chiara Fiorentini, Paola Perotti, Sandra Sigala, and Alfredo Berruti

activity and better survival for cancer patients treated with immune checkpoint inhibitors ( 17 ), was found in a minority of ACC neoplasms ( 18 ). Moreover, the majority of ACC patients has a hormone-secreting disease and glucocorticoids are known to exert

Open access

Jennifer Walsh, Neil Gittoes, Peter Selby, and the Society for Endocrinology Clinical Committee

if vitamin D deficiency or suppressed PTH Second-line treatments Glucocorticoids (inhibit 1,25OHD production) In lymphoma, other granulomatous diseases or 25OHD poisoning Prednisolone 40 mg daily Usually effective in 2–4 days

Open access

Ferdinand Roelfsema, Diana van Heemst, Ali Iranmanesh, Paul Takahashi, Rebecca Yang, and Johannes D Veldhuis

pills in women, substance abuse, neuropsychiatric illness or acute or chronic systemic disease. A complete medical history, physical examination and screening biochemistry tests were normal. No subject had been exposed to glucocorticoids within the

Open access

Aneta Gawlik, Michael Shmoish, Michaela F Hartmann, Stefan A Wudy, Zbigniew Olczak, Katarzyna Gruszczynska, and Ze’ev Hochberg

-transformed concentrations of steroid metabolites (androgens, glucocorticoids and mineralocorticoids) in liver diseases (L1) and with non-liver disease features (L0) patients. Table 3 Ratio of steroid metabolites (enzyme activity): differences between patients

Open access

I Azzam, S Gilad, R Limor, N Stern, and Y Greenman

the stimulatory effect of GHS on corticotroph function is sensitive to glucocorticoid feedback, as the ACTH response to hexarelin was suppressed by pre-treatment with dexamethasone ( 27 ) but enhanced in patients with Addison’s disease and in healthy

Open access

Carlo Cinque, Manuela Zinni, Anna Rita Zuena, Chiara Giuli, Sebastiano G Alemà, Assia Catalani, Paola Casolini, and Roberto Cozzolino

; however, this kind of assessment could be influenced by events, often unknown to experimenters, that took place immediately before sampling. This is particularly important for glucocorticoid hormones. Furthermore, in most cases, blood withdrawal requires